scholarly journals Chewing time and chewing strokes in different dentofacial deformities

2012 ◽  
Vol 24 (2) ◽  
pp. 130-133 ◽  
Author(s):  
Melissa Nara de Carvalho Picinato-Pirola ◽  
Francisco Veríssimo de Mello-Filho ◽  
Luciana Vitaliano Voi Trawitzki

PURPOSE: To verify whether the number of chewing strokes and the chewing time are influenced by dentofacial deformities in habitual free mastication. METHODS: Participants were 15 patients with diagnosis of class II dentofacial deformity (GII), 15 with class III (GIII), and 15 healthy control individuals with no deformity (CG). Free habitual mastication of a cornstarch cookie was analyzed, considering the number of chewing strokes and the time needed to complete two mastications. Strokes were counted by considering the opening and closing movements of the mandible. The time needed to consume each bite was determined using a digital chronometer, started after the placement of the food in the oral cavity and stopped when each portion was swallowed. RESULTS: There were no differences between groups regarding both the number of strokes and the chewing time. However, with regards to the number of strokes, CG and GII presented a significant concordance between the first and the second chewing situation, which was not observed in GIII. The analysis of time showed significant concordance between the first and second chewing situation in CG, reasonable concordance in GII, and discordance in GIII. CONCLUSION: Dentofacial deformities do not influence the number of chewing strokes or the chewing time. However, class III individuals do not show uniformity regarding these aspects.

2016 ◽  
Vol 64 (4) ◽  
pp. 453-459
Author(s):  
Aline Monise SEBASTIANI ◽  
Nelson Luis Barbosa REBELATTO ◽  
Leandro Eduardo KLÜPPEL ◽  
Delson João da COSTA ◽  
Fernando ANTONINI ◽  
...  

ABSTRACT The combination of orthodontic therapy and orthognathic surgery is a well-established treatment modality for the correction of dentofacial deformities. When these deformities are more severe, involving hypoplastic midface, surgical techniques not used routinely in the treatment of facial changes are required, such as the Le Fort III osteotomy or variations of this technique. Few studies have reported the use of this technique or its modifications in non-syndromic patients. This paper demonstrates the orthodontic-surgical resolution of a patient with dentofacial deformity with severe malocclusion Class III, involving midface hypoplasia, with a modification technique of a Le Fort III osteotomy associated with Le Fort I and sagittal of the rami osteotomies. After three years of postoperative follow-up, the patient demonstrates significant improvement in chewing ability, no functional complaints, and high satisfaction with the aesthetics and improved quality of life.


2018 ◽  
Vol 2018 ◽  
pp. 1-6
Author(s):  
Rafael Correia Cavalcante ◽  
Isabela Polesi Bergamaschi ◽  
Aline Monise Sebastiani ◽  
Fabiano Galina ◽  
Marina Fanderuff ◽  
...  

Introduction. Class II division I malocclusions are the most common dentofacial deformities seen in clinical practice. Severe cases or cases in which growth has ceased may require full correction combining orthodontic and surgical treatment. We report a case of a total mandibular subapical alveolar osteotomy, performed to correct a class II division I dentofacial deformity. Case Report. A 19-year-old female patient was referred to the oral and maxillofacial surgery department at the Federal University of Paraná with chin aesthetic complaints as well as class II malocclusion. The proposed treatment was total mandibular subapical alveolar osteotomy, retaining the chin position and eliminating the need for genioplasty, since, although the patient presented with a class II dentofacial deformity, the chin was well positioned. Under general anesthesia, a “V-shaped” incision was conducted from the right retromolar region to the left retromolar region. A ring of cortical bone was removed around the mental foramen, with the aim to create a space around the mental nerve. Fixation was conducted with plates and screws of the 2.0 system. The patient on six-year follow-up showed osteotomy stability, a better overall occlusion, and outcome satisfaction.


2012 ◽  
Vol 41 (7) ◽  
pp. 830-834 ◽  
Author(s):  
M.N.C. Picinato-Pirola ◽  
W. Mestriner ◽  
O. Freitas ◽  
F.V. Mello-Filho ◽  
L.V.V. Trawitzki

2011 ◽  
Vol 56 (10) ◽  
pp. 972-976 ◽  
Author(s):  
L.V.V. Trawitzki ◽  
J.B. Silva ◽  
S.C.H. Regalo ◽  
F.V. Mello-Filho

2019 ◽  
Vol 9 (2) ◽  
pp. 20-22
Author(s):  
Faisal Rasheed ◽  
Zubair Hassan Awaisi ◽  
Muhammad Inam Elahi ◽  
Abid Hussain Kanju ◽  
Zeenat Naz Sahito ◽  
...  

