Lifestyle Changes to Reduce Cardiovascular Risk

2009 ◽  
pp. 50-56
Author(s):  
J. Mendive ◽  
E. McGregor ◽  
F. Sacks
PLoS ONE ◽  
2021 ◽  
Vol 16 (4) ◽  
pp. e0249923
Author(s):  
Orsolya Kiss ◽  
Mate Babity ◽  
Attila Kovacs ◽  
Judit Skopal ◽  
Hajnalka Vago ◽  
...  

The significance of cardiology screening of referees is not well established. Cardiovascular risk factors and diseases were examined in asymptomatic Hungarian elite handball referees undergoing extended screening: personal/family history, physical examination, 12-lead ECG, laboratory tests, body-composition analysis, echocardiography, and cardiopulmonary exercise testing. Holter-ECG (n = 8), blood pressure monitorization (n = 10), cardiac magnetic resonance imaging (CMR; n = 27) and computer tomography (CCT; n = 4) were also carried out if needed. We examined 100 referees (age: 29.6±7.9years, male: 64, training: 4.3±2.0 hours/week), cardiovascular risk factors were: positive medical history: 24%, overweight: 10%, obesity: 3%, dyslipidaemia: 41%. Elevated resting blood pressure was measured in 38%. Stress-ECG was positive due to ECG-changes in 16%, due to elevated exercise blood pressure in 8%. Echocardiography and/or CMR identified abnormalities in 19%. A significant number of premature ventricular contractions was found on the Holter-ECG in two cases. The CCT showed myocardial bridge or coronary plaques in one-one case. We recommended lifestyle changes in 58%, new/modified antihypertensive or lipid-lowering therapy in 5%, iron-supplementation in 22%. By our results, a high percentage of elite Hungarian handball referees had cardiovascular risk factors or diseases, which, combined with physical and psychological stress, could increase the possibility of cardiovascular events. Our study draws attention to the importance of cardiac screening in elite handball referees.


Author(s):  
Diego Vannuzzo ◽  
Simona Giampaoli

Cardiovascular primary prevention is a coordinated set of actions at community and individual level aimed at eradicating, eliminating, or compressing at later ages the impact of cardiovascular diseases and their related disability. Its aim is healthy ageing. Cardiovascular epidemiology has elucidated the role of cardiovascular risk factors, forming the basis of strategies to reduce cardiovascular risk and subsequent disease. There is evidence that cardiovascular primary prevention works if three strategies are implemented together: a population strategy (particularly through a widespread adoption of healthy lifestyles) which aims to keep everyone at low risk from infancy and reduces the cardiovascular risk profile of the whole community; an individualized high-risk strategy through lifestyle changes also prophylactic evidence-based drugs if necessary; and an individualized intermediate-risk strategy which may benefit from non-invasive assessment of subclinical disease and end organ damage.


Circulation ◽  
2010 ◽  
Vol 122 (4) ◽  
pp. 406-441 ◽  
Author(s):  
Nancy T. Artinian ◽  
Gerald F. Fletcher ◽  
Dariush Mozaffarian ◽  
Penny Kris-Etherton ◽  
Linda Van Horn ◽  
...  

2015 ◽  
Vol 13 (1) ◽  
pp. 29-35 ◽  
Author(s):  
Torunn Kristin Nestvold ◽  
Erik Waage Nielsen ◽  
Judith Krey Ludviksen ◽  
Hilde Fure ◽  
Anne Landsem ◽  
...  

