Lipid body formation by Thraustochytrium aureum (ATCC 34304) in response to cell age

2008 ◽  
Vol 25 (5) ◽  
pp. 1103-1109 ◽  
Author(s):  
Eun-Jin Jeh ◽  
Rangarajulu Senthil Kumaran ◽  
Byung-Ki Hur
Keyword(s):  
Lipid Body ◽  
Body Formation ◽  
Thraustochytrium Aureum ◽  
Cell Age

scholarly journals Leptin Induces Macrophage Lipid Body Formation by a Phosphatidylinositol 3-Kinase- and Mammalian Target of Rapamycin-dependent Mechanism

2007 ◽  
Vol 283 (4) ◽  
pp. 2203-2210 ◽  
Author(s):  
Clarissa M. Maya-Monteiro ◽  
Patricia E. Almeida ◽  
Heloisa D'Ávila ◽  
Aline S. Martins ◽  
Ana Paula Rezende ◽  
...  
Keyword(s):  
Mammalian Target Of Rapamycin ◽  
Lipid Body ◽  
Target Of Rapamycin ◽  
Phosphatidylinositol 3 Kinase ◽  
Body Formation ◽  
Dependent Mechanism ◽  
Phosphatidylinositol 3

scholarly journals PPAR-γ activation by Tityus serrulatus venom regulates lipid body formation and lipid mediator production

Toxicon ◽  
2015 ◽  
Vol 93 ◽  
pp. 90-97 ◽  
Author(s):  
Karina Furlani Zoccal ◽  
Francisco Wanderley Garcia Paula-Silva ◽  
Claudia da Silva Bitencourt ◽  
Carlos Artério Sorgi ◽  
Karla de Castro Figueiredo Bordon ◽  
...  
Keyword(s):  
Lipid Body ◽  
Lipid Mediator ◽  
Body Formation ◽  
Ppar Γ ◽  
Tityus Serrulatus ◽  
Tityus Serrulatus Venom

scholarly journals Lipopolysaccharide-Induced Leukocyte Lipid Body Formation In Vivo: Innate Immunity Elicited Intracellular Loci Involved in Eicosanoid Metabolism

2002 ◽  
Vol 169 (11) ◽  
pp. 6498-6506 ◽  
Author(s):  
Patrícia Pacheco ◽  
Fernando A. Bozza ◽  
Rachel N. Gomes ◽  
Marcelo Bozza ◽  
Peter F. Weller ◽  
...  
Keyword(s):  
Innate Immunity ◽  
Lipid Body ◽  
Body Formation ◽  
Eicosanoid Metabolism

scholarly journals Histoplasma capsulatumCell Wall β-Glucan Induces Lipid Body Formation through CD18, TLR2, and Dectin-1 Receptors: Correlation with Leukotriene B4Generation and Role in HIV-1 Infection

2009 ◽  
Vol 182 (7) ◽  
pp. 4025-4035 ◽  
Author(s):  
Carlos Artério Sorgi ◽  
Adriana Secatto ◽  
Caroline Fontanari ◽  
Walter Miguel Turato ◽  
Caroline Belangér ◽  
...  
Keyword(s):  
Lipid Body ◽  
Body Formation ◽  
Hiv 1

scholarly journals Lipid body formation plays a central role in cell fate determination during developmental differentiation ofMyxococcus xanthus

2009 ◽  
Vol 74 (2) ◽  
pp. 497-517 ◽  
Author(s):  
Egbert Hoiczyk ◽  
Michael W. Ring ◽  
Colleen A. McHugh ◽  
Gertrud Schwär ◽  
Edna Bode ◽  
...  
Keyword(s):  
Cell Fate ◽  
Lipid Body ◽  
Cell Fate Determination ◽  
Body Formation ◽  
Fate Determination ◽  
Developmental Differentiation

OR.71. Histoplasma capsulatum Cell Wall β-glucan Induced Lipid Body Formation and LTB4 Generation Through TLR2 And CD18 Receptors

2008 ◽  
Vol 127 ◽  
pp. S30
Author(s):  
Carlos Sorgi ◽  
Alexandra Medeiros ◽  
Adriana Secatto ◽  
Caroline Fontanari ◽  
Walter Turato ◽  
...  
Keyword(s):  
Cell Wall ◽  
Histoplasma Capsulatum ◽  
Lipid Body ◽  
Body Formation

scholarly journals ATF3 protects against atherosclerosis by suppressing 25-hydroxycholesterol–induced lipid body formation

2012 ◽  
Vol 209 (4) ◽  
pp. 807-817 ◽  
Author(s):  
Elizabeth S. Gold ◽  
Stephen A. Ramsey ◽  
Mark J. Sartain ◽  
Jyrki Selinummi ◽  
Irina Podolsky ◽  
...  
Keyword(s):  
Foam Cell ◽  
Cell Formation ◽  
Lipid Body ◽  
Chronic Inflammatory Disease ◽  
Intersection Point ◽  
Prediction Algorithm ◽  
Foam Cell Formation ◽  
Macrophage Foam Cell ◽  
Body Formation

Atherosclerosis is a chronic inflammatory disease characterized by the accumulation of lipid-loaded macrophages in the arterial wall. We demonstrate that macrophage lipid body formation can be induced by modified lipoproteins or by inflammatory Toll-like receptor agonists. We used an unbiased approach to study the overlap in these pathways to identify regulators that control foam cell formation and atherogenesis. An analysis method integrating epigenomic and transcriptomic datasets with a transcription factor (TF) binding site prediction algorithm suggested that the TF ATF3 may regulate macrophage foam cell formation. Indeed, we found that deletion of this TF results in increased lipid body accumulation, and that ATF3 directly regulates transcription of the gene encoding cholesterol 25-hydroxylase. We further showed that production of 25-hydroxycholesterol (25-HC) promotes macrophage foam cell formation. Finally, deletion of ATF3 in Apoe−/− mice led to in vivo increases in foam cell formation, aortic 25-HC levels, and disease progression. These results define a previously unknown role for ATF3 in controlling macrophage lipid metabolism and demonstrate that ATF3 is a key intersection point for lipid metabolic and inflammatory pathways in these cells.

Mechanisms of lipid-body formation

1999 ◽  
Vol 24 (3) ◽  
pp. 109-115 ◽  
Author(s):  
Denis J Murphy ◽  
Jean Vance
Keyword(s):  
Lipid Body ◽  
Body Formation
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