scholarly journals The Role of Eye-Movements in Eye-Movement Desensitization and Reprocessing: Eye-Movements Lower the Number of Intrusive Thoughts of Negative Memories

2021 ◽  
Author(s):  
◽  
Gauranga Jeram Patel

<p>Eye-Movement Desensitization and Reprocessing (EMDR) was developed as a treatment for Post-Traumatic Stress Disorder (PTSD), and involves the patient thinking about a traumatic event while simultaneously moving their eyes from side to side. Despite substantial support for the efficacy of EMDR questions remain regarding how eye-movements contribute to therapy. One explanation is that eye-movements tax a part of working memory known as the central executive; however, the exact mechanism involved is still unclear. Previous eye-movement research has focussed on self-ratings of vividness and emotionality of negative memories as the primary outcome measures. The focus of the current research was to examine the effect of eye-movements on the suppression of negative autobiographical memories in addition to vividness and emotionality. Non-clinical participants were asked to recall negative autobiographical memories and then verbally reported ratings of vividness and emotionality. In the eye-movement conditions, which varied by speed and direction of movement, eye-movements were stimulated using dots on a computer screen. Participants were then asked to avoid thinking of their memories, and intrusive thoughts were measured by pressing a computer key. Six experiments found that, overall, the effect of eye-movements on self-ratings was inconsistent, but that eye-movements reliably improved suppression of negative autobiographical memories. The findings also support the central executive explanation for the effectiveness of eye-movements in EMDR.</p>

2021 ◽  
Author(s):  
◽  
Gauranga Jeram Patel

<p>Eye-Movement Desensitization and Reprocessing (EMDR) was developed as a treatment for Post-Traumatic Stress Disorder (PTSD), and involves the patient thinking about a traumatic event while simultaneously moving their eyes from side to side. Despite substantial support for the efficacy of EMDR questions remain regarding how eye-movements contribute to therapy. One explanation is that eye-movements tax a part of working memory known as the central executive; however, the exact mechanism involved is still unclear. Previous eye-movement research has focussed on self-ratings of vividness and emotionality of negative memories as the primary outcome measures. The focus of the current research was to examine the effect of eye-movements on the suppression of negative autobiographical memories in addition to vividness and emotionality. Non-clinical participants were asked to recall negative autobiographical memories and then verbally reported ratings of vividness and emotionality. In the eye-movement conditions, which varied by speed and direction of movement, eye-movements were stimulated using dots on a computer screen. Participants were then asked to avoid thinking of their memories, and intrusive thoughts were measured by pressing a computer key. Six experiments found that, overall, the effect of eye-movements on self-ratings was inconsistent, but that eye-movements reliably improved suppression of negative autobiographical memories. The findings also support the central executive explanation for the effectiveness of eye-movements in EMDR.</p>


2016 ◽  
Vol 10 (1) ◽  
pp. 13-22 ◽  
Author(s):  
Gauranga Jeram Patel ◽  
John McDowall

In dismantling eye movement desensitization and reprocessing (EMDR) therapy, researchers have found that the central executive is likely responsible for the effect of eye movements on negative memories. Arguably, however, researchers have not satisfactorily explained central executive mechanisms responsible. One possible central executive mechanism is that of suppression. The aim of this research was to evaluate the effect of eye movements on vividness, emotionality, and suppression of memories. Thirty-one nonclinical participants in Experiment 1 completed fast- and no-eye-movement conditions. Thirty-three nonclinical participants in Experiment 2 completed fast-, slow-, and no-eye-movement conditions. Number of intrusions during a suppression period and self-ratings of vividness and emotionality were the dependent variables in both experiments. Experiment 2 also included a measure of central executive capacity. Results from both experiments supported the hypotheses and showed that fast eye movements resulted in fewer intrusions than no- and slow-eye-movement conditions. Experiment 2 also found a correlation between number of intrusions after fast eye movements and central executive capacity. Limitations of this research are discussed as well as possibilities for future research and implications for understanding EMDR therapy.


Healthcare ◽  
2022 ◽  
Vol 10 (1) ◽  
pp. 133
Author(s):  
Nicolae Goga ◽  
Costin-Anton Boiangiu ◽  
Andrei Vasilateanu ◽  
Alexandru-Filip Popovici ◽  
Marius-Valentin Drăgoi ◽  
...  

In this paper, we describe an actuator-based EMDR (eye movement desensitization and reprocessing) virtual assistant system that can be used for the treatment of participants with traumatic memories. EMDR is a psychological therapy designed to treat emotional distress caused by a traumatic event from the past, most frequently in post-traumatic stress disorder treatment. We implemented a system based on video, tactile, and audio actuators which includes an artificial intelligence chatbot, making the system capable of acting autonomously. We tested the system on a sample of 31 participants. Our results showed the efficiency of the EMDR virtual assistant system in reducing anxiety, distress, and negative cognitions and emotions associated with the traumatic memory. There are no such systems reported in the existing literature. Through the present research, we fill this gap by describing a system that can be used by patients with traumatic memories.


