Infantile Water Intoxication After a Swimming Lesson

PEDIATRICS ◽  
1982 ◽  
Vol 70 (4) ◽  
pp. 599-600 ◽  
Author(s):  
Gerald N. Goldberg ◽  
Elmer S. Lightner ◽  
Wayne Morgan ◽  
Sid Kemberling
Keyword(s):  
Intracranial Pressure ◽  
Signs And Symptoms ◽  
Water Intoxication ◽  
Increased Intracranial Pressure ◽  
Accidental Ingestion ◽  
Near Drowning ◽  
Early Infant

This report emphasizes a danger inherent in early infant swimming programs, ie, the need to observe the infant closely for ingestion of excessive amounts of water. A physician faced with a history compatible with near-drowning or possible aspiration of water must consider the diagnosis of acute water intoxication. Accidental ingestion of water can produce hyponatremia with resulting signs and symptoms of acute water intoxication, ie, lethargy, vomiting, seizures, and increased intracranial pressure.

Aggressive Cranial Vault Decompression for Cranial Hyperostosis: Technical Case Report of Two Cases

2005 ◽  
Vol 57 (suppl_1) ◽  
pp. E212-E212 ◽  
Author(s):  
J. Mocco ◽  
Ricardo J. Komotar ◽  
Brad E. Zacharia ◽  
Neil A. Feldstein ◽  
Jeffrey N. Bruce
Keyword(s):  
Intracranial Pressure ◽  
Signs And Symptoms ◽  
Operative Management ◽  
Cranial Vault ◽  
Increased Intracranial Pressure ◽  
The Past ◽  
Cranial Nerve Dysfunction ◽  
Surgical Options ◽  
Nerve Dysfunction ◽  
Clinically Significant

Abstract OBJECTIVE AND IMPORTANCE: Camurati-Engelmann's disease, also known as progressive diaphysial dysplasia, is a disorder of the bone metabolism. Neurological manifestations of progressive diaphysial dysplasia include cranial nerve dysfunction, generalized weakness, cerebellar herniation, and increased intracranial pressure. In the past, surgical intervention has been of limited and temporary benefit. We present two patients with cranial hyperostosis secondary to Camurati-Engelmann's disease who were treated successfully with a single surgery involving a combination of multiple craniotomies for cranial vault decompression. CLINICAL PRESENTATION: Two patients presented with signs and symptoms of increased intracranial pressure secondary to Camurati-Engelmann's syndrome. Radiological workup revealed marked cranial hyperostosis. INTERVENTION: The patients underwent aggressive cranial vault decompression. Multiple craniotomies were performed, and the inner table was then drilled down until the bone was 1 cm thick. CONCLUSION: Effective surgical options are needed for clinically significant cranial hyperostosis. In an effort to further define operative management in these patients, we describe a single, aggressive surgical procedure that may be used for successful cranial decompression.

scholarly journals Pediatric Idiopathic Intracranial Hypertension Post-measles Vaccine: A Case Report

2021 ◽  
Author(s):  
Ahmed Arafa Helaly ◽  
Ahmed Ezzat ◽  
Mohammed Gamal Abdellatif
Keyword(s):  
Intracranial Pressure ◽  
Intracranial Hypertension ◽  
Idiopathic Intracranial Hypertension ◽  
Signs And Symptoms ◽  
Normal Brain ◽  
Measles Vaccine ◽  
Increased Intracranial Pressure ◽  
Csf Pressure ◽  
General Signs ◽  
Sixth Nerve

Idiopathic intracranial hypertension is characterized by an increase in intracranial pressure with no significant structural cause. The diagnostic criteria include general signs and symptoms of generalized increased intracranial pressure or papilledema, increased Cerebrospinal Fluid (CSF) pressure (>250 mm H2 O), and normal brain imaging. It usually occurs in adults but rarely can be seen in children. We report a 7-year-old child with a manifestation of increased intracranial tension after the measles vaccine, associated with bilateral sixth nerve palsy and bilateral facial palsy with lower motor neuron features. Normal magnetic resonance imaging and CSF results were normal, except for an increase in CSF pressure. The symptoms began to be resolved after lumbar puncture with the complete resolution of symptoms after one and half months. We add also acetazolamide at the proper dose to reduce intracranial pressure from the first day of diagnosis.

scholarly journals Central neurocytomas of uncommon locations: report of two cases

2006 ◽  
Vol 64 (4) ◽  
pp. 1015-1018 ◽  
Author(s):  
Francinaldo Lobato Gomes ◽  
Luciano Ricardo França ◽  
Samuel Tau Zymberg ◽  
Sérgio Cavalheiro
Keyword(s):  
Differential Diagnosis ◽  
Intracranial Pressure ◽  
Obstructive Hydrocephalus ◽  
Signs And Symptoms ◽  
Pineal Region ◽  
Additional Treatment ◽  
Third Ventriculostomy ◽  
Increased Intracranial Pressure ◽  
The Brain ◽  
Uncommon Location

We report two patients with central neurocytomas at an uncommon location in the brain. The first, a 58-year-old man presenting with signs and symptoms of increased intracranial pressure, had a tumor located at the pineal region. The second, a 21-year-old woman with tumor in the aqueductal region had worsening migraine-like headaches and diplopia. Both patients had obstructive hydrocephalus treated by neuroendoscopic third ventriculostomy and biopsy of the tumors. No additional treatment was done. We conclude that neurocytomas should be considered in the differential diagnosis of tumors located in the pineal and aqueductal regions.

scholarly journals Mechanisms of visual loss in papilledema

2007 ◽  
Vol 23 (5) ◽  
pp. E5 ◽  
Author(s):  
Clemens M. Schirmer ◽  
Thomas R. Hedges
Keyword(s):  
Intracranial Pressure ◽  
Visual Loss ◽  
Treatment Options ◽  
Signs And Symptoms ◽  
Illustrative Case ◽  
Potential Treatment ◽  
Increased Intracranial Pressure ◽  
Intracranial Mass Lesion ◽  
Optic Discs

✓ The term “papilledema” indicates swelling of the optic discs secondary to increased intracranial pressure. Papilledema can be caused by an intracranial mass lesion or by other factors. Visual symptoms frequently accompany papilledema, which can lead to permanent visual loss if left untreated. Starting with an illustrative case, the authors review the pathophysiology of the visual signs and symptoms of papilledema. They also briefly review potential treatment options, focusing on the role of the neurosurgeon in the treatment of patients with papilledema.

Systemic Weber-Christian disease presenting as an intracranial mass lesion

1980 ◽  
Vol 52 (1) ◽  
pp. 134-137 ◽  
Author(s):  
John R. Mangiardi ◽  
Z. Harry Rappaport ◽  
Joseph Ransohoff
Keyword(s):  
Intracranial Pressure ◽  
Signs And Symptoms ◽  
Mass Lesion ◽  
Increased Intracranial Pressure ◽  
Content Type ◽  
Intracranial Mass ◽  
Intracranial Mass Lesion ◽  
Fine Print

✓ A case is reported in which systemic Weber-Christian disease presented as a dural mass causing the signs and symptoms of increased intracranial pressure. The literature and possible pathogenesis of this entity are discussed.

scholarly journals Evaluation of cerebrovascular hemodynamics in patients with idiopathic intracranial hypertension using transcranial Doppler

2020 ◽  
Vol 56 (1) ◽  
Author(s):  
Mahmoud M. Allam ◽  
Hatem A. Almasry ◽  
Sandra M. Ahmed ◽  
Youssuf G. Taha ◽  
Mohammed I. Oraby
Keyword(s):  
Intracranial Pressure ◽  
Intracranial Hypertension ◽  
Lumbar Puncture ◽  
Transcranial Doppler ◽  
Idiopathic Intracranial Hypertension ◽  
Pulsatility Index ◽  
Research Work ◽  
Increased Intracranial Pressure ◽  
Significant Relation ◽  
Before And After

Abstract Background Idiopathic intracranial hypertension (IIH) is a disorder of increased intracranial pressure without a clear cause and can have serious visual effects. Previous research work suggests that transcranial Doppler measurements of pulsatility index correlate accurately with elevated intracranial pressure. Objective To assess the cerebrovascular hemodynamic changes in patients with IIH using transcranial Doppler before and after lumbar puncture and CSF withdrawal. Methods An interventional study conducted on 40 patients (31 females and 9 males) fulfilling the modified Dandy criteria for diagnosis of idiopathic intracranial hypertension, MRI brain, and MRV was done to the patients. Lumbar puncture was done for all included patients to measure intracranial pressure and CSF withdrawal. Transcranial Doppler was performed for all included before and after lumbar puncture and CSF withdrawal and the following parameters were measured: peak systolic velocity (PSV), end diastolic velocity (EDV), resistivity index (RI), and pulsatility index (PI). Results Significant relation was found between grades of papilledema and PSV, RI, and PI (p value 0.012, 0.025, 0.016) but no significant relation was found between grades of papilledema and EDV (0.102). Significant changes occurred in parameters of TCD pre- and post-CSF withdrawal including PSV, EDV, and PI (p value 0.001, 0.015, 0.019) denoting a significant change in cerebral hemodynamics after CSF withdrawal which denotes a decrease in intracranial pressure. Conclusion Increased intracranial pressure significantly affects cerebral blood flow. A normalization of transcranial Doppler parameters occurs following lowering of intracranial pressure through lumbar puncture and CSF withdrawal.

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