scholarly journals Dr. A. I. Yushchenko. The relationship of the lower mesenteric sympathetic node to the innervation of the bladder and the volume of automatic movements of the last day. - Archive of Biological Sciences, T. VI, issue 5

2020 ◽  
Vol VI (4) ◽  
pp. 213-214
Author(s):  
B. I. Vorotynskiy
Keyword(s):  
Biological Sciences ◽  
Nervous System ◽  
Sympathetic Nervous System ◽  
Morphological Properties ◽  
The Central Nervous System ◽  
Sympathetic Nervous ◽  
The Right ◽  
Relationship Of ◽  
The Relationship ◽  
The Brain

Physiology has not yet given us positive data on the function of the sympathetic nervous system. Only recently, histologists have been able to prove that the nerve cells located in large sympathetic nodes do not differ in their morphological properties from cells of the central nervous system. This gives the right to assume that in the sympathetic nodes there are independent centers that are in the same dependence on the higher centers, as, for example, the centers of the spinal cord from the brain. We have very little data on the relationship of the sympathetic nervous system to the innervations of the bladder. On this basis, the author began to study this issue in the physiological laboratory of prof. Pavlova at the M. Academy.

L-Dopa metabolism in genetically hypertensive mice: effect of pargyline

1987 ◽  
Vol 65 (12) ◽  
pp. 2390-2395 ◽  
Author(s):  
Nguyen T. Buu ◽  
Johanne Duhaime ◽  
Karoly Racz ◽  
Otto Kuchel ◽  
Gunther Schlager
Keyword(s):  
Nervous System ◽  
Sympathetic Nervous System ◽  
Monoamine Oxidase ◽  
Hydroxylase Activity ◽  
Sympathetic Nervous ◽  
The Relationship ◽  
The Brain ◽  
Genetically Hypertensive

This study on the role of the sympathetic nervous system in the development of hypertension involves the measurement of dopamine and norepinephrine accumulation in various tissues of the hypertensive and random-bred normotensive strains of mice at basal levels, and following a pargyline–L-dopa treatment. Under such a treatment, designed to suppress the homeostatic action of monoamine oxidase and to better expose the relationship between dopamine and norepinephrine, the brain and heart of the hypertensive mice accumulated more dopamine than the normotensive mice. There was a significantly lower norepinephrine accumulation in the heart of the hypertensive mice in spite of comparable dopamine-β-hydroxylase activity in this tissue between the two strains of mice. Under the pargyline–L-dopa treatment, the brain and heart of the older mice in both hypertensive and normotensive strains accumulated significantly (p < 0.05) more dopamine than those of their younger counterparts, while their norepinephrine accumulation remained unchanged. The results demonstrated different patterns of response of dopamine and norepinephrine in the development of hypertension.

FLUID, ELECTROLYTE, GASTROINTESTINAL, AND HAIR CHANGES IN ADRENALECTOMIZED-SYMPATHECTOMIZED DOGS

1957 ◽  
Vol 35 (1) ◽  
pp. 983-992
Author(s):  
R. A. Cleghorn ◽  
J. L. A. Fowler
Keyword(s):  
Nervous System ◽  
Gastrointestinal Tract ◽  
Sympathetic Nervous System ◽  
Adrenal Insufficiency ◽  
Adrenal Cortex ◽  
The Other ◽  
Pathological Changes ◽  
Sympathetic Nervous ◽  
Relationship Of ◽  
The Relationship

Five male dogs were adrenalectomized and sympathectomized and maintained by adrenocortical extract for periods of weeks to months. Their general behavior did not differ from those subject to adrenalectomy alone, but in three it was observed that the hair grew long and curly. In the other two a lesser change may have been missed. Following withdrawal of the hormone, death occurred rather suddenly in three, and adrenal insufficiency developed sooner in the other two than in dogs only adrenalectomized. Pathological changes in the gastrointestinal tract were qualitatively the same following death after hormone withdrawal in the adrenalectomized–sympathectomized animals and in dogs only adrenalectomized. Blood changes appeared to be no different in the two groups. The urinary changes with respect to water and electrolytes differed in that the sympathectomized group appeared to take in less water and excrete less urine, but to put out more sodium and chloride, in the few days following withdrawal of hormone. These findings are discussed in particular with respect to the relationship of adrenergic function of the sympathetic nervous system and hormones of the adrenal cortex.