Introduction: Hyoid bone is attached to mandible and cranium by different ligaments and muscular attachments and plays important role in maintaining of functions of oral cavity such as posture of tongue. Hyoid bone is attached to mandible and cranium by different ligaments and muscular attachments and plays important role in maintaining of functions of oral cavity such as posture of tongue. Materials & Method: Study was carried out in Nishtar Institute of Dentistry, Multan in May-June 2019. Lateral cephalogram of 113 patients were taken from orthodontic department and drawn by using 3H pencil and acetate matte sheets. Skeletal malocclusions were distributed into Class I, Class II and Class III on the bases of ANB angle. Hyoid bone position was determined using method devised by Bibby and Preston. IBM SPSS was used to assess the statistical data and mean, correlations and one sample t-test were applied. Result: Data shows that in population of Multan, Pakistan, hyoid bone is posterior and downward in position and lies close to mandibular plane. Mean value of C3-H, Hyoid angle and H-RGn was 28.76mm ± 4.01mm, 15.26º ± 5.85º and 33.6mm ± 6.12mm. Conclusion: It is concluded that hyoid bone is positioned more downward and backward direction in Skeletal Class I cases from given sample, moderately in Class III and less in Class II cases.


Healthcare ◽  
2021 ◽  
Vol 9 (4) ◽  
pp. 413
Author(s):  
Hui-Ling Chen ◽  
Jason Chen-Chieh Fang ◽  
Chia-Jung Chang ◽  
Ti-Feng Wu ◽  
I-Kuan Wang ◽  
...  

Background. Previous studies have shown that environmental cadmium exposure could disrupt salivary gland function and is associated with dental caries and reduced bone density. Therefore, this cross-sectional study attempted to determine whether tooth decay with tooth loss following cadmium exposure is associated with some dental or skeletal traits such as malocclusions, sagittal skeletal pattern, and tooth decay. Methods. Between August 2019 and June 2020, 60 orthodontic patients with no history of previous orthodontics, functional appliances, or surgical treatment were examined. The patients were stratified into two groups according to their urine cadmium concentrations: high (>1.06 µg/g creatinine, n = 28) or low (<1.06 µg/g creatinine, n = 32). Results. The patients were 25.07 ± 4.33 years old, and most were female (female/male: 51/9 or 85%). The skeletal relationship was mainly Class I (48.3%), followed by Class II (35.0%) and Class III (16.7%). Class I molar relationships were found in 46.7% of these patients, Class II molar relationships were found in 15%, and Class III molar relationships were found in 38.3%. The mean decayed, missing, and filled surface (DMFS) score was 8.05 ± 5.54, including 2.03 ± 3.11 for the decayed index, 0.58 ± 1.17 for the missing index, and 5.52 ± 3.92 for the filled index. The mean index of complexity outcome and need (ICON) score was 53.35 ± 9.01. The facial patterns of these patients were within the average low margin (26.65 ± 5.53 for Frankfort–mandibular plane angle (FMA)). There were no significant differences in the above-mentioned dental indices between patients with high urine cadmium concentrations and those with low urine cadmium concentrations. Patients were further stratified into low (<27, n = 34), average (27–34, n = 23), and high (>34, n = 3) FMA groups. There were no statistically significant differences in the urine cadmium concentration among the three groups. Nevertheless, a marginally significant p-value of 0.05 for urine cadmium concentration was noted between patients with low FMA and patients with high FMA. Conclusion. This analysis found no association between environmental cadmium exposure and dental indices in our orthodontic patients.


Genetics ◽  
1999 ◽  
Vol 153 (2) ◽  
pp. 621-641 ◽  
Author(s):  
Dawn A Thompson ◽  
Franklin W Stahl

AbstractMeiotic exchange occurs preferentially between homologous chromatids, in contrast to mitotic recombination, which occurs primarily between sister chromatids. To identify functions that direct meiotic recombination events to homologues, we screened for mutants exhibiting an increase in meiotic unequal sister-chromatid recombination (SCR). The msc (meiotic sister-chromatid recombination) mutants were quantified in spo13 meiosis with respect to meiotic unequal SCR frequency, disome segregation pattern, sporulation frequency, and spore viability. Analysis of the msc mutants according to these criteria defines three classes. Mutants with a class I phenotype identified new alleles of the meiosis-specific genes RED1 and MEK1, the DNA damage checkpoint genes RAD24 and MEC3, and a previously unknown gene, MSC6. The genes RED1, MEK1, RAD24, RAD17, and MEC1 are required for meiotic prophase arrest induced by a dmc1 mutation, which defines a meiotic recombination checkpoint. Meiotic unequal SCR was also elevated in a rad17 mutant. Our observation that meiotic unequal SCR is elevated in meiotic recombination checkpoint mutants suggests that, in addition to their proposed monitoring function, these checkpoint genes function to direct meiotic recombination events to homologues. The mutants in class II, including a dmc1 mutant, confer a dominant meiotic lethal phenotype in diploid SPO13 meiosis in our strain background, and they identify alleles of UBR1, INP52, BUD3, PET122, ELA1, and MSC1-MSC3. These results suggest that DMC1 functions to bias the repair of meiosis-specific double-strand breaks to homologues. We hypothesize that the genes identified by the class II mutants function in or are regulators of the DMC1-promoted interhomologue recombination pathway. Class III mutants may be elevated for rates of both SCR and homologue exchange.


2021 ◽  
Vol 1943 (1) ◽  
pp. 012091
Author(s):  
M Subono ◽  
I R N Alima ◽  
E I Auerkari
Keyword(s):  
Class Ii ◽  

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