scholarly journals A randomized split mouth clinical trial of the application of the desensitizer agents for tooth sensitivity

Author(s):  
K. Hanisha Reddy ◽  
Afroz Kalmee Syed ◽  
Dasarathi Alivelu ◽  
Haranath Danda ◽  
Ramya Alla

Background: Dentin hypersensitivity (DH) affects 3%-75% of the people and is one of the morbid tooth conditions. Hence in the present study we aim to examine the clinical effectiveness of 3 different desensitizing agents in decreasing pain of DH in time of 1 month.Methods: Fifty subjects with cervical DH in at least one tooth in any three of the 4 quadrants were selected. VAS was used to note the pain. Each quadrant in an individual was randomly assigned. Profluorid varnish, Admira protect, and PRG‑Barrier coat was used. VAS scores for the tactile and air stimuli were noted immediately after application, 1 week, and after 1 month. The data was analyzed keeping p<0.05 as significant.Results: VAS significantly reduced for all three groups from the base line (p<0.001). Admira protect showed significant reduction of hypersensitivity scores at 1 month compared to other groups (p<0.001).Conclusions: Admira protect was better at lowering the pain due to DH than PRG‑barrier coat and Profluorid varnish after 1 month of application.

2014 ◽  
Vol 39 (5) ◽  
pp. E186-E194 ◽  
Author(s):  
CRG Torres ◽  
TM Silva ◽  
BM Fonseca ◽  
ALLS Sales ◽  
P Holleben ◽  
...  

SUMMARY The aim of this study was to evaluate the efficacy of three desensitizing agents to provide relief to dentin hypersensitivity after one session in a four-week follow-up. Forty selected patients participated in a double-blind study following a split-mouth model. One application of the desensitizing agents (A, Admira Protect [Voco]; B, Bifluorid 12 [Voco]; and C, Colgate Pro-Relief in office [Colgate Palmolive]) was performed in three different quadrants for each patient. Each tooth was evaluated by tactile and evaporative stimuli, and the sensitivity response was measured using the Visual Analogue Scale. Evaluations were performed at baseline, immediately after treatment, and after one, two, three, and four weeks. The application of Kruskal-Wallis and Dunn multiple comparisons tests (5%) for both tactile and evaporative stimuli showed that all agents presented a significant desensitizing effect. In groups A and B this relief was maintained for four and three weeks, respectively, as measured by tactile stimulus and for four weeks with evaporative stimulus. The desensitizing effect for group C was maintained for two weeks for both tactile and evaporative stimuli. It is concluded that all desensitizing agents tested were effective in reducing sensitivity compared to baseline values. One application of Admira Protect and Bifluorid 12 presented a longer-lasting desensitizing effect than did Colgate Pro-Relief (applied in the office) on both tactile and evaporative stimuli.


2018 ◽  
Vol 29 (1) ◽  
pp. 51 ◽  
Author(s):  
Periasamy Ravishankar ◽  
Venkatnagaraj Viswanath ◽  
Durvasulu Archana ◽  
Venkatesan Keerthi ◽  
Saranya Dhanapal ◽  
...  

2019 ◽  
Vol 22 (6) ◽  
pp. 522
Author(s):  
AngambakkamRajasekaran PradeepKumar ◽  
Venkatnagaraj Viswanath ◽  
Kamna Singh ◽  
Kuzhanchinathan Manigandan ◽  
HaseenaBegum Iqbal ◽  
...  

2021 ◽  
Vol 3 (2) ◽  
pp. 69-72
Author(s):  
Nemakal Sumana ◽  
Chakravarthy YSHS ◽  
Ch Susmitha S S ◽  
Patcha Harika ◽  
Sravani Bontu ◽  
...  

