The formation of an artificial vagina by Baldwin's method

1930 ◽  
Vol 26 (2) ◽  
pp. 218-218
Author(s):  
I. F. Kharitonov
Keyword(s):  
Small Intestine ◽  
Mucous Membrane ◽  
Satisfactory Result ◽  
Congenital Absence ◽  
Near Future ◽  
Artificial Vagina

Session 4 / XII 1929 Dr. I. F. Kharitonov (speaker Dr. A. N. Syzganov). The formation of an artificial vagina by Baldwin's method with a completely satisfactory result for the near future was produced by the operator in one case of congenital absence of this organ; the method is not safe, fraught with complications; in the reported case, the mucous membrane of the newly formed vagina continues to secrete juice from the small intestine, which strongly irritates the surrounding tissues.

scholarly journals Intestinal epithelial dysplasia

2020 ◽  
pp. 91-94
Author(s):  
T. E. Taranushenko ◽  
E. M. Vasilyeva ◽  
E. V. Antsiferova ◽  
L. N. Karpova ◽  
Y. S. Shamova
Keyword(s):  
Small Intestine ◽  
Rare Disease ◽  
Histological Examination ◽  
Mucous Membrane ◽  
Clinical Case ◽  
Clinical Presentation ◽  
Epithelial Dysplasia ◽  
Intestinal Epithelial ◽  
Electronic Microscopy

The article presents a clinical case of intestinal epithelial dysplasia (tufting enteropathy), a rare disease which relates to congenital diarrheas, has a particular clinical presentation and is life-threating. The diagnosis was confirmed by histological examination and electronic microscopy of mucous membrane of small intestine. Additional evidence is substantiated for the existence of prenatal markers of intestinal epithelial dysplasia represented as polyhydramnios and intrauterine dilatation of fetal intestinal loops. These symptoms are considered to be a clinical hallmark of sodium and chloride diarrheas and are described in single cases in enteropathy called a microvillus inclusion disease.

Determination of the fat content in the pancreas and mucous membrane (mucosa) of the small intestine of pigs

1979 ◽  
Vol 13 (5) ◽  
pp. 546-548
Author(s):  
V. P. Pakhomov ◽  
N. N. Evgrafova ◽  
V. T. Sapleva
Keyword(s):  
Small Intestine ◽  
Mucous Membrane ◽  
Fat Content

scholarly journals MORPHOLOGICAL CHANGES IN THE SMALL INTESTINE OF RATS WITH ACUTE GENERALIZED PERITONITIS

2019 ◽  
pp. 110-113
Author(s):  
I. Ya. Dzyubanovsky ◽  
B. M. Vervega ◽  
S. R. Pіdruchna ◽  
N. A. Melnyk
Keyword(s):  
Endothelial Cells ◽  
Small Intestine ◽  
Mucous Membrane ◽  
Morphological Changes ◽  
Histological Study ◽  
Abdominal Cavity ◽  
Inflammatory Infiltration ