scholarly journals Evészavartünetek, problematikus evési magatartás egyes kamaszkori krónikus betegségekben

2020 ◽  
Vol 161 (44) ◽  
pp. 1872-1876
Author(s):  
Melinda Cserép ◽  
Irena Szumska
Keyword(s):  
Eating Disorder ◽  
Disordered Eating ◽  
Chronic Illnesses ◽  
Food Sources ◽  
Adequate Treatment ◽  
Negative Body Image ◽  
Bowel Diseases ◽  
Dietary Regime ◽  
Inflammatory Bowel ◽  
Complications And Mortality

Összefoglaló. A táplálkozás iránti figyelmet igénylő krónikus betegségek (így a coeliakia, az 1-es típusú diabetes és a gyulladásos bélbetegségek), valamint az evészavartünetek kapcsolata egyelőre kevéssé széleskörűen tanulmányozott terület serdülők körében. Az eddigi kutatási eredmények kiemelik a testkép- és evészavartünetek iránti magasabb kockázatot ezekben a betegségekben. Rizikótényező lehet az evési szokások megváltozása, a táplálkozásra irányuló fokozott figyelem és az ételekkel, valamint az elfogyasztásuk következményeivel kapcsolatos aggodalom. A korai felismerést nehezíti, hogy az evészavartünetek sokáig rejtve maradhatnak, és átfedést mutathatnak a krónikus betegség egyes tüneteivel (például testsúlyváltozás, hasmenés, hányás, hasfájás). A fennálló evészavar-érintettség gyengítheti a krónikus betegség hatékony terápiáját, súlyosbíthatja a szövődményeket, és növelheti a mortalitási rizikót. A tünetek korai felismerése és az adekvát terápia csak multidiszciplináris szemléletben és teammunkával, a szomatikus gondozás és a pszichoterápia összehangolásával lehet eredményes. Orv Hetil. 2020; 161(44): 1872–1876. Summary. Disordered eating is not a well-understood phenomenon in diet-related chronic illnesses (e.g., celiac disease, type 1 diabetes, inflammatory bowel diseases) among adolescents. Previous research found a higher risk for eating disorders and negative body image among these patients. Following the prescribed or suggested dietary regime may lead to increased food awareness and become an eating disorder risk factor. Further risks may be the altered eating patterns, the fear from unknown food sources and its consequences. In many cases, eating disorder characteristics may be hidden and the symptoms of chronic illness (e.g., changes in weight, diarrhoea, vomiting, abdominal pain) and disordered eating can mimic each other. Disordered eating can worsen the effective therapy of physical illnesses and increase complications and mortality. Early diagnosis and adequate treatment can only be provided through multidisciplinary approach and teamwork. Orv Hetil. 2020; 161(44): 1872–1876.

Sa574 RISK FACTORS FOR DISORDERED EATING AMONG PATIENTS WITH INFLAMMATORY BOWEL DISEASES

2021 ◽  
Vol 160 (6) ◽  
pp. S-557-S-558
Author(s):  
Gianna Stoleru ◽  
Andrew R. Leopold ◽  
Amanda Auerbach ◽  
Shelley Nehman ◽  
Uni Wong
Keyword(s):  
Risk Factors ◽  
Disordered Eating ◽  
Inflammatory Bowel Diseases ◽  
Bowel Diseases ◽  
Inflammatory Bowel

Eating disorder or disordered eating: undiagnosed inflammatory bowel disease mimicking eating disorder

2018 ◽  
Vol 104 (10) ◽  
pp. 1004-1006
Author(s):  
Rachel Elizabeth Harris ◽  
Rachel Tayler ◽  
Richard K Russell
Keyword(s):  
Inflammatory Bowel Disease ◽  
Eating Disorder ◽  
Disordered Eating ◽  
Bowel Disease ◽  
Eating Habits ◽  
Delayed Diagnosis ◽  
Important Differential Diagnosis ◽  
Gi Symptoms ◽  
Incorrect Diagnosis ◽  
Inflammatory Bowel