scholarly journals The Brain-Heart Connection in Takotsubo Syndrome: The Central Nervous System, Sympathetic Nervous System, and Catecholamine Overload

2020 ◽  
Vol 2020 ◽  
pp. 1-5 ◽  
Author(s):  
Xiaopu Wang ◽  
Junyu Pei ◽  
Xinqun Hu
Keyword(s):  
Nervous System ◽  
Sympathetic Nervous System ◽  
Physiological Stress ◽  
Acute Chest Pain ◽  
Takotsubo Syndrome ◽  
The Central Nervous System ◽  
Sympathetic Nervous ◽  
As Stress ◽  
Artery Obstruction ◽  
The Brain

Takotsubo syndrome (TTS), also known as stress cardiomyopathy, is a type of acute heart failure syndrome triggered by intense psychological or physiological stress. TTS typically manifests as acute chest pain, dyspnea or syncope that mimics an acute myocardial infarction but does not involve coronary artery obstruction. The current understanding of the pathogenesis of TTS suggests that sympathetic nervous system (SNS) activation plays a central role. Specifically, stress can activate the SNS and lead to the over-release of catecholamine, which have toxic effects on myocardial tissue when present at excessive levels. However, the brain changes associated with TTS and the connection between the brain and the heart in patients with this disease remain unclear. In recent years, several published reports have revealed the role of this brain-heart connection in the pathogenesis of TTS. This review summarizes recent studies regarding SNS activation, catecholamine overload, and the brain-heart connection in patients with TTS from both pathophysiological and mechanistic aspects.

FLUID, ELECTROLYTE, GASTROINTESTINAL, AND HAIR CHANGES IN ADRENALECTOMIZED-SYMPATHECTOMIZED DOGS

1957 ◽  
Vol 35 (11) ◽  
pp. 983-992
Author(s):  
R. A. Cleghorn ◽  
J. L. A. Fowler
Keyword(s):  
Nervous System ◽  
Gastrointestinal Tract ◽  
Sympathetic Nervous System ◽  
Adrenal Insufficiency ◽  
Adrenal Cortex ◽  
The Other ◽  
Pathological Changes ◽  
Sympathetic Nervous ◽  
Relationship Of ◽  
The Relationship

Five male dogs were adrenalectomized and sympathectomized and maintained by adrenocortical extract for periods of weeks to months. Their general behavior did not differ from those subject to adrenalectomy alone, but in three it was observed that the hair grew long and curly. In the other two a lesser change may have been missed. Following withdrawal of the hormone, death occurred rather suddenly in three, and adrenal insufficiency developed sooner in the other two than in dogs only adrenalectomized. Pathological changes in the gastrointestinal tract were qualitatively the same following death after hormone withdrawal in the adrenalectomized–sympathectomized animals and in dogs only adrenalectomized. Blood changes appeared to be no different in the two groups. The urinary changes with respect to water and electrolytes differed in that the sympathectomized group appeared to take in less water and excrete less urine, but to put out more sodium and chloride, in the few days following withdrawal of hormone. These findings are discussed in particular with respect to the relationship of adrenergic function of the sympathetic nervous system and hormones of the adrenal cortex.

On humoral factors in nervous activity

1935 ◽  
Vol 31 (6) ◽  
pp. 777-787
Author(s):  
D. S. Vorontsov
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Nervous Activity ◽  
Active Part ◽  
Humoral Factors ◽  
The Central Nervous System ◽  
Relationship Of ◽  
The Relationship

Not only in the peripheral working organs, irritating substances are formed, which, as we can see, take an active part in their regulation, but also in the central nervous system, in the relationship of its individual elements, such substances apparently play an important role.

