Quantitative Analysis of Highly Cited Literatures on Premature Coronary Heart Disease in CNKI

2021 ◽  
Vol 11 (09) ◽  
pp. 3959-3968
Author(s):  
芳 陈
Keyword(s):  
Coronary Heart Disease ◽  
Heart Disease ◽  
Quantitative Analysis ◽  
Premature Coronary Heart Disease ◽  
Highly Cited

Homocysteine and Risk of Premature Coronary Heart Disease

Circulation ◽  
1996 ◽  
Vol 94 (9) ◽  
pp. 2154-2158 ◽  
Author(s):  
Paula M. Gallagher ◽  
Raymond Meleady ◽  
Denis C. Shields ◽  
Kok Soon Tan ◽  
Dorothy McMaster ◽  
...  
Keyword(s):  
Coronary Heart Disease ◽  
Heart Disease ◽  
Premature Coronary Heart Disease

scholarly journals Renin-angiotensin system gene polymorphisms and premature coronary heart disease

2005 ◽  
Vol 6 (1) ◽  
pp. 38-42 ◽  
Author(s):  
Cevad Sekuri ◽  
F Sirri Cam ◽  
Ertugrul Ercan ◽  
Istemihan Tengiz ◽  
Abdi Sagcan ◽  
...  
Keyword(s):  
Coronary Heart Disease ◽  
Heart Disease ◽  
Gene Polymorphisms ◽  
Renin Angiotensin System ◽  
Angiotensin System ◽  
Renin Angiotensin ◽  
Premature Coronary Heart Disease

2P-0382 Homocysteine and premature coronary heart disease

2003 ◽  
Vol 4 (2) ◽  
pp. 122-123
Author(s):  
M. Ezhov ◽  
V. Naumov ◽  
M. Afanasieva ◽  
S. Pokrovsky
Keyword(s):  
Coronary Heart Disease ◽  
Heart Disease ◽  
Premature Coronary Heart Disease

Familial hypercholesterolaemia

2020 ◽  
pp. 343-348
Author(s):  
Perry Elliott ◽  
Pier D. Lambiase ◽  
Dhavendra Kumar
Keyword(s):  
Coronary Heart Disease ◽  
Heart Disease ◽  
Inborn Error ◽  
Familial Hypercholesterolaemia ◽  
Low Density Lipoprotein ◽  
Density Lipoprotein ◽  
Low Density ◽  
Clinical Criteria ◽  
Premature Coronary Heart Disease ◽  
Coronary Artery Atherosclerosis

Familial hypercholesterolaemia (FH) is an inborn error of metabolism that leads to accumulation of low-density lipoprotein cholesterol (LDL-C) particles in the blood and premature coronary artery atherosclerosis. This chapter covers the clinical criteria for the diagnosis of FH, the genetics that underpins the condition, cascade testing, premature coronary heart disease, and treatment methods.

Risk Factors for Premature Coronary Heart Disease after Successful Liver Transplantation in Adults

1996 ◽  
Vol 6 (4) ◽  
pp. 178-187
Author(s):  
Susan L Smith
Keyword(s):  
Risk Factors ◽  
Coronary Heart Disease ◽  
Liver Transplantation ◽  
Heart Disease ◽  
Solid Organ Transplantation ◽  
Solid Organ ◽  
Transplant Recipients ◽  
Kidney Transplant Recipients ◽  
Pharmacological Agents ◽  
Premature Coronary Heart Disease

As solid-organ transplantation has evolved into a highly effective treatment for end-stage organ disease, the long-term health implications of chronic exposure of recipients to immunosuppressants and other pharmacological agents are becoming more apparent. Coronary heart disease has long been known to plague kidney transplant recipients and more recently has been found to affect heart transplant recipients disproportionately. Coronary heart disease after liver transplantation, however, is less well known. The purpose of this study was to examine risk factors for premature coronary heart disease in asymptomatic adult recipients of liver transplants. Nutrition-related risk factors for coronary heart disease (obesity and hyperlipidemia) were measured in 29 patients before and after liver transplantation. Changes with respect to primary immunosuppression protocol (cyclosporine plus corticosteroid vs tacrolimus plus corticosteroid) were compared. Risk factors that had not been present before transplantation were apparent in both groups by 6 months after transplantation. Although obesity and hyperlipidemia were not found to be independent risk factors for coronary heart disease, they were clinically important when considered in combination. Cyclosporine was associated with significantly higher serum lipid concentrations than was tacrolimus.

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