scholarly journals Risk Model of Air Pollution Exposure (NO2, SO2, TSP and Dust) and Smoking Habits To The Lung Function Of Bus Driver In Palembang City

2019 ◽  
Vol 13 (4) ◽  
pp. 204
Author(s):  
Elvi Sunarsih ◽  
Suheryanto Suheryanto ◽  
Rini Mutahar ◽  
Rahmi Garmini
Keyword(s):  
Air Pollution ◽  
Risk Model ◽  
Risk Estimation ◽  
Smoking Habit ◽  
Hazard Index ◽  
Mean Value ◽  
Cross Sectional ◽  
Short Period ◽  
Air Pollution Exposure ◽  
Pollution Exposure

AbstractNitrogen dioxide (NO2), carbon monoxide (CO), sulfur dioxide (SO2), Particulate Matter (PM10) and TSP are the most common and harmful air pollutants to humans. In short period, air pollution exposure at 5 ppm for 10 minutes to humans causes dyspnea, and when the level is increase to 800 ppm could cause 100% mortality in animals. This study was an analytical research, with cross sectional design and risk analysis. One hundred subjects were analyzed in this study. The results showed that non-cancer Hazard Index (HI) for realtime exposure with mean value was NO2: 1.85; SO2: 2.92; TSP: 7.09; and PM10: 11.7 (HI value ≥1). The results of the analysis test for FVC lung capacity to non-cancer risk estimation of NO2, SO2, TSP and PM10 indicated that there was no significant relationship (p > 0,05). Variable of smoking habit is the most dominant variable (OR = 12,542) which affect respiratory disorders. The exposure of NO2, SO2, TSP and PM10 in Palembang City bus drivers is considered risky to non-cancer health subjects, so control is needed. This control could be performed by lowering the level of inhalation intake (I) of the RfC and the first simulation model for exposure could start from the second year to the sixth year.  

scholarly journals Noncommunicable Respiratory Disease and Air Pollution Exposure in Malawi (CAPS). A Cross-Sectional Study

2019 ◽  
Vol 199 (5) ◽  
pp. 613-621 ◽  
Author(s):  
Rebecca Nightingale ◽  
Maia Lesosky ◽  
Graham Flitz ◽  
Sarah J. Rylance ◽  
Jamilah Meghji ◽  
...  
Keyword(s):  
Air Pollution ◽  
Respiratory Disease ◽  
Cross Sectional Study ◽  
Sectional Study ◽  
Cross Sectional ◽  
Air Pollution Exposure ◽  
Pollution Exposure

scholarly journals Factors influencing health behaviours in response to the air quality health index: a cross-sectional study in Hamilton, Canada

2016 ◽  
Vol 59 (1) ◽  
pp. 17-29 ◽  
Author(s):  
Sally Radisic ◽  
K. Bruce Newbold ◽  
John Eyles ◽  
Allison Williams
Keyword(s):  
Public Health ◽  
Air Pollution ◽  
Air Quality ◽  
Health Index ◽  
Cross Sectional ◽  
Factors Influencing ◽  
Air Pollution Exposure ◽  
Quality Health ◽  
Air Quality Health Index ◽  
Pollution Exposure

Research associating adverse health effects with air pollution exposure is robust. Public health authorities recognize the need to implement population health strategies that protect public health from air pollution exposure. The Air Quality Health Index (AQHI) is a public health initiative that is intended to protect the public's health from exposure to air pollution. The aim of this research was to identify and explain factors influencing AQHI adoption at the individual level and to establish intervention strategies. A cross-sectional survey with both quantitative and qualitative questions was administered in Hamilton, Ontario, Canada, during the months of June to October 2012. Logistic regression and the Health Belief Model are used to explore the data. Demographics (gender, age, education, and area of residence), knowledge/understanding, and individual risk perceptions (neighbourhood air effects on health) were found to be significant predictors of AQHI adoption. The perceived benefits of AQHI adoption included protection of health for self and those cared for via familial and (or) occupational duties, whereas the perceived barriers of AQHI adoption included lack of knowledge about where to check and lack of time required to check and follow AQHI health messages. Also, self-efficacy was uncovered as a factor influencing AQHI adoption. Accordingly, increases in AQHI adoption could be achieved via increasing AQHI knowledge among low socioeconomic status females, communicating the benefits of AQHI adoption to “at-risk” populations and implementing supports for males to follow AQHI health messages.

