Pathological Prosody
Prosody in speech arises from fundamental frequency (f0), temporal measures, amplitude, and voice quality. These parameters in various combinations signal linguistic, emotional-attitudinal, and personal identity information. Prosodic processing engages cortical, subcortical, cerebellar, and brainstem systems, and damage to any of these can result in dysprosody. Despite the crucial role played by prosody in communication, little attention has been paid to clinical evaluation and even less to treatment. Numerous research studies of deficient emotional prosodic production and recognition reveal temporal deficits in association with left hemisphere damage and pitch processing failure in right hemisphere damage. Limited attempts at linguistic-prosodic testing suggest that damage to either cerebral hemisphere or to subcortical systems can affect production as well as comprehension. Treatment of linguistic dysprosody has revealed challenges in protocol design and performance evaluation. Prosodic insufficiency, present in cortical, subcortical, and cerebellar disease, has substantial implications for communicative function and deserves to be a strong focus of future research.