scholarly journals The Role of Pubertal Timing in the Development of Peer Victimization and Offending From Early- to Mid-Adolescence

2021 ◽  
pp. 027243162110022
Author(s):  
Therése Skoog ◽  
Sabina Kapetanovic
Keyword(s):  
Peer Victimization ◽  
Peer Relations ◽  
Negative Impact ◽  
Developmental Trajectories ◽  
Pubertal Timing ◽  
Growth Curve Analysis ◽  
Early Puberty ◽  
Early Pubertal Timing ◽  
Latent Growth Curve Analysis

We used latent growth curve analysis to extend research on associations between early puberty and adverse peer relations by examining the role of pubertal timing in the developmental trajectories of peer victimization and offending from early- to mid-adolescence. We made use of three-wave longitudinal data collected annually from a cohort of Swedish adolescents ( N = 1,515, 51% girls, [Formula: see text]age at T1 = 13.0 years). The results revealed negative developmental trends for peer victimization and offending. Early pubertal timing was linked to higher initial levels and a steeper decrease of peer victimization and offending. The only effect of pubertal timing that differed between the genders was that the initial level of offending was stronger for boys than girls. In conclusion, the negative impact of early pubertal timing on peer victimization and offending occurs in the early stages of adolescence and disappears thereafter.

Longer Exposure to Obesity, Slimmer Chance of College? Body Weight Trajectories, Non-Cognitive Skills, and College Completion

2016 ◽  
Vol 49 (2) ◽  
pp. 203-227 ◽  
Author(s):  
Yen-hsin Alice Cheng
Keyword(s):  
Body Weight ◽  
Cognitive Skills ◽  
Negative Impact ◽  
Developmental Trajectories ◽  
College Completion ◽  
Interaction Terms ◽  
Cognitive Traits ◽  
Weight Trajectories ◽  
The Impact

The NLSY97 data were used to explore the patterns of developmental trajectories of body weight in adolescence and how they affected the likelihood of college completion in young adulthood among 2,275 youths aged 13 and 14 in Wave 1. A strong weight trajectory gradient was found for rates of college completion. The study further explored the role of non-cognitive traits in the association between weight trajectories and college attainment. Non-cognitive traits were found to partially mediate the impact of certain weight trajectories on the likelihood of college completion. Some moderating effects of conscientiousness were also found. The findings from the gender and weight trajectory interaction terms showed that a stronger negative impact of weight trajectory on college completion is only observed for women in the late-teen-onset overweight group. This study highlights the importance of using a longitudinal weight measure and the role of non-cognitive traits in adolescent obesity research.

scholarly journals Cortisol Response to Psychosocial Stress in Chinese Early Puberty Girls: Possible Role of Depressive Symptoms

2015 ◽  
Vol 2015 ◽  
pp. 1-5 ◽  
Author(s):  
Ying Sun ◽  
Fang Deng ◽  
Yang Liu ◽  
Fang-Biao Tao
Keyword(s):  
Depressive Symptoms ◽  
Salivary Cortisol ◽  
Social Stress ◽  
Stress Reactivity ◽  
Pubertal Timing ◽  
Stress Test ◽  
Cortisol Concentration ◽  
Early Puberty ◽  
Cortisol Reactivity

Objective. The present study aimed at investigating unique patterns of salivary cortisol reactivity and recovery in response to a social stressor among girls with early puberty and exploring possible role of depressive symptom in this association.Design. Case-control study.Patients. Fifty-six girls with early puberty and age- and body mass index- (BMI-) matched normal puberty controls(n=56)were selected.Measurements. Salivary cortisol was measured in response to the Groningen Social Stress Test for Children.Results. Girls with early puberty had higher cortisol concentration at the end of the GSST (C3), cortisol concentration 20 min after the end of the GSST (C4), and AUC increment (AUCi) compared to non-early puberty girls. Depressive symptoms correlated with blunted HPA reactivity among girls with early puberty.Conclusion. This study demonstrated the disturbance effect of objectively examined early pubertal timing on HPA axis responses. It also suggested that stress reactivity might be blunted for individuals with depressive symptoms.

scholarly journals Parenting Styles as a Moderator of the Association between Pubertal Timing and Chinese Adolescents’ Smoking Behavior