We describe the case of a patient with ongoing weight loss, low mood and previously undisclosed gastrointestinal (GI) symptoms initially diagnosed with an eating disorder and subsequently diagnosed with ulcerative colitis over a year following initial presentation. This patient exhibited disordered eating secondary to the worsening symptoms of undiagnosed inflammatory bowel disease (IBD) and had altered her eating habits to reduce the diarrhoea and rectal bleeding she was experiencing, contributing to significant weight loss.The implications of a delayed diagnosis of IBD or incorrect diagnosis of eating disorder are severe both physically and psychologically. We discuss factors in the assessment of patients which may raise suspicion of organic GI disease such as IBD—an important differential diagnosis in those with non-specific GI symptoms and suspected eating disorder—and highlight baseline investigations which should be performed to ensure a diagnosis of IBD is not missed in these patients.

scholarly journals Gastrointestinalis manifesztációk monogénes primer immundefektusokban

2018 ◽  
Vol 159 (49) ◽  
pp. 2050-2056
Author(s):  
Gábor Veres ◽  
Zsuzsanna Gaál
Keyword(s):  
Inflammatory Bowel Disease ◽  
Bowel Disease ◽  
Early Onset ◽  
Genetic Alterations ◽  
Epigenetic Alterations ◽  
Adequate Treatment ◽  
Next Generation Sequencing Technology ◽  
Bowel Diseases ◽  
Continuous Interaction ◽  
Inflammatory Bowel

Abstract: Although very early onset inflammatory bowel disease that develops in early childhood (before the age of 6 years) has a different etiology from Crohn’s disease and ulcerative colitis, it is also characterized by chronic inflammation of the gastrointestinal tract. Basically, very early onset inflammatory bowel disease should be considered as an immunodeficiency with monogenic origin where both gastrointestinal manifestations and symptoms of immunodeficiencies may develop in variable combinations. However, in the future, the evaluation of genetic alterations in the background of the disease will probably be performed by next-generation sequencing technology; one should also consider that the sequence of the DNA stands in continuous interaction with a wide variety of environmental effects, among which nutrition should be emphasized by all means. Epigenetic alterations that are induced by environmental factors, could contribute to the pathogenesis of inflammatory bowel diseases that develop during childhood, therefore, they should also be identified during further research. It has a key significance to establish the diagnosis of very early onset inflammatory bowel disease as early as possible, because this could give the opportunity to start the adequate treatment which is bone marrow transplantation in the case of monogenic immunodeficiencies. Orv Hetil. 2018; 159(49): 2050–2056.

Localization and Activity of Inflammatory Bowel Disease Using 111ln Leukocyte Imaging

1988 ◽  
Vol 27 (03) ◽  
pp. 83-86 ◽  
Author(s):  
B. Briele ◽  
F. Wolf ◽  
H. J. Biersack ◽  
F. F. Knapp ◽  
A. Hotze
Keyword(s):  
Inflammatory Bowel Disease ◽  
Crohn’S Disease ◽  
Crohn's Disease ◽  
Disease Activity ◽  
Bowel Disease ◽  
Cell Uptake ◽  
Disease Extent ◽  
Bowel Diseases ◽  
Inflammatory Bowel ◽  
Leukocyte Imaging

A prospective study was initiated to compare the clinically proven results concerning localization/extent and activity of inflammatory bowel diseases with those of 111ln-oxine leukocyte imaging. All patients studied were completely examined with barium enema x-ray, clinical and laboratory investigations, and endoscopy with histopathology. A total of 31 leukocyte scans were performed in 15 patients (12 with Crohn’s disease, 3 with ulcerative colitis). The scans were graded by comparing the cell uptake of a lesion (when present) and a bone marrow area providing a count ratio (CR). The inflammatory lesions were correctly localized on 26 leukocyte scans, and in 21 scans the scintigraphically estimated extent of disease was identical to endoscopy. In 5 cases the disease extent was underestimated, 4 scans in patients with relapse of Crohn’s disease were falsely negative, and in one patient with remission truly negative. The scintigraphically assessed disease activity was also in a good agreement with clinical disease activity based on histopathology in all cases. We conclude that leukocyte imaging provides valuable information about localization and activity of inflammatory bowel disease.

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