Absolute transmitted light plethysmography for assessment of dental pulp vitality through quantification of pulp chamber hematocrit by a three-layer model

2008 ◽  
Vol 13 (5) ◽  
pp. 054023 ◽  
Author(s):  
Satoko Kakino ◽  
Yuzo Takagi ◽  
Setsuo Takatani
2009 ◽  
Vol 2009 ◽  
pp. 1-12 ◽  
Author(s):  
Eugene Chen ◽  
Paul V. Abbott

Dental pulp testing is a useful and essential diagnostic aid in endodontics. Pulp sensibility tests include thermal and electric tests, which extrapolate pulp health from sensory response. Whilst pulp sensibility tests are the most commonly used in clinical practice, they are not without limitations and shortcomings. Pulp vitality tests attempt to examine the presence of pulp blood flow, as this is viewed as a better measure of true health than sensibility. Laser Doppler flowmetry and pulse oximetry are examples of vitality tests. Whilst the prospect is promising, there are still many practical issues that need to be addressed before vitality tests can replace sensibility tests as the standard clinical pulp diagnostic test. With all pulp tests, the results need to be carefully interpreted and closely scrutinised as false results can lead to misdiagnosis which can then lead to incorrect, inappropriate, or unnecessary treatment.


2019 ◽  
Vol 3 (2) ◽  

Objective: To search for a dental pulp preservative method in experimental pulpitis. Methods: 20 rabbits with experimental pulpitis were selected for this study; Teeth were randomly assigned to 2 groups (experimental and control groups) based on the final irrigation methods. In experimental group (double-pinhole drainage), the two pinholes were drilled in middle 1/3 dental cervix of buccal labial surface of tooth; the pinholes were inserted into infant transfusion needles then the two syringes were contacted with them, one for suck, another for irrigation. In control group, pinhole was sealed. Rabbits were executed in 1, 3, 5, 7 day. The inflammation in pulp chamber was assessed after 1, 3, 5, and 7 days. Results: At the 1, 5 day time intervals, control group reported more inflammation than experimental group (P < .05). There was no significant difference between two groups at the other time intervals (P > .05), and in all groups the inflammation decreased over time. Conclusion: Dental pulp can be preserved by double-pinhole drainage efficiently.


Author(s):  
Sarah Bi ◽  
Laura Martinez ◽  
Justin Bequette ◽  
Andrew Peitzsch ◽  
William D'Angelo

2015 ◽  
Vol 67 (6) ◽  
pp. 872-886
Author(s):  
Olga Rudzka ◽  
Rodrigo Giacaman ◽  
Renata Chałas
Keyword(s):  

2012 ◽  
Vol 33 (3) ◽  
pp. 366-375 ◽  
Author(s):  
Marion Hirmer ◽  
Sergey N. Danilov ◽  
Stephan Giglberger ◽  
Jürgen Putzger ◽  
Andreas Niklas ◽  
...  

1991 ◽  
Vol 38 (4) ◽  
pp. 346-352 ◽  
Author(s):  
J.M. Schmitt ◽  
R.L. Webber ◽  
E.C. Walker

2019 ◽  
Vol 98 (8) ◽  
pp. 912-919 ◽  
Author(s):  
T. Liang ◽  
H. Zhang ◽  
Q. Xu ◽  
S. Wang ◽  
C. Qin ◽  
...  

Dentin sialophosphoprotein (DSPP) is an extracellular matrix protein highly expressed by odontoblasts in teeth. DSPP mutations in humans may cause dentinogenesis imperfecta (DGI), an autosomal dominant dentin disorder. We recently generated a mouse model (named “ DsppP19L/+ mice”) that expressed a mutant DSPP in which the proline residue at position 19 was replaced by a leucine residue. We found that the DsppP19L/+ and DsppP19L/P19L mice at a younger age displayed a tooth phenotype resembling human DGI type III characterized by enlarged dental pulp chambers, while the teeth of older DsppP19L/+ and DsppP19L/P19L mice had smaller dental pulp chambers mimicking DGI type II. The teeth of DsppP19L/+ and DsppP19L/P19L mice had a narrower pulp chamber roof predentin layer, thinner pulp chamber roof dentin, and thicker pulp chamber floor dentin. In addition, these mice also had increased enamel attrition, accompanied by excessive deposition of peritubular dentin. Immunohistochemistry, in situ hybridization, and real-time polymerase chain reaction analyses showed that the odontoblasts in both DsppP19L/+ and DsppP19L/P19L mice had reduced DSPP expression, compared to the wild-type mice. We also observed that the levels of DSPP expression were much higher in the roof-forming odontoblasts than in the floor-forming odontoblasts in the wild-type mice and mutant mice. Moreover, immunohistochemistry showed that while the immunostaining signals of dentin sialoprotein (N-terminal fragment of DSPP) were decreased in the dentin matrix, they were remarkably increased in the odontoblasts of the DsppP19L/+ and DsppP19L/P19L mice. Consistently, our in vitro studies showed that the secretion of the mutant DSPP was impaired and accumulated within endoplasmic reticulum. These findings suggest that the dental phenotypes of the mutant mice were associated with the intracellular retention of the mutant DSPP in the odontoblasts of the DSPP-mutant mice.


1988 ◽  
Vol 10 (2) ◽  
pp. 146-148 ◽  
Author(s):  
J. Daley ◽  
E. Boyd ◽  
J. Cooper ◽  
P. O'Driscoll
Keyword(s):  

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