Effects of electromagnetic radiation of mobile phones on the central nervous system

2002 ◽  
Vol 24 (1) ◽  
pp. 49-62 ◽  
Author(s):  
K.-A. Hossmann ◽  
D.M. Hermann
2015 ◽  
Vol 8 (3) ◽  
pp. 2267-2277
Author(s):  
Dr. Esmail Ali

Devices such as mobile phones, wireless internet modems, and radios and televisions, which occupy an important place in social life, produce electromagnetic fields (EMFs). Widespread use of these devices in daily life increases the intensity of exposure to EMFs on a day to day basis. Investigation of the effects on health of devices such as mobile phones used in close proximity to the body is attracting considerable interest from scientists. Mobile phones manufactured using the latest technology operate in a high frequency range (300–3000 MHz). This further heightens concerns regarding the effect of mobile phones on human health. Most Global System for Mobile Communications (GSM) operators in Europe, Asia, and Africa use a frequency of 900 MHz. With the rapid development of electronic information and communication techniques, exposure to electromagnetic fields (EMFs) has increased dramatically. Some studies have focused on the biological effects of electromagnetic radiation. Microwave radiation has been reported as producing adverse effects in the central nervous system (CNS), including headache, sleep disorders, anxiety, cognitive dysfunction and neurogenesis impairment in both humans and animals. However, the direct effects of microwave radiation on neurodevelopment and the underlying mechanisms for any such effects remain unknown. As per today’s global scenario use of mobile phone is increasing day by day for communication. Due to its constant use, the electromagnetic radiation (EMR) emitted from the cell phone, base station and other household appliances cause adverse effects on human health. There is an increase concern about the interaction of EMR generated from mobile phones, with the human organs specially with brain because of its close and long proximity to human brain during the mobile usage. Concerns have shown whether these exposures could have effect on brain and central nervous system (CNS).


2012 ◽  
Vol 33 (6) ◽  
pp. 527-533 ◽  
Author(s):  
Manuel Murbach ◽  
Maria Christopoulou ◽  
Pedro Crespo-Valero ◽  
Peter Achermann ◽  
Niels Kuster

Author(s):  
Gladys Harrison

With the advent of the space age and the need to determine the requirements for a space cabin atmosphere, oxygen effects came into increased importance, even though these effects have been the subject of continuous research for many years. In fact, Priestly initiated oxygen research when in 1775 he published his results of isolating oxygen and described the effects of breathing it on himself and two mice, the only creatures to have had the “privilege” of breathing this “pure air”.Early studies had demonstrated the central nervous system effects at pressures above one atmosphere. Light microscopy revealed extensive damage to the lungs at one atmosphere. These changes which included perivascular and peribronchial edema, focal hemorrhage, rupture of the alveolar septa, and widespread edema, resulted in death of the animal in less than one week. The severity of the symptoms differed between species and was age dependent, with young animals being more resistant.


Author(s):  
John L.Beggs ◽  
John D. Waggener ◽  
Wanda Miller ◽  
Jane Watkins

Studies using mesenteric and ear chamber preparations have shown that interendothelial junctions provide the route for neutrophil emigration during inflammation. The term emigration refers to the passage of white blood cells across the endothelium from the vascular lumen. Although the precise pathway of transendo- thelial emigration in the central nervous system (CNS) has not been resolved, the presence of different physiological and morphological (tight junctions) properties of CNS endothelium may dictate alternate emigration pathways.To study neutrophil emigration in the CNS, we induced meningitis in guinea pigs by intracisternal injection of E. coli bacteria.In this model, leptomeningeal inflammation is well developed by 3 hr. After 3 1/2 hr, animals were sacrificed by arterial perfusion with 3% phosphate buffered glutaraldehyde. Tissues from brain and spinal cord were post-fixed in 1% osmium tetroxide, dehydrated in alcohols and propylene oxide, and embedded in Epon. Thin serial sections were cut with diamond knives and examined in a Philips 300 electron microscope.


Author(s):  
Ezzatollah Keyhani

Acetylcholinesterase (EC 3.1.1.7) (ACHE) has been localized at cholinergic junctions both in the central nervous system and at the periphery and it functions in neurotransmission. ACHE was also found in other tissues without involvement in neurotransmission, but exhibiting the common property of transporting water and ions. This communication describes intracellular ACHE in mammalian bone marrow and its secretion into the extracellular medium.


Author(s):  
S.S. Spicer ◽  
B.A. Schulte

Generation of monoclonal antibodies (MAbs) against tissue antigens has yielded several (VC1.1, HNK- 1, L2, 4F4 and anti-leu 7) which recognize the unique sugar epitope, glucuronyl 3-sulfate (Glc A3- SO4). In the central nervous system, these MAbs have demonstrated Glc A3-SO4 at the surface of neurons in the cerebral cortex, the cerebellum, the retina and other widespread regions of the brain.Here we describe the distribution of Glc A3-SO4 in the peripheral nervous system as determined by immunostaining with a MAb (VC 1.1) developed against antigen in the cat visual cortex. Outside the central nervous system, immunoreactivity was observed only in peripheral terminals of selected sensory nerves conducting transduction signals for touch, hearing, balance and taste. On the glassy membrane of the sinus hair in murine nasal skin, just deep to the ringwurt, VC 1.1 delineated an intensely stained, plaque-like area (Fig. 1). This previously unrecognized structure of the nasal vibrissae presumably serves as a tactile end organ and to our knowledge is not demonstrable by means other than its selective immunopositivity with VC1.1 and its appearance as a densely fibrillar area in H&E stained sections.


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