scholarly journals Correction to Toxic Effects and Molecular Mechanism of Different Types of Silver Nanoparticles to the Aquatic Crustacean Daphnia magna

2020 ◽  
Vol 54 (3) ◽  
pp. 2049-2049
Author(s):  
Jing Hou ◽  
Yue Zhou ◽  
Chunjie Wang ◽  
Shiguo Li ◽  
Xiangke Wang
2017 ◽  
Vol 51 (21) ◽  
pp. 12868-12878 ◽  
Author(s):  
Jing Hou ◽  
Yue Zhou ◽  
Chunjie Wang ◽  
Shiguo Li ◽  
Xiangke Wang

Author(s):  
Marlon Luiz Neves Silva ◽  
Diego José Nogueira ◽  
Jéssica Schveitzer Köerich ◽  
Vitor Pereira Vaz ◽  
Naiara Mottim Justino ◽  
...  

2021 ◽  
Vol 22 (5) ◽  
pp. 2536
Author(s):  
Rong-Jane Chen ◽  
Chiao-Ching Huang ◽  
Rosita Pranata ◽  
Yu-Hsuan Lee ◽  
Yu-Ying Chen ◽  
...  

Silver nanoparticles pose a potential risk to ecosystems and living organisms due to their widespread use in various fields and subsequent gradual release into the environment. Only a few studies have investigated the effects of silver nanoparticles (AgNPs) toxicity on immunological functions. Furthermore, these toxic effects have not been fully explored. Recent studies have indicated that zebrafish are considered a good alternative model for testing toxicity and for evaluating immunological toxicity. Therefore, the purpose of this study was to investigate the toxicity effects of AgNPs on innate immunity using a zebrafish model and to investigate whether the natural compound pterostilbene (PTE) could provide protection against AgNPs-induced immunotoxicity. Wild type and neutrophil- and macrophage-transgenic zebrafish lines were used in the experiments. The results indicated that the exposure to AgNPs induced toxic effects including death, malformation and the innate immune toxicity of zebrafish. In addition, AgNPs affect the number and function of neutrophils and macrophages. The expression of immune-related cytokines and chemokines was also affected. Notably, the addition of PTE could activate immune cells and promote their accumulation in injured areas in zebrafish, thereby reducing the damage caused by AgNPs. In conclusion, AgNPs may induce innate immune toxicity and PTE could ameliorate this toxicity.


2014 ◽  
Vol 466-467 ◽  
pp. 232-241 ◽  
Author(s):  
Fabianne Ribeiro ◽  
Julián Alberto Gallego-Urrea ◽  
Kerstin Jurkschat ◽  
Alison Crossley ◽  
Martin Hassellöv ◽  
...  

Life Sciences ◽  
2021 ◽  
pp. 120183
Author(s):  
Tolulope Peter Saliu ◽  
Thanutchaporn Kumrungsee ◽  
Kenshu Miyata ◽  
Hikaru Tominaga ◽  
Nao Yazawa ◽  
...  

2015 ◽  
Vol 2015 ◽  
pp. 1-12 ◽  
Author(s):  
Eun Kyung Sohn ◽  
Seyed Ali Johari ◽  
Tae Gyu Kim ◽  
Jin Kwon Kim ◽  
Ellen Kim ◽  
...  

To better understand the potential ecotoxicological impact of silver nanoparticles (AgNPs) and silver nanowires (AgNWs) released into freshwater environments, the toxicities of these nanomaterials were assessed and compared using Organization for Economic Cooperation and Development (OECD) test guidelines, including a “Daphniasp., acute immobilization test,” “Fish, acute toxicity test,” and “freshwater alga and cyanobacteria, growth inhibition test.” Based on the estimated median lethal/effective concentrations of AgNPs and AgNWs, the susceptibility to the nanomaterials was different among test organisms (daphnia > algae > fish), suggesting that the AgNPs are classified as “category acute 1” forDaphnia magna, “category acute 2” forOryzias latipes, and “category acute 1” forRaphidocelis subcapitata, while the AgNWs are classified as “category acute 1” forDaphnia magna, “category acute 2” forOryzias latipes, and “category acute 2” forRaphidocelis subcapitata, according to the GHS (Globally Harmonized System of Classification and Labelling of Chemicals). In conclusion, the present results suggest that more attention should be paid to prevent the accidental or intentional release of silver nanomaterials into freshwater aquatic environments.


Molecules ◽  
2021 ◽  
Vol 26 (21) ◽  
pp. 6610
Author(s):  
Ana T. Rufino ◽  
Ana Ramalho ◽  
Adelaide Sousa ◽  
José Miguel P. Ferreira de Oliveira ◽  
Paulo Freitas ◽  
...  

Silver nanoparticles (AgNP) have been increasingly incorporated into food-related and hygiene products for their unique antimicrobial and preservative properties. The consequent oral exposure may then result in unpredicted harmful effects in the gastrointestinal tract (GIT), which should be considered in the risk assessment and risk management of these materials. In the present study, the toxic effects of polyethyleneimine (PEI)-coated AgNP (4 and 19 nm) were evaluated in GIT-relevant cells (Caco-2 cell line as a model of human intestinal cells, and neutrophils as a model of the intestinal inflammatory response). This study also evaluated the putative protective action of dietary flavonoids against such harmful effects. The obtained results showed that AgNP of 4 and 19 nm effectively induced Caco-2 cell death by apoptosis with concomitant production of nitric oxide, irrespective of the size. It was also observed that AgNP induced human neutrophil oxidative burst. Interestingly, some flavonoids, namely quercetin and quercetagetin, prevented the deleterious effects of AgNP in both cell types. Overall, the data of the present study provide a first insight into the promising protective role of flavonoids against the potentially toxic effects of AgNP at the intestinal level.


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