Comparison of the efficacy of growth factor collagen and antibiotic collagen on colon anastomosis in  experimental animals with peritonitis

2021 ◽  
Author(s):  
Mehmet Aykut Yıldırım ◽  
Murat Çakır ◽  
Sıddıka Fındık ◽  
Ömer Kişi ◽  
Mustafa Şentürk
Keyword(s):  
Growth Factor ◽  
Colon Anastomosis ◽  
Experimental Animals

scholarly journals Effect of clenbuterol on growth, nitrogen and energy balances and endocrine status in food-restricted sheep : research communication

2002 ◽  
Vol 73 (3) ◽  
Author(s):  
L.A. Cardoso ◽  
O. Taveira
Keyword(s):  
Growth Factor ◽  
Body Mass ◽  
The Body ◽  
Serum Concentrations ◽  
Experimental Animals ◽  
Endocrine Status ◽  
Energy Retention ◽  
Serra Da Estrela ◽  
Energy Balances ◽  
Energy Maintenance

The aim of this study was to examine the effect of the b2-adrenoceptor clenbuterol on food-restricted sheep. Clenbuterol was administered as a dietary admixture (4 mg/ kg diet) to a group of male Serra da Estrela sheep (n = 6).The animals were housed individually in metabolic cages and fed for 45 days at 65 % of estimated requirement for energy maintenance. An untreated group with the same energy intake level was included as a control. Changes in body mass, nitrogen and energy balances and insulin, insulin-like growth factor (IGF-1), and triiodothyronine (T3) levels in the experimental animals were monitored. During the 4th week of the trial, clenbuterol-treated sheep showed increased mass gains, greater energy retention and serum IGF-1 levels and decreased T3 serum concentrations. This study showed that clenbuterol may induce a protective effect in sheep subjected to periods of food deprivation, based on the body mass and digestible energy effects manifested by treated animals.

Mycophenolate mofetil impairs healing of left sided colon anastomosis and insulin-like growth factor-I tends to neutralize this impairment

2001 ◽  
Vol 120 (5) ◽  
pp. A342-A343
Author(s):  
Gregor Baumann ◽  
Roman Inglin ◽  
Dorothee Schoenfeld ◽  
Olaf Dirsch ◽  
Joerg Zeeh ◽  
...  
Keyword(s):  
Mycophenolate Mofetil ◽  
Growth Factor ◽  
Colon Anastomosis ◽  
Insulin Like Growth Factor ◽  
Factor I ◽  
Growth Factor I

Expression of vascular endothelial growth factor and its receptors in the cochlea of various experimental animals

2005 ◽  
Vol 125 (11) ◽  
pp. 1152-1157 ◽  
Author(s):  
Pasqualina M. Picciotti ◽  
Angela Torsello ◽  
Italo Cantore ◽  
Egidio Stigliano ◽  
Gaetano Paludetti ◽  
...  
Keyword(s):  
Vascular Endothelial Growth Factor ◽  
Growth Factor ◽  
Vascular Endothelial ◽  
Experimental Animals ◽  
Endothelial Growth Factor

scholarly journals Pharmacokinetic Modeling of Hepatocyte Growth Factor in Experimental Animals and Humans

2013 ◽  
Vol 102 (1) ◽  
pp. 237-249 ◽  
Author(s):  
Tomoko Sugiura ◽  
Saki Takahashi ◽  
Kazusa Sano ◽  
Tetsushi Abe ◽  
Kazuhiro Fukuta ◽  
...  
Keyword(s):  
Growth Factor ◽  
Hepatocyte Growth Factor ◽  
Pharmacokinetic Modeling ◽  
Experimental Animals ◽  
Hepatocyte Growth

scholarly journals A Review of the Genetics and Pathogenesis of Syndactyly in Humans and Experimental Animals: A 3-Step Pathway of Pathogenesis

2019 ◽  
Vol 2019 ◽  
pp. 1-10 ◽  
Author(s):  
Mohammad M. Al-Qattan
Keyword(s):  
Extracellular Matrix ◽  
Retinoic Acid ◽  
Growth Factor ◽  
Canonical Pathway ◽  
Fibroblast Growth ◽  
Experimental Animals ◽  
Fibroblast Growth Factor 8 ◽  
Morphogenetic Protein ◽  
Ecm Degradation ◽  
Web Space

Embryology of normal web space creation and the genetics of syndactyly in humans and experimental animals are well described in the literature. In this review, the author offers a 3-step pathway of pathogenesis for syndactyly. The first step is initiated either by the overactivation of the WNT canonical pathway or the suppression of the Bone Morphogenetic Protein (BMP) canonical pathway. This leads to an overexpression of Fibroblast Growth Factor 8 (FGF8). The final step is the suppression of retinoic acid in the interdigital mesenchyme leading to suppression of both apoptosis and extracellular matrix (ECM) degradation, resulting in syndactyly.

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