The effect of electrical stimulation combined with dynamic strength training on healthy individuals

1998 ◽  
Vol 7 (3) ◽  
pp. 101-106
Author(s):  
Lee N. Burkett ◽  
Wayne T. Phillips ◽  
Brent Alvar ◽  
Lyle Bartelt ◽  
William Stone
2002 ◽  
Vol 46 (1) ◽  
pp. 274-274 ◽  
Author(s):  
Marten Munneke ◽  
Zuzana de Jong ◽  
Aeilko H. Zwinderman ◽  
Theodora P. M. Vliet Vlieland ◽  
Johanna M. W. Hazes

2006 ◽  
Vol 91 (12) ◽  
pp. 5107-5112 ◽  
Author(s):  
E. Klimcakova ◽  
J. Polak ◽  
C. Moro ◽  
J. Hejnova ◽  
M. Majercik ◽  
...  

Abstract Context: Obesity is characterized by a low-grade inflammatory state, which could play a role in insulin resistance. Dynamic strength training improves insulin sensitivity. Objective: The objective of this study was to investigate, in obese subjects, whether the insulin sensitizing effect of dynamic strength training is associated with changes in plasma levels and gene expression of adipokines potentially involved in the development of insulin resistance. Design: Twelve obese male subjects were investigated before and at the end of 3 months of dynamic strength training. Insulin sensitivity was evaluated using euglycemic-hyperinsulinemic clamp. Blood samples and needle biopsy samples of sc abdominal adipose tissue were obtained. The plasma levels and adipose tissue mRNA levels of adiponectin, leptin, IL-1β, IL-6, and TNF-α were determined. Results: The training induced an increase in the whole-body glucose disposal rate by 24% (P = 0.04). The body weight was not altered during the training. Plasma levels of leptin decreased during the training (16.6 ± 6.3 vs. 13.1 ± 5.7 ng/ml) by 21% (P < 0.02), whereas no change in plasma levels of other adipokines and C-reactive protein was observed. Gene expression of the investigated adipokines was not changed in sc adipose tissue during the training. Conclusions: In obese subjects, the dynamic strength training resulted in an improvement of whole-body insulin sensitivity. The increase in insulin sensitivity was not associated with training-induced modifications of plasma levels or adipose tissue gene expression of adipokines supposedly involved in the development of insulin resistance.


2019 ◽  
Vol 26 (14) ◽  
pp. 1483-1492 ◽  
Author(s):  
Dominique Hansen ◽  
Ana Abreu ◽  
Patrick Doherty ◽  
Heinz Völler

When added to endurance training, dynamic strength training leads to significantly greater improvements in peripheral muscle strength and power output in patients with cardiovascular disease, which may be relevant to enhance the patient’s prognosis. As a result, dynamic strength training is recommended in the rehabilitative treatment of many different cardiovascular diseases. However, what strength training intensity should be selected remains under intense debate. Evidence is nonetheless emerging that high-intensity strength training (≥70% of one-repetition maximum) is more effective to increase acutely myofibrillar protein synthesis, cause neural adaptations and, in the long term, increase muscle strength, when compared to low-intensity strength training. Moreover, multiple studies report that high-intensity strength training causes fewer increments in (intra-)arterial blood pressure and cardiac output, as opposed to low-intensity strength training, thus potentially pointing towards sufficient medical safety for the cardiovascular system. The aim of this systematic review is therefore to discuss this line of evidence, which is in contrast to current clinical practice, and to re-open the debate as to what dynamic strength training intensities should actually be applied.


2014 ◽  
Vol 14 (7) ◽  
pp. 695-702 ◽  
Author(s):  
Marcos D. M. Drummond ◽  
Bruno P. Couto ◽  
Izabela G. Augusto ◽  
Sara A. Rodrigues ◽  
Leszek A. Szmuchrowski

1992 ◽  
Vol 24 (Supplement) ◽  
pp. S21 ◽  
Author(s):  
R. Topp ◽  
A. E. Mikesky ◽  
J. K. Wigglesworth ◽  
J. E. Edwards

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