Polycystic Ovary Syndrome (PCOS) is the most common endocrine-metabolic pathology among women at reproductive age. It has a multifactorial character, and its etiology has not yet been precisely explained. The pathogenesis of PCOS is related to metabolic issues such as hyperandrogenism and insulin resistance, and is also associated with obesity, type 2 diabetes and increased cardiovascular risk. Manifestations such as irregular menstruation, acne, hisurtism and androgenic alopecia are common in addition to the consequent psychological and quality of life impacts. Thus, knowing that the adoption of healthy habits have therapeutic impacts in the face of various signs and symptoms of PCOS, there is a need to analyze the influence of lifestyle on the prognosis of PCOS. The present study carried out its searches in the PUBMED database, using the descriptors "polycystic ovary syndrome", "life style" and "prognosis", using the Boolean operator "and". Inclusion criteria were used: articles written in Portuguese, English and Spanish, published in the last 5 years. 19 results were obtained, 5 of which were excluded, resulting in 14 articles chosen for theoretical reference. The influence of lifestyle on the prognosis of PCOS is notorious, especially in patients with overweight and obesity. Thus, healthy behaviors have the potential to improve pathological conditions and bad habits demonstrate that they can induce clinical manifestations of PCOS in predisposed people. Lifestyle changes, mainly associated with weight loss, show improvements in aspects such as insulin resistance, free testosterone, acne, hirsutism and reproductive function, reduced cardiovascular risk, in addition to positively influence to the psychic and quality of life in analyzed patients. Therapeutic strategies that combine lifestyle changes and drug interventions have been shown to be more effective, as well as strategies structered with the monitoring of professionals tend to increase adherence to treatment.


2019 ◽  
Author(s):  
Xuan Zhou ◽  
Julius van der Werf ◽  
Kristin Carson-Chahhoud ◽  
Guiyan Ni ◽  
John McGrath ◽  
...  

AbstractBoth genetic and non-genetic factors can predispose individuals to cardiovascular risk. Finding ways to alter these predispositions is important for cardiovascular disease (CVD) prevention. Here, we use a novel whole-genome framework to estimate genetic and non-genetic effects on—hence their predispositions to—cardiovascular risk and determine whether they vary with respect to lifestyle factors. We performed analyses on the Atherosclerosis Risk in Communities Study (ARIC, N=6,896-7,180) and validated findings using the UK Biobank (UKBB, N=14,076-34,538). Cardiovascular risk was measured using 23 traits in the ARIC and eight traits in the UKBB, such as body mass index (BMI), resting heart rate, white blood cell count and blood pressure; and lifestyle factors included information on physical activity, smoking, alcohol consumption and dietary intake. Physical activity altered both genetic and non-genetic effects on heart rate and BMI, genetic effects on HDL cholesterol level, and non-genetic effects on waist-to-hip ratio. Alcohol consumption altered both genetic and non-genetic effects on BMI, while smoking altered non-genetic effects on heart rate, pulse pressure, and white blood cell count. In addition, saturated fat intake modified genetic effects on BMI, and total daily energy intake modified non-genetic effects on waist-to-hip ratio. These results highlight the relevance of lifestyle changes for CVD prevention. We also stratified individuals according to their genetic predispositions and showed notable differences in the effects of lifestyle on cardiovascular risk across stratified groups, implying the need for individualizing lifestyle changes for CVD prevention. Finally, we showed that neglecting lifestyle modulation of genetic and non-genetic effects will on average reduce SNP heritability estimates of cardiovascular traits by a small yet significant amount, primarily owing to overestimation of residual variance. Thus, current SNP heritability estimates for cardiovascular traits, which commonly do not consider modulating effects of lifestyle covariates, are likely underestimated.


Author(s):  
Thomas Bardin

Asymptomatic hyperuricaemia is a frequent finding and evidence is growing that it could be an independent cardiovascular risk marker. Recent studies challenge the dogma not to treat asymptomatic hyperuricaemia. However, no urate-lowering drug has been approved for the management of asymptomatic hyperuricaemia in Western countries, because the lack of large randomized control trials precludes assessment of benefits. Asymptomatic hyperuricaemia should lead to lifestyle changes and promote the search for cardiovascular risk factors amenable to therapy, in order to lower the hazards of cardiovascular disease and mortality.


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