On the issue of the genetic origins of the Yakut epic formation

2021 ◽  
pp. 160-169
Author(s):  
Vasiliy N. Ivanov ◽  
◽  
Antonina F. Koriakina ◽  
Ruslan N. Anisimov
Keyword(s):  
Genetic Origins

Language in mental retardation: Individual and syndromic differences, and neurogenetic variation 1Based on a keynote presentation at the Third European Conference on Psychological Theory and Research in Mental Retardation, Geneva, September 1st, 2000.

2001 ◽  
Vol 60 (3) ◽  
pp. 161-178 ◽  
Author(s):  
Jean A. Rondal
Keyword(s):  
Mental Retardation ◽  
Language Development ◽  
Oral Language ◽  
Psychological Theory ◽  
Genetic Syndromes ◽  
Explanatory Variables ◽  
Keynote Presentation ◽  
Genetic Origins ◽  
The Sixties ◽  
The Individual

Predominantly non-etiological conceptions have dominated the field of mental retardation (MR) since the discovery of the genetic etiology of Down syndrome (DS) in the sixties. However, contemporary approaches are becoming more etiologically oriented. Important differences across MR syndromes of genetic origin are being documented, particularly in the cognition and language domains, differences not explicable in terms of psychometric level, motivation, or other dimensions. This paper highlights the major difficulties observed in the oral language development of individuals with genetic syndromes of mental retardation. The extent of inter- and within-syndrome variability are evaluated. Possible brain underpinnings of the behavioural differences are envisaged. Cases of atypically favourable language development in MR individuals are also summarized and explanatory variables discussed. It is suggested that differences in brain architectures, originating in neurological development and having genetic origins, may largely explain the syndromic as well as the individual within-syndrome variability documented. Lastly, the major implications of the above points for current debates about modularity and developmental connectionism are spelt out.

Individual and Societal Well-Being

2018 ◽  
pp. 698-722
Author(s):  
Shigehiro Oishi ◽  
Samantha J. Heintzleman
Keyword(s):  
Social Psychology ◽  
Cognitive Processes ◽  
Well Being ◽  
Life Outcomes ◽  
Cultural Meanings ◽  
Research Topics ◽  
Psychological Science ◽  
The Social ◽  
Genetic Origins

This chapter highlights the contributions that have been made by personality and social psychology, respectively and together, to the science of well-being. Since its humble beginning in the 1930s, the science of well-being has grown to become one of the most vibrant research topics in psychological science today. The personality tradition of well-being research has shown that it is possible to measure well-being reliably, that self-reported well-being predicts important life outcomes, and that well-being has nontrivial genetic origins. The social psychology tradition has illuminated that there are various cultural meanings of well-being, that responses to well-being questions involve multiple cognitive processes, that happiness is experienced often in relationship contexts, and that it is possible to improve one’s well-being. Finally, there are recent methodological integrations of the personality and social psychology perspectives that delineate person–situation interactions.

scholarly journals Large-scale sequencing of flatfish genomes provides insights into the polyphyletic origin of their specialized body plan

2021 ◽  
Author(s):  
Zhenming Lü ◽  
Li Gong ◽  
Yandong Ren ◽  
Yongjiu Chen ◽  
Zhongkai Wang ◽  
...  
Keyword(s):  
Retinoic Acid ◽  
Large Scale ◽  
Body Plan ◽  
Monophyletic Group ◽  
Transcriptomic Data ◽  
Flatfish Species ◽  
Genetic Origins

AbstractThe evolutionary and genetic origins of the specialized body plan of flatfish are largely unclear. We analyzed the genomes of 11 flatfish species representing 9 of the 14 Pleuronectiforme families and conclude that Pleuronectoidei and Psettodoidei do not form a monophyletic group, suggesting independent origins from different percoid ancestors. Genomic and transcriptomic data indicate that genes related to WNT and retinoic acid pathways, hampered musculature and reduced lipids might have functioned in the evolution of the specialized body plan of Pleuronectoidei. Evolution of Psettodoidei involved similar but not identical genes. Our work provides valuable resources and insights for understanding the genetic origins of the unusual body plan of flatfishes.

Co-occurrence of ADHD and low IQ has genetic origins

2003 ◽  
Vol 124B (1) ◽  
pp. 41-47 ◽  
Author(s):  
J. Kuntsi ◽  
T.C. Eley ◽  
A. Taylor ◽  
C. Hughes ◽  
P. Asherson ◽  
...  
Keyword(s):  
Genetic Origins ◽  
Low Iq

scholarly journals Genetic analysis of self-associating immunoglobulin G rheumatoid factors from two rheumatoid synovia implicates an antigen-driven response.

