scholarly journals Reduced Aircraft Noise Pollution During COVID-19 Lockdown Is Beneficial to Public Cardiovascular Health: a Perspective on the Reduction of Transportation-Associated Pollution

Author(s):  
Omar Hahad ◽  
Andreas Daiber ◽  
Thomas Münzel
2021 ◽  
Vol 67 (4) ◽  
Author(s):  
Iraida Redondo ◽  
Jaime Muriel ◽  
Cristina de Castro Díaz ◽  
José I. Aguirre ◽  
Diego Gil ◽  
...  

AbstractUrbanization constitutes one of the major transformations of natural habitats, creating new areas characterized by multiple potential wildlife stressors. Birds that live in highly anthropized zones are confronted with physiological and behavioural challenges caused by these stressors. Here, we investigated if several health parameters differed between three subpopulations of tree sparrow nestlings subjected to different levels of anthropogenic pollution, and particularly noise pollution: a quiet rural area, a noisy rural area adjacent to an airport and a heavily urbanized area. We compared body condition, oxidative stress markers and baseline corticosterone levels, expecting urban nestlings to be in overall worse condition as compared to rural (rural and rural airport) birds. In addition, we expected nestlings exposed to aircraft noise to show intermediate stress levels. We found that rural-airport nestlings had the highest levels of antioxidant capacity of plasma and did not differ from rural counterparts in the rest of the parameters. By contrast, urban nestlings were in slightly worse body condition and had lower antioxidant capacity than rural and rural-airport individuals. Our results suggest that aircraft noise does not constitute a significant stressor for nestlings. In contrast, urban conditions constitute a more challenging situation, negatively impacting different physiological systems. Although nestlings seem able to buffer these challenges in the short-term, further research should explore the long-term potential consequences of early exposure to these conditions.


2016 ◽  
Vol 16 (C) ◽  
pp. 85 ◽  
Author(s):  
Rosa Maria Bruno ◽  
Ugo Faraguna ◽  
Enrica Bonanni ◽  
Marina Di Pilla ◽  
Marco Di Galante ◽  
...  

2019 ◽  
Vol 40 (Supplement_1) ◽  
Author(s):  
S Steven ◽  
K Frenis ◽  
S Kroeller-Schoen ◽  
S Kalinovic ◽  
J Helmstaedter ◽  
...  

Abstract Background Environmental noise pollution has been identified as a cardiovascular risk and is characterized by moderate hypertension, endothelial dysfunction, increased oxidative stress, and inflammation. We have gained insights into the mechanism by which these consequences occur by exposing mice lacking the critical NADPH oxidase subunit gp91phox to aircraft noise. Mice were protected from the effects of aircraft noise exposure. NADPH oxidase is believed to be the mediator by which angiotensin II increases oxidative stress, making investigation into the additive effect of noise and hypertension an important subject in modern cardiovascular health research. Methods and results C57Bl/6J mice were implanted with subcutaneous osmotic mini-pumps, delivering a moderate dose of 0.5mg/kg/d of angiotensin II for 7 days. Immediately following the implantation, half the mice were exposed to aircraft noise for 7 days at a maximum sound pressure level of 85 dB(A) and a mean sound pressure level of 72 dB(A), a level at which hearing loss does not occur*. Non-invasive blood pressure measurements revealed an additive increase in blood pressure in noise-exposed hypertensive mice. Following sacrifice, endothelial dysfunction was evaluated through isometric tension recordings of 3mm aortic ring segments. These recordings support the blood pressure measurements and indicate a more serious impairment in acetylcholine-induced vasorelaxation in hypertensive mice exposed to noise than the hypertensive or noise only controls. Whole blood stimulated with phorbol 12,13-dibutyrate (PDBu) or zymosan A showed an additive increase in oxidative burst in in noise-exposed hypertensive mice. Dihydroethidium (DHE) staining was used to assess the presence of vascular and cerebral oxidative stress, showing similar additive effects in mice with hypertension plus noise exposure. High performance liquid chromatography (HPLC) measurement of 2-hydroxyethidium further confirmed additive increase of oxidative stress in the aorta and brain. Western blot analysis of aortic tissue revealed highest levels of gp91phox in mice with hypertension plus noise exposure and indicated a decrease in the ratio of P-eNOSSer1177:eNOS as well as a decrease in the ratio of eNOS dimer:monomer, exposing eNOS uncoupling as a potential pathomechanism for endothelial dysfunction and gp91phox as a source for the oxidative stress.Ongoing immunohistochemical and flow cytometric investigations will characterize the role of immune cells in these adverse effects. Conclusion Herein, we present novel data demonstrating additive noise-induced cardiovascular consequences on developing hypertension. Noise has previously been established as a cardiovascular risk factor, but the effects have not been determined in pre-existing or developing cardiovascular disease. Our results show a cumulative effect between noise exposure and hypertension and forge an important link between environmental stressors and cardiovascular health. Acknowledgement/Funding Boehringer Ingelheim Foundation


