scholarly journals Relationship of plasma levels of C‐reactive protein and adiponectin and change of lean body mass in obese postmenopausal women supplemented with safflower oil

2013 ◽  
Vol 27 (S1) ◽  
Author(s):  
Rachel M Cole ◽  
Rebecca Andridge ◽  
Martha A Belury
Keyword(s):  
Postmenopausal Women ◽  
Lean Body Mass ◽  
Body Mass ◽  
Plasma Levels ◽  
Safflower Oil ◽  
C Reactive Protein ◽  
Reactive Protein ◽  
Relationship Of

scholarly journals Nutritional profile of asymptomatic alcoholic patients

2011 ◽  
Vol 48 (2) ◽  
pp. 112-118 ◽  
Author(s):  
Maria Beatriz Sobral-Oliveira ◽  
Joel Faintuch ◽  
Dulce Reis Guarita ◽  
Claudia P. Oliveira ◽  
Flair J. Carrilho
Keyword(s):  
Lean Body Mass ◽  
Body Mass ◽  
Alcoholic Pancreatitis ◽  
C Reactive Protein ◽  
Chronic Alcoholic Pancreatitis ◽  
Chronic Alcoholic ◽  
Vitamin Deficiencies ◽  
Reactive Protein ◽  
Amyloid A ◽  
Visceral Disease

CONTEXT: Alcoholism may interfere with nutritional status, but reports are often troubled by uncertainties about ingested diet and organ function, as well as by ongoing abuse and associated conditions. OBJECTIVE: To identify nutritional and body compartment changes in stable alcoholics without confounding clinical and dietetic variables, a prospective observational pilot study was designed. Three well-matched populations were considered: subjects with chronic alcoholic pancreatitis, alcoholics without visceral disease, and healthy never-drinking adults (controls). METHODS: Subjects (n = 60) were asymptomatic males with adequate diet, no superimposed disease or complication, and alcohol-free for at least 6 months. After exclusions, 48 patients were compared. Variables encompassed dietary recall, bioimpedance analysis, biochemical profile and inflammatory markers. Main outcome measures were body fat, lean body mass, serum lipids, C-reactive protein, and selected minerals and vitamins. RESULTS: Both alcoholic populations suffered from reduced lean body mass (P = 0.001), with well-maintained body fat.Magnesium was depleted, and values of vitamin D and B12 correlated with alcohol abuse. LDL and total cholesterol was increased in alcoholics without pancreatitis (P = 0.04), but not in those with visceral damage. C-reactive protein and serum amyloid A correlated with duration of excessive drinking (P = 0.01). CONCLUSIONS: Undernutrition (diminished lean body mass, risk of magnesium and vitamin deficiencies) contrasted with dyslipidemia and increased cardiovascular risk. This second danger was masked during chronic pancreatitis but not in alcoholics without visceral disease. Further studies should focus special requirements of this population.

scholarly journals Relationship of Circulating Total Homocysteine and C-Reactive Protein to Trabecular Bone in Postmenopausal Women

2007 ◽  
Vol 10 (4) ◽  
pp. 395-403 ◽  
Author(s):  
Shilpa N. Bhupathiraju ◽  
D. Lee Alekel ◽  
Jeanne W. Stewart ◽  
Laura N. Hanson ◽  
Kristine M. Shedd ◽  
...  
Keyword(s):  
Trabecular Bone ◽  
Postmenopausal Women ◽  
C Reactive Protein ◽  
Reactive Protein ◽  
Total Homocysteine ◽  
Relationship Of

Relationship between insulin sensitivity and the triglyceride–HDL-C ratio in overweight and obese postmenopausal women: a MONET study

2007 ◽  
Vol 32 (6) ◽  
pp. 1089-1096 ◽  
Author(s):  
Antony D. Karelis ◽  
Stephanie M. Pasternyk ◽  
Lyne Messier ◽  
David H. St-Pierre ◽  
Jean-Marc Lavoie ◽  
...  
Keyword(s):  
Oxygen Consumption ◽  
Insulin Sensitivity ◽  
Muscle Strength ◽  
Postmenopausal Women ◽  
Body Mass ◽  
Visceral Fat ◽  
Peak Oxygen Consumption ◽  
Glucose Disposal ◽  
C Reactive Protein ◽  
Reactive Protein

The objective of this cross-sectional study was to examine the relationship between the triglyceride–HDL-cholesterol ratio (TG:HDL-C) and insulin sensitivity in overweight and obese sedentary postmenopausal women. The study population consisted of 131 non-diabetic overweight and obese sedentary postmenopausal women (age; 57.7 ± 5.0 y; body mass index (BMI), 32.2 ± 4.3 kg/m2). Subjects were characterized by dividing the entire cohort into tertiles based on the TG:HDL-C (T1 < 0.86 vs. T2= 0.86 to 1.35 vs. T3 > 1.35, respectively). We measured (i) insulin sensitivity (using the hyperinsulinenic–euglycemic clamp and homeostasis model assessment (HOMA)), (ii) body composition (using dual-energy X-ray absorptiometry), (iii) visceral fat (using computed tomography), (iv) plasma lipids, C-reactive protein, 2 h glucose concentration during an oral glucose tolerance test (2 h glucose), as well as fasting glucose and insulin, (v) peak oxygen consumption, and (vi) lower-body muscle strength (using weight training equipment). Significant correlations were observed between the TG:HDL-C and the hyperinsulinemic–euglycemic clamp (r = –0.45; p < 0.0001), as well as with HOMA (r = 0.42; p < 0.0001). Moreover, the TG:HDL-C significantly correlated with lean body mass, visceral fat, 2 h glucose, C-reactive protein, and muscle strength. Stepwise regression analysis showed that the TG:HDL-C explained 16.4% of the variation in glucose disposal in our cohort, which accounted for the greatest source of unique variance. Other independent predictors of glucose disposal were 2 h glucose (10.1%), C-reactive protein (CRP; 7.6%), and peak oxygen consumption (5.8%), collectively (including the TG:HDL-C) explaining 39.9% of the unique variance. In addition, the TG:HDL-C was the second predictor for HOMA, accounting for 11.7% of the variation. High levels of insulin sensitivity were associated with low levels of the TG:HDL-C. In addition, the TG:HDL-C was a predictor for glucose disposal rates and HOMA values in our cohort of overweight and obese postmenopausal women.

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