scholarly journals Potential Short-Term Air Pollution Effects on Rheumatoid Arthritis Activity in Metropolitan Areas in the North of Italy: A Cross-Sectional Study

2021 ◽  
Vol 18 (16) ◽  
pp. 8490
Author(s):  
Francesca Ingegnoli ◽  
Tania Ubiali ◽  
Tommaso Schioppo ◽  
Valentina Longo ◽  
Antonella Murgo ◽  
...  
Keyword(s):  
Air Pollution ◽  
Disease Activity ◽  
Cross Sectional Study ◽  
Short Term ◽  
Cross Sectional ◽  
Short Term Exposure ◽  
Air Pollution Exposure ◽  
Synthetic Dmards ◽  
Pollution Exposure ◽  
The Impact

Rheumatoid arthritis (RA) flare is related to increased joint damage, disability, and healthcare use. The impact of short-term air pollution exposure on RA disease activity is still a matter of debate. In this cross-sectional study, we investigated whether short-term exposure to particulate matter (PM)10, PM2.5, nitrogen dioxide (NO2), and ozone (O3) affected RA disease activity (DAS28 and SDAI) in 422 consecutive RA residents in Lombardy, North of Italy. Air pollutant concentrations, estimated by Regional Environmental Protection Agency (Lombardy—Italy) at the municipality level, were used to assign short-term exposure from the day of enrolment, back to seven days. Some significant negative associations emerged between RA disease activity, PM10, and NO2, whereas some positive associations were observed for O3. Patients were also stratified according to their ongoing Disease-Modifying anti-Rheumatic Drugs (DMARDs) treatment: no DMARDs (n = 25), conventional synthetic DMARDs (n = 108), and biological or targeted synthetic DMARDs (n = 289). Therapy interaction seemed partially able to influence the relationship between short-term air pollution exposure and RA disease activity (PM2.5 levels and DAS28 at the day of the visit-O3 levels and disease activity scores for the seven days before the evaluation). According to our results, the impact of short-term air pollution exposure (seven days) minimally impacts disease activity. Moreover, our study suggests therapy could alter the response to environmental factors. Further evidence is needed to elucidate determinants of RA flare and its management.

scholarly journals Air pollution exposure and lung function until age 16 years: the PIAMA birth cohort study

2018 ◽  
Vol 52 (3) ◽  
pp. 1800218 ◽  
Author(s):  
Edith B. Milanzi ◽  
Gerard H. Koppelman ◽  
Henriette A. Smit ◽  
Alet H. Wijga ◽  
Marieke Oldenwening ◽  
...  
Keyword(s):  
Air Pollution ◽  
Lung Function ◽  
Secondary School ◽  
Time Windows ◽  
Forced Expiratory Volume ◽  
Birth Cohort Study ◽  
Cross Sectional ◽  
School Time ◽  
Air Pollution Exposure ◽  
Pollution Exposure

Evidence for the effects of air pollution exposure on lung function growth into adolescence is scarce. We investigated associations of air pollution exposure with lung function and lung function growth until age 16.We conducted both longitudinal (n=915) and cross-sectional (n=721) analyses of associations of air pollution exposure with forced expiratory volume in 1 s (FEV1) and forced vital capacity (FVC) growth from ages eight to 16 and FEV1 and FVC at age 16. We estimated residential concentrations of nitrogen dioxide (NO2), “soot” and particulate matter (PMx, where x is the 50% cut-off aerodynamic diameter in µm) with diameters of <2.5 µm (PM2.5), <10 µm (PM10) and 2.5–10 µm (PMcoarse) during the preschool, primary school and secondary school time windows by land use regression models. Associations with (growth in) FEV1 and FVC were analysed by linear (mixed effects) regression.Higher air pollution exposure was associated with reduced FEV1 growth (e.g. adjusted difference −0.26% (95% CI −0.49 to −0.03%) per interquartile range increase in secondary school PM2.5) and lower FEV1 (adjusted difference −2.36% (95% CI −3.76 to −0.94%)), but was not adversely associated with FVC. Associations with FEV1 were stronger in boys than girls and were not modified by asthma status.Higher air pollution exposure may lead to increased airway obstruction, but not reduced lung volume in adolescence.

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