1999 ◽  
Vol 5 (2) ◽  
pp. 120-125 ◽  
Author(s):  
M. J. MacCulloch

Eye movement desensitisation and reprocessing (EMDR) was described by Shapiro (1989a,b) as a new method for treating post-traumatic stress disorder (PTSD). In May 1987, while walking in the park, Shapiro noticed that her own disturbing thoughts changed then disappeared “without any conscious effort” (Shapiro, 1995) when they had been temporally paired with diagonal upward to and fro eye movements. Over the next six months Shapiro worked with approximately 70 people to develop a procedure based on the temporal pairing of distressing images and thoughts with various eye movements. Shapiro began to develop strategies to unblock stalled emotional processing, which was initiated by EMDR in non-patients. She successfully tried the method on a Vietnam veteran suffering from severe PTSD and then embarked upon a trial of EMDR on a mixed group of victims of rape, molestation and Vietnam combat trauma. Initially, EMDR achieved wide recognition as a new breakthrough treatment for PTSD. This was, in part, because of very positive early reports (e.g. Wolpe & Abrams, 1991), but also because the EMDR effect appeared to occur with unprecedented speed, often in cases of PTSD that had previously resisted treatment by many other methods over a long period.


2009 ◽  
Vol 40 (7) ◽  
pp. 1215-1223 ◽  
Author(s):  
A. Liedl ◽  
M. O'Donnell ◽  
M. Creamer ◽  
D. Silove ◽  
A. McFarlane ◽  
...  

BackgroundPain and post-traumatic stress disorder (PTSD) are frequently co-morbid in the aftermath of a traumatic event. Although several models attempt to explain the relationship between these two disorders, the mechanisms underlying the relationship remain unclear. The aim of this study was to investigate the relationship between each PTSD symptom cluster and pain over the course of post-traumatic adjustment.MethodIn a longitudinal study, injury patients (n=824) were assessed within 1 week post-injury, and then at 3 and 12 months. Pain was measured using a 100-mm Visual Analogue Scale (VAS). PTSD symptoms were assessed using the Clinician-Administered PTSD Scale (CAPS). Structural equation modelling (SEM) was used to identify causal relationships between pain and PTSD.ResultsIn a saturated model we found that the relationship between acute pain and 12-month pain was mediated by arousal symptoms at 3 months. We also found that the relationship between baseline arousal and re-experiencing symptoms, and later 12-month arousal and re-experiencing symptoms, was mediated by 3-month pain levels. The final model showed a good fit [χ2=16.97, df=12, p>0.05, Comparative Fit Index (CFI)=0.999, root mean square error of approximation (RMSEA)=0.022].ConclusionsThese findings provide evidence of mutual maintenance between pain and PTSD.


2019 ◽  
Vol 6 (3) ◽  
pp. 299-308
Author(s):  
John E Buonora ◽  
Patrick M Krum ◽  
Tomás Eduardo Ceremuga

Post-traumatic stress disorder (PTSD) continues to be one of the most common mental health disorders in the United States and may occur in response to traumatic experiences. Currently, there are no interventions that prevent the development of PTSD. L-Theanine (L-Th), a major compound in green tea has been found to decrease anxiety and prevent memory impairment and may have potential effects in the prevention of PTSD. Sixty rats were divided into six experimental groups: control vehicle, control L-Th, control naïve, PTSD vehicle, PTSD Pre-L-Th (prophylactic), PTSD Post-L-Th (non-prophylactic). PTSD was induced by a 3-day restraint/tail shock stress model. The effects of L-Th on neurobehavior were evaluated by Elevated Plus-Maze (EPM), Morris Water Maze (MWM), and Forced Swim Test (FST). Our study found that the total food intake weight of PTSD Pre-L-Th (prophylactic) rats were significantly increased compared to that of PTSD vehicle rats (p = .04). Administration of L-Th 24 hours before the initial PTSD event or for 10 days following the last PTSD stress event did not statistically improve mean open arm exploration on the EPM, spatial memory, and learning in the MWM or behavioral despair measured by the FST (p > 0.05). Although the 3-day restraint/tail shock stress model caused stress in the rodents, it did not produce reported PTSD-like anxiety and depression or spatial memory loss. The effect of Pre-L-Th or Post-L-Th treatment, on the neurobehavioral functions could not be effectively evaluated. However, this study provides a foundation for future studies to try different rodent PTSD models to induce PTSD-like neurobehavioral impairments to explore dosage, frequency, as well as the duration of L-Th administration before and/or after the post-traumatic event. The 3-day restraint/tail shock stress model caused stress in the rodents, Pre-L-Theanine treatment preconditioned the PTSD rats to endure stress.


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