Intake of Anti-Epileptic Drugs and their Influences on Sexual Dysfunctions

2021 ◽  
Vol 3 (2) ◽  
Author(s):  
Roheela Yasmeen ◽  
Nida Mobeen ◽  
Muhammad Amjad Khan ◽  
Irfan Aslam ◽  
Samia Chaudhry
Keyword(s):  
Quality Of Life ◽  
Sexual Dysfunction ◽  
Neurological Disorders ◽  
Penile Erection ◽  
The Central Nervous System ◽  
Relationship Of ◽  
The Relationship ◽  
The Brain ◽  
Psychiatric Problems

Epilepsy which is also called seizures disorder is an uncontrolled action of the central nervous system. Itis not a single disease but a set of neurological disorders. Actually in this situation, the brain does notreceive a precise signal and as a result an abnormal condition is produced that is usually involuntary inaction. In this review, we aimed to focus on the relationship of anti-epileptic drugs with sexual dysfunctionand adaptation of better remedies that improve a patient’s family life. Sexual dysfunction is a commoncomorbidity in people with epilepsy which badly affects their quality of life. Sexual dysfunction is causedby different factors like psychiatric problems, anti-epileptic drugs (AEDs) and social factors etc. Sexualdysfunctions include ejaculatory failure, lessen libido, penile erection in men and irregular menstrual cyclein women. Common drugs such as Topiramate, Gabapentin (GBP), Valproate (VA), Carbamazepine (CBZ),Olanzapine (OL) and Risperidone (RTG) that are in practice to treat epilepsy usually produced adverseeffect on sexual dysfunction. Even though a lot of studies have been carried out to control sexualdysfunction in epilepsy’s patient, but still research is going on. Medicine such as Cyproheptadine,Mianserin, Buspirone, Yohimbine were found better to treat epilepsy with minimum side effects of sexualdysfunction. Moreover, it is also seen that certain vasodilators, folate , and vitamin supplements areeffective in improving the quality of life.

Effect of voluntary vs. artificial activation on the relationship of muscle torque to speed

1990 ◽  
Vol 69 (6) ◽  
pp. 2215-2221 ◽  
Author(s):  
G. A. Dudley ◽  
R. T. Harris ◽  
M. R. Duvoisin ◽  
B. M. Hather ◽  
P. Buchanan
Keyword(s):  
Knee Extensor ◽  
Voluntary Activation ◽  
Knee Extensors ◽  
Angular Velocities ◽  
The Central Nervous System ◽  
The Right ◽  
Artificial Activation ◽  
Relationship Of ◽  
The Relationship ◽  
Eccentric Actions

The speed-torque relationship of the right knee extensor muscle group was investigated in eight untrained subjects (28 +/- 2 yr old). Torque was measured at a specific knee angle during isokinetic concentric or eccentric actions at nine angular velocities (0.17-3.66 rad/s) and during isometric actions. Activation was by "maximal" voluntary effort or by transcutaneous tetanic electrical stimulation that induced an isometric torque equal to 60% (STIM 1) or 45% (STIM 2) of the voluntary isometric value. Torque increased (P less than 0.05) to 1.4 times isometric as the speed of eccentric actions increased to 1.57 rad/s for STIM 1 and STIM 2. Thereafter, increases in eccentric speed did not further increase torque. Torque did not increase (P greater than 0.05) above isometric for voluntary eccentric actions. As the speed of concentric actions increased from 0.00 to 3.66 rad/s, torque decreased (P less than 0.05) more (P less than 0.05) for both STIM 1 and STIM 2 (two-thirds) than for voluntary activation (one-half). As a result of these responses, torque changed three times as much (P less than 0.05) across speeds of concentric and eccentric actions with artificial (3.4-fold) than voluntary (1.1-fold) activation. The results indicate that with artificial activation the normalized speed-torque relationship of the knee extensors in situ is remarkably similar to that of isolated muscle. The relationship for voluntary activation, in contrast, suggests that the ability of the central nervous system to activate the knee extensors during maximal efforts depends on the speed and type of muscle action performed.

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