scholarly journals SENSORI-MOTOR RECOVERY IN POST-STROKE SHOULDER SUBLUXATION: A NON INVASIVE CORRELATION STUDY

2020 ◽  
Vol 8 (2.1) ◽  
pp. 7418-7423
Author(s):  
Ushnish Mukherjee ◽  
◽  
Swapan Kumar Mishra ◽  
Prabir Mandal ◽  
Subhra Mandal ◽  
...  
2019 ◽  
Vol 30 (8) ◽  
pp. 821-837 ◽  
Author(s):  
Sharon Israely ◽  
Gerry Leisman

Abstract Individuals post-stroke sustain motor deficits years after the stroke. Despite recent advancements in the applications of non-invasive brain stimulation techniques and Deep Brain Stimulation in humans, there is a lack of evidence supporting their use for rehabilitation after brain lesions. Non-invasive brain stimulation is already in use for treating motor deficits in individuals with Parkinson’s disease and post-stroke. Deep Brain Stimulation has become an established treatment for individuals with movement disorders, such as Parkinson’s disease, essential tremor, epilepsy, cerebral palsy and dystonia. It has also been utilized for the treatment of Tourette’s syndrome, Alzheimer’s disease and neuropsychiatric conditions such as obsessive-compulsive disorder, major depression and anorexia nervosa. There exists growing scientific knowledge from animal studies supporting the use of Deep Brain Stimulation to enhance motor recovery after brain damage. Nevertheless, these results are currently not applicable to humans. This review details the current literature supporting the use of these techniques to enhance motor recovery, both from human and animal studies, aiming to encourage development in this domain.


2012 ◽  
Vol 55 ◽  
pp. e151-e152
Author(s):  
I. Loubinoux ◽  
L. Vaysse ◽  
A. Béduer ◽  
F. Seichepine ◽  
E. Flahaut ◽  
...  

Author(s):  
Ushnish Mukherjee ◽  
Sourav Kundu ◽  
Rachit Gulati ◽  
Pankaj Kumar Mandal

Background: The prevalence of pain in affected shoulder among post-stroke patients ranges from 34% to 84%. Numerous theories exist to explain the patho-mechanics behind development of Post-stroke shoulder pain, but its relationship with the sensori-motor recovery of the affected limb is still controversial. This study was conducted to detect the correlation, if any, between post-stroke shoulder pain and sensori-motor recovery of the affected upper limb.  Methods: This observational longitudinal study was conducted on 73 patients of both sexes within the age group of 45-65 years having presentation of post-stroke (duration<6weeks) shoulder pain. Pain intensity was recorded in numerical rating scale (NRS). Sensorimotor recovery of the affected limb was assessed by Fugl- Meyer assessment scale of upper extremity (FMA-UE). Data were collected at the baseline (visit1), at 6 weeks (visit 2), 12 weeks (visit 3) and at the end of the study i.e., 24 weeks (visit 4).  Results: Statistically significant negative correlations were found between severity of pain (assessed with NRS) and sensory-motor recovery (assessed with FMA-UE) on each visit with correlation coefficients (Spearman rho, r) being r=-0.890, p=0.000 on visit1, r=-0.685, p=0.000 on visit2, r=-0.629, p=0.000 on visit3 and r=-0.458, p=0.000 on visit 4.Conclusions: Post-stroke shoulder pain plays a significant negative role in sensori-motor recovery of the affected upper limb requiring early intervention.


2019 ◽  
Vol 68 ◽  
pp. 6-14 ◽  
Author(s):  
Sarah A. Roelker ◽  
Mark G. Bowden ◽  
Steven A. Kautz ◽  
Richard R. Neptune

Stroke ◽  
2020 ◽  
Vol 51 (Suppl_1) ◽  
Author(s):  
David J Lin ◽  
Alison M Cloutier ◽  
Kimberly S Erler ◽  
Jessica M Cassidy ◽  
Samuel B Snider ◽  
...  

Introduction: Injury to the corticospinal tract (CST) has been shown to have a major effect on upper extremity motor recovery after stroke. This study aimed to examine how well CST injury, measured from neuroimaging acquired during the acute stroke workup, predicts upper extremity motor recovery. Methods: Patients (N = 48) with upper extremity weakness after ischemic stroke were assessed using the upper extremity Fugl-Meyer (FM) during the acute stroke hospitalization and again at 3-month follow-up. CST injury was quantified and compared, using four different methods, from images obtained as part of the stroke standard-of-care workup. Logistic and linear regression were performed using CST injury to predict delta FM. Injury to primary motor and premotor cortices were included as potential modifiers of the effect of CST injury on recovery. Results: 48 patients were enrolled 4.2 ± 2.7 days post-stroke and completed this study. CST injury distinguished patients who reached their recovery potential (as predicted from initial impairment) from those who did not, with AUC values ranging from 0.75 to 0.8. In addition, CST injury explained ~20% of the variance in the magnitude of upper extremity recovery, even after controlling for the severity of initial impairment. Results were consistent when comparing four different methods of measuring CST injury. Extent of injury to primary motor and premotor cortices did not significantly influence the predictive value that CST injury had for recovery. Conclusions: Structural injury to the CST, as estimated from standard-of-care imaging available during the acute stroke hospitalization, is a robust way to distinguish patients who achieve their predicted recovery potential and explains a significant amount of the variance in post-stroke upper extremity motor recovery.


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