2021 ◽  
Vol 18 (17) ◽  
pp. 8903
Author(s):  
Hui Ling ◽  
Yaqin Yan ◽  
En Fu ◽  
Amin Zhu ◽  
Jianren Zhang ◽  
...  
Keyword(s):  
Parenting Style ◽  
Pubertal Timing ◽  
Smoking Behavior ◽  
Adolescent Smoking ◽  
Chinese Adolescents ◽  
Chinese Version ◽  
Negative Outcomes ◽  
Early Pubertal Timing ◽  
The Relationship

Background: Pubertal timing refers to the timing of an individual regarding pubertal sexual maturation, both physiologically and psychologically. Existing research shows that pubertal timing is associated with behavioral problems. This study investigated the role of parenting style in the relationship between pubertal timing and Chinese adolescents’ smoking behavior. Methods: The study examined the association of pubertal timing, parenting style and adolescents’ smoking behavior, using the Pubertal Development Scale (Chinese version), Simplified Parenting Style Scale-Chinese version, and three items related to adolescents’ smoking situation. Participants were 1391 Chinese adolescents aged 11–16 years old (53.41% boys). Hierarchical linear regression analyses assessed the moderating role of parenting style on the association between pubertal timing and adolescent smoking behavior. Results: The results indicated that parenting style moderates the relationship between pubertal timing and adolescent smoking behavior. For male adolescents, father rejection moderated the relationship between early pubertal timing and smoking behavior. For female adolescents, father rejection, father emotional warmth, and mother emotional warmth moderated the relationship between pubertal timing and smoking behavior. Conclusions: Findings from the study highlight the importance of parenting style, which may influence the negative outcomes associated with early pubertal timing and can help improve interventions aimed at reducing these negative outcomes.

Pubertal timing and depressive symptoms in late adolescence: The moderating role of individual, peer, and parental factors

2013 ◽  
Vol 25 (2) ◽  
pp. 455-471 ◽  
Author(s):  
Amelie Benoit ◽  
Eric Lacourse ◽  
Michel Claes
Keyword(s):  
Depressive Symptoms ◽  
Pubertal Timing ◽  
Emotional Problems ◽  
Depression Symptoms ◽  
Parental Rejection ◽  
Deviant Peer Affiliation ◽  
Early Puberty ◽  
Peer Affiliation ◽  
Moderating Role

AbstractThis longitudinal study examined personal-accentuation and contextual-amplification models of pubertal timing. In these models, individual and contextual risk factors during childhood and adolescence can magnify the effects of early or late puberty on depression symptoms that occur years later. The moderating role of prepubertal individual factors (emotional problems in late childhood) and interpersonal factors (deviant peer affiliation, early dating, perceived peer popularity, and perceived parental rejection during adolescence) were tested. A representative sample of 1,431 Canadian adolescents between 10–11 and 16–17 years of age was followed biannually. In line with the personal-accentuation model, early puberty has been shown to be a predictor for depression in both girls and boys who presented emotional problems in childhood. This effect was also noted for late maturing boys. Consistent with the contextual-amplification model, early puberty predicted later depression in youth who perceived greater parental rejection. Interpersonal experiences such as early dating in girls and deviant peer affiliation in boys predicted depression in early maturers as well. For girls, early dating was also found to be amplified by childhood emotional problems. In line with biopsychosocial models, results indicate that the effect of pubertal timing on depressive symptoms must be conceptualized through complex interactions between characteristics of adolescents' interpersonal relationships and prepubertal vulnerabilities.

scholarly journals Childhood adversity, pubertal timing and self-harm: a longitudinal cohort study

2021 ◽  
pp. 1-9
Author(s):  
Abigail Emma Russell ◽  
Carol Joinson ◽  
Elystan Roberts ◽  
Jon Heron ◽  
Tamsin Ford ◽  
...  
Keyword(s):  
Structural Equation ◽  
Pubertal Timing ◽  
Childhood Adversity ◽  
Height Velocity ◽  
Age At Menarche ◽  
Peak Height Velocity ◽  
Early Puberty ◽  
Increased Risk ◽  
Early Pubertal Timing ◽  
Self Harm