Author(s):  
Peter Preisendörfer ◽  
Lucie Herold ◽  
Karin Kurz

Abstract This article investigates whether and to what extent unfavorable local environmental conditions furnish an important motivator for environmental protest. We do so using individual-level data on objective and subjectively perceived residential road traffic and aircraft noise pollution, pertaining to the cities of Mainz (Germany) and Zurich (Switzerland). By referring to fine-grained noise data, we are able to test the predictive power of grievances and self-interest in explaining protest participation more stringently than has been the case in most previous studies. Theoretically, our study is inspired by Klandermans’ socio-psychological framework of political protest, the pressure-response approach, the self-interest perspective, and the collective-interest model. Our empirical findings only partially confirm the grievances assumption that unfavorable local environmental conditions in the form of residential road traffic and aircraft noise stimulate environmental protest. Noise caused by airplanes seems to be more “protest-inducing” than that produced by road traffic. It is not so much the objectively measurable noise level as its subjective perception and evaluation that are deciding factors. However, in line with Klandermans’ protest framework and other theories of political protest, there are more influential drivers of environmental protest, such as environmental concerns and a left-wing political ideology. Thus, the effects of residential road traffic and aircraft noise turn out to be relatively moderate. Ultimately, this means that our tailored measures of grievances corroborate a relatively well-established finding of protest research.


2019 ◽  
Vol 266 ◽  
pp. 02005
Author(s):  
Azlina Md. Yassin ◽  
Mohd Lizam Mohd Diah ◽  
Edie Ezwan Mohd Safian ◽  
Mohd Yamani Yahya ◽  
Sulzakimin Mohammad ◽  
...  

The objective of this paper is to examine the effect of aircraft noise on residential property price within the case study area, and the main focus of this research was the distance of selected residential housed from Kuching International Airport (KIA). Aircraft noise is a source of noise pollution and act as environment factor that affect the house prices. Environmental disamenities from water and noise pollution will caused the houses to sell at lower price, accounted 20.8% less than houses located in area without noise interference. Apparently, the noise produced by the aircraft has even larger negative impact on house prices as compared to road traffic noise and railway noise. This study adopted quantitative approach in answering the objective of the paper. The findings were based on the secondary data which including 210 property transaction data within year 2015. The range of areas for this study was limited to selected residential terrace houses that located within 10.0 km from Kuching International Airport (KIA). The findings from Multiple Regression Analysis (MRA) shows that the property prices located nearer to the airport (<2.5 km from KIA) in selected case study areas have been sold with lower price. Moreover, the prices of the properties located distance from KIA were not negatively impacted by the aircraft noise due to the other pulling factor that has larger impact to the property. Indeed, the location of the property, public amenities, transportation system, neighborhood factor and facilities also has close relationship to the property price.


2017 ◽  
Vol 27 (1) ◽  
pp. e10
Author(s):  
R.M. Bruno ◽  
U. Faraguna ◽  
M. Di Pilla ◽  
M. Di Galante ◽  
T. Banfi ◽  
...  

2021 ◽  
Vol 20 (1) ◽  
Author(s):  
Aina Roca-Barceló ◽  
Adelaide Nardocci ◽  
Breno Souza de Aguiar ◽  
Adeylson G. Ribeiro ◽  
Marcelo Antunes Failla ◽  
...  