Dentin hypersensitivity is one of the most common presenting symptoms in dental practice. It may range from mild discomfort to severe pain affecting the person. Laser desensitization has been introduced as a useful tool for the treatment of hypersensitivity. Gluma Dentin Bond is an adhesive system, where the primer contains 5% glutaraldehyde and 35% hydroxyethyl methacrylate. Practitioners have reported a strong desensitizing effect of the Gluma® system on dentin The aim of this study is to evaluate and compare the clinical effectiveness of Diode laser and Gluma ®desensitizing agent in the treatment of dentin hypersensitivity. 24 patients aged between 20 and 50 years was included to assess tooth sensitivity, a controlled air stimulus (evaporative stimulus) was used. Sensitivity was measured using a 10-cm Visual Aanalog Scale (VAS) score. The teeth was randomly allocated to two groups i.e., Group I or II using the lottery method. Gluma® showed a statistically significant reduction in the VAS score as compared to diode laser 1, 2- and 4-weeks follow-up period (p &#60; 0.05).  The result of the present investigation revealed that application of Gluma® resulted in better control dentin hypersensitivity as compared to diode laser.


2011 ◽  
Vol 22 (2) ◽  
pp. 157-161 ◽  
Author(s):  
Jorgiana Silva de Assis ◽  
Lidiany Karla Azevedo Rodrigues ◽  
Cristiane Sá Roriz Fonteles ◽  
Regina Cláudia Ramos Colares ◽  
André Mattos Brito de Souza ◽  
...  

The aim of this study was to evaluate the efficacy of two desensitizing agents in the reduction of dentin hypersensitivity in a randomized, double-blind, split-mouth clinical trial. Seventy-seven teeth from 13 patients that presented some degree of sensitivity to probing and/or air stimulation were treated with one of the following desensitizing agents: Oxa-Gel (G1), Sensi Kill (G2) and placebo gel (G3 - control). According to paired t-test, all treatments, even the placebo gel, were capable of reducing sensitivity scores for both stimuli. Analysis of data by ANOVA and Tukey's test (?=0.05) showed that the sensitivity scores were significantly lower only for Sensi Kill in comparison to the other products (Oxa-Gel and placebo), when air stimulus was applied. It may be concluded that treatment with Sensi Kill presented a slightly better performance in reducing dentin hypersensitivity when compared to the other desensitizing agent.


BMJ Open ◽  
2021 ◽  
Vol 11 (3) ◽  
pp. e044653
Author(s):  
Ana Paula Taboada Sobral ◽  
Elaine Marcilio Santos ◽  
Ana Cecilia Aranha ◽  
Paulo Vinícius Soares ◽  
Caroline Moraes Moriyama ◽  
...  

IntroductionDentin hypersensitivity (DH) is defined as high sensitivity of the vital dentin when exposed to thermal, chemical or tactile stimuli. Two mechanisms are required for the occurrence of DH: (1) the dentin must be exposed and (2) the dentinal tubules must be open and connected to the pulp. Molar–incisor hypomineralisation (MIH) is a qualitative abnormality of a genetic origin that affects tooth enamel and, in most cases, is accompanied by DH. The control of tooth sensitivity is fundamental to the successful treatment of MIH. The aim of the proposed randomised, controlled, clinical trial is to evaluate the effectiveness of different protocols for the control of DH in patients with teeth affected by MIH.Methods and analysisOne hundred and forty patients who meet the inclusion criteria will be allocated to four groups. Group 1 will be the control group (placebo). In Group 2, sensitive teeth will be sealed with PermaSeal (Ultradent). In Group 3, sensitive teeth will receive low-level laser (LLL, AsGaAl) at a wavelength of 780 nm (Laser XT Therapy, DMC, São Carlos, Brazil). In Group 4, sensitive teeth will be treated with both LLL and PermaSeal (Ultradent). DH will be evaluated 15 min after the application of the treatments and the patients will be reevaluated 1 week, 1 month, 3 months and 6 months after the treatments. The primary outcome of this study is change in pain/sensitivity, when evaluated through a Visual Analogue Scale, to determine the effectiveness of the proposed treatments, as well as differences among the evaluation times for each proposed treatment.Ethics and disseminationThis protocol has been ethically approved by the local medical ethical committee (protocol number: 4.020.261). Results will be submitted to international peer-reviewed journals and presented at international conferences.Trial registration numberNCT04407702.


10.2341/07-5 ◽  
2007 ◽  
Vol 32 (6) ◽  
pp. 544-548 ◽  
Author(s):  
T. Pamir ◽  
H. Dalgar ◽  
B. Onal

Clinical Relevance Three desensitizing agents with different active ingredients exhibited similar effects in the treatment of dentin hypersensitivity by mechanical blockage.


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