Abstract Background The occurrence of early childhood adversity is strongly linked to later self-harm, but there is poor understanding of how this distal risk factor might influence later behaviours. One possible mechanism is through an earlier onset of puberty in children exposed to adversity, since early puberty is associated with an increased risk of adolescent self-harm. We investigated whether early pubertal timing mediates the association between childhood adversity and later self-harm. Methods Participants were 6698 young people from a UK population-based birth cohort (ALSPAC). We measured exposure to nine types of adversity from 0 to 9 years old, and self-harm when participants were aged 16 and 21 years. Pubertal timing measures were age at peak height velocity (aPHV – males and females) and age at menarche (AAM). We used generalised structural equation modelling for analyses. Results For every additional type of adversity; participants had an average 12–14% increased risk of self-harm by 16. Relative risk (RR) estimates were stronger for direct effects when outcomes were self-harm with suicidal intent. There was no evidence that earlier pubertal timing mediated the association between adversity and self-harm [indirect effect RR 1.00, 95% confidence interval (CI) 1.00–1.00 for aPHV and RR 1.00, 95% CI 1.00–1.01 for AAM]. Conclusions A cumulative measure of exposure to multiple types of adversity does not confer an increased risk of self-harm via early pubertal timing, however both childhood adversity and early puberty are risk factors for later self-harm. Research identifying mechanisms underlying the link between childhood adversity and later self-harm is needed to inform interventions.

Female pubertal timing and problem behaviour

2013 ◽  
Vol 37 (4) ◽  
pp. 357-365 ◽  
Author(s):  
Therése Skoog ◽  
Håkan Stattin ◽  
Zdena Ruiselova ◽  
Metin Özdemir
Keyword(s):  
Adolescent Girls ◽  
Pubertal Timing ◽  
Problem Behaviour ◽  
Early Puberty ◽  
Two Cultures ◽  
Peer Socialization ◽  
Problem Behaviours ◽  
Early Maturing ◽  
Developmental Significance

We tested the peer-socialization/contextual-amplification explanation for the link between early female puberty and problem behaviour. We propose that in cultures with high tolerance for adolescent heterosexual involvement, early puberty should be linked with problem behaviour—not in other cultures. We compared girls in two cultures (Slovakia and Sweden) that differ in acceptance of adolescent girls’ heterosexual involvement. Findings supported the hypothesis by showing that in Sweden, a culture that facilitates adolescent heterosexual involvement, early-maturing girls reported more problem behaviours than in Slovakia. The mediation link (heterosexual involvement as the mechanism linking early puberty with problem behaviour) was moderated by culture. The findings expand our understanding of the role of macro-cultural contexts in the developmental significance of female puberty.

scholarly journals GENETICS IN ENDOCRINOLOGY: Genetic etiologies of central precocious puberty and the role of imprinted genes

2020 ◽  
Vol 183 (4) ◽  
pp. R107-R117
Author(s):  
Stephanie A Roberts ◽  
Ursula B Kaiser
Keyword(s):  
Precocious Puberty ◽  
Pubertal Timing ◽  
Association Studies ◽  
Hypogonadotropic Hypogonadism ◽  
Central Precocious Puberty ◽  
Imprinted Genes ◽  
Genome Wide Association Studies ◽  
Early Puberty ◽  
Imprinted Gene

Pubertal timing is regulated by the complex interplay of genetic, environmental, nutritional and epigenetic factors. Criteria for determining normal pubertal timing, and thus the definition of precocious puberty, have evolved based on published population studies. The significance of the genetic influence on pubertal timing is supported by familial pubertal timing and twin studies. In contrast to the many monogenic causes associated with hypogonadotropic hypogonadism, only four monogenic causes of central precocious puberty (CPP) have been described. Loss-of-function mutations in Makorin Ring Finger Protein 3(MKRN3), a maternally imprinted gene on chromosome 15 within the Prader–Willi syndrome locus, are the most common identified genetic cause of CPP. More recently, several mutations in a second maternally imprinted gene, Delta-like noncanonical Notch ligand 1 (DLK1), have also been associated with CPP. Polymorphisms in both genes have also been associated with the age of menarche in genome-wide association studies. Mutations in the genes encoding kisspeptin (KISS1) and its receptor (KISS1R), potent activators of GnRH secretion, have also been described in association with CPP, but remain rare monogenic causes. CPP has both short- and long-term health implications for children, highlighting the importance of understanding the mechanisms contributing to early puberty. Additionally, given the role of mutations in the imprinted genes MKRN3 and DLK1 in pubertal timing, other imprinted candidate genes should be considered for a role in puberty initiation.

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