Abstract Background Noise pollution is increasingly recognised as a public health hazard, yet limited evidence is available from low- and middle-income countries (LMIC), particularly for specific sources. Here, we investigated the association between day-night average (Ldn) aircraft noise and the risk of death due to cardiovascular disease (CVD), stroke and coronary heart disease (CHD) at small-area level around São Paulo‘s Congonhas airport, Brazil during the period 2011–2016. Methods We selected 3259 census tracts across 16 districts partially or entirely exposed to ≥50 dB aircraft noise levels around the Congonhas airport, using pre-modelled 5 dB Ldn noise  bands (≤50 dB to > 65 dB). We estimated the average noise exposure per census tract using area-weighting. Age, sex and calendar year-specific death counts for CVD, stroke and CHD were calculated by census tract, according to the residential address at time of death. We fitted Poisson regression models to quantify the risk associated with aircraft noise exposure, adjusting for age, sex, calendar year and area-level covariates including socioeconomic development, ethnicity, smoking and road traffic related noise and air pollution. Results After accounting for all covariates, areas exposed to the highest levels of noise (> 65 dB) showed a relative risk (RR) for CVD and CHD of 1.06 (95% CI: 0.94; 1.20) and 1.11 (95%CI: 0.96; 1.27), respectively, compared to those exposed to reference noise levels (≤50 dB). The RR for stroke ranged between 1.05 (95%CI: 0.95;1.16) and 0.91 (95%CI: 0.78;1.11) for all the noise levels assessed. We found a statistically significant positive trend for CVD and CHD mortality risk with increasing levels of noise (p = 0.043 and p = 0.005, respectively). No significant linear trend was found for stroke. Risk estimates were generally higher after excluding road traffic density, suggesting that road traffic air and noise pollution are potentially important confounders. Conclusions This study provides some evidence that aircraft noise is associated with increased risk of CVD and CHD mortality in a middle-income setting. More research is needed to validate these results in other LMIC settings and to further explore the influence of residual confounding and ecological bias. Remarkably, 60% of the study population living near the Congonhas airport (~ 1.5 million) were exposed to aircraft noise levels > 50 dB, well above those recommended by the WHO (45 dB), highlighting the need for public health interventions.


2013 ◽  
Vol 726-731 ◽  
pp. 1037-1040
Author(s):  
Dun Niu ◽  
Yun Feng Ma ◽  
Lei Zhu ◽  
Zheng Liu ◽  
Xiao Min Hu

Based on the environmental impact assessment automation system (C4ISRE) and environmental remote sensing technology, integrating virtual reality simulation method, the environmental impact assessment of aircraft noise pollution was simulated by system simulation. Took Environmental Impact Assessment for new school site of Northeastern University for example, the simulation for environmental impact assessment of noise pollution was realized, which provided an effective way to further study Informatization Environmental Assessment.


Circulation ◽  
2020 ◽  
Vol 141 (Suppl_1) ◽  
Author(s):  
Jason M Collins ◽  
Rahul Gondalia ◽  
Anne E Justice ◽  
Katelyn Holliday ◽  
James Stewart ◽  
...  

Background: Noise pollution is common and can affect health, but mechanisms underlying this relationship remain incompletely characterized. We therefore examined the novel association between aircraft noise and DNA methylation (DNAm), an environmentally modifiable epigenetic phenomenon that affects gene expression and is associated with cardiovascular and neurological disease. Methods: We conducted a methylome-wide association study in race/ethnicity-stratified subpopulations of 4,535 post-menopausal Women’s Health Initiative participants (mean age: 64.4 years; 59.6% white; 24.7% African American; 15.7% Hispanic/Latina). We estimated DNAm in whole blood as a proportion of methylated cytosines at each Cytosine-phosphate-Guanine (CpG) site using the Illumina 450k BeadChip. In cooperation with the Federal Aviation Administration, we generated day-night-average sound level (DNL) contours around 90 major U.S. airports using the Aviation Environmental Design Tool. The DNL applies a penalty for nighttime exposure and is the primary metric for informing policy in the U.S. We estimated geocoded participant address-specific, annual, DNL aircraft noise exposures in decibels (dB) from the contours on the day of blood draw. We categorized aircraft noise exposures as non-exposed or exposed (DNL < 45 or ≥ 45dB) and used linear mixed-effects models to estimate CpG site-specific DNAm-noise associations adjusted for sociodemographic, behavioral, and technical covariates. We combined site-specific associations across race-ethnicity strata in fixed-effects, inverse variance-weighted meta-analyses and then identified the significant ( P < 1.0x10 -7 ), non-heterogeneous ( P Cochran’s Q > 0.10) associations among them. Results: Overall, DNAm was inversely associated with aircraft noise at cg21525369 (Chromosome 15; P = 6.40x10 -8 ). Cg21525369 is 56 kb upstream from ACAN , a gene encoding an extracellular matrix protein (aggrecan). Aggrecan is expressed in response to ultrasound; forms perineural nets around cochlear hair cells and auditory brainstem neurons; affects afferent extension, synaptic transmission, and neuronal vulnerability; deposits in tympanic membranes of patients with sensorineural hearing loss, and redistributes in auditory brainstem after deafferentation. Conclusions: The biologically plausible DNAm-noise association among postmenopausal U.S. women suggests aircraft noise pollution may affect methylation of DNA near a gene previously associated with hearing, although its cardiovascular, otologic, and neurological implications remain unclear pending sensitivity analysis, external replication, mediation analysis, and functional characterization.


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