1992 ◽  
Vol 175 (3) ◽  
pp. 831-842 ◽  
Author(s):  
T Olee ◽  
E W Lu ◽  
D F Huang ◽  
R W Soto-Gil ◽  
M Deftos ◽  
...  
Keyword(s):  
Amino Acid ◽  
Heavy Chain ◽  
Immunoglobulin M ◽  
Natural Antibody ◽  
Antibody Repertoire ◽  
Rheumatoid Factors ◽  
Variable Regions ◽  
Genetic Origins ◽  
V Genes ◽  
Vh Gene

Although much has been learned about the molecular basis of immunoglobulin M (IgM) rheumatoid factors (RFs) in healthy individuals and in patients with mixed cryoglobulinemia and rheumatoid arthritis, little is known about the genetic origins of the potentially pathogenic IgG RFs in the inflamed rheumatoid synovia of patients. Recently, we generated from unmanipulated synovium B cells several hybridomas that secreted self-associating IgG RFs. To delineate the genetic origins of such potentially pathogenic RFs, we adapted the anchored polymerase chain reaction to rapidly clone and characterize the expressed Ig V genes for the L1 and the D1 IgG RFs. Then, we identified the germline counterparts of the expressed L1 IgG RF V genes. The results showed that the L1 heavy chain was encoded by a Vh gene that is expressed preferentially during early ontogenic development, and that is probably located within 240 kb upstream of the Jh locus. The overlap between this RF Vh gene and the restricted fetal antibody repertoire is reminiscent of the natural antibody-associated Vh genes, and suggests that at least part of the "potential pathogenic" IgG RFs in rheumatoid synovium may derive from the "physiological" natural antibody repertoire in a normal immune system. Indeed, the corresponding germline Vh gene for L1 encodes the heavy chain of an IgM RF found in a 19-wk-old fetal spleen. Furthermore, the comparisons of the expressed RF V genes and their germline counterparts reveal that the L1 heavy and light chain variable regions had, respectively, 16 and 7 somatic mutations, which resulted in eight and four amino acid changes. Strikingly, all eight mutations in the complementarity determining regions of the V gene-encoded regions were replacement changes, while only 6 of 11 mutations in the framework regions caused amino acid changes. Combined with L1's high binding affinity toward the Fc fragment, these results suggest strongly that the L1 IgG RF must have been driven by the Fc antigen.

scholarly journals Transfer RNAs of potato (Solanum tuberosum) mitochondria have different genetic origins

1990 ◽  
Vol 18 (13) ◽  
pp. 3689-3696 ◽  
Author(s):  
Laurence Marechal-Drouard ◽  
Pierre Guillemaut ◽  
Anne Cosset ◽  
Michèle Arbogast ◽  
Frédérique Weber ◽  
...  
Keyword(s):  
Solanum Tuberosum ◽  
Transfer Rnas ◽  
Genetic Origins

The Right to Know Genetic Origins: A Harmful Value

2014 ◽  
Vol 44 (4) ◽  
pp. 5-6 ◽  
Author(s):  
Kimberly Leighton
Keyword(s):  
Right To Know ◽  
The Right To Know ◽  
Genetic Origins ◽  
The Right

scholarly journals Rereading of the genetic origins of cancer: the puzzle of all eras

2019 ◽  
Author(s):  
Berjas Abumsimir ◽  
Yassine Kasmi ◽  
Moulay Mustapha Ennaji
Keyword(s):  
Driving Force ◽  
Development Process ◽  
Evolution Process ◽  
Biological Phenomenon ◽  
Genetic Origins ◽  
Genetic Phenomenon

This is a scientific introduction to review the concept of cancer from a purely genetic perspective in which we try to link the development of tumors by the driving force of the evolution of living beings: the mutations. And to prove the assumption that cancer diversity has a parallel route instead of the whole development process of the cell. Calling for expanding the rolling concept of cancer and tumors definition in general. Which may contribute to altering the angle of our vision to this dangerous biological phenomenon. This call is based on the hypothesis that cancer is a genetic phenomenon that has arisen on the margins of the evolution process, that has continued since the beginning of creation. The pieces of evidence and indications that support this view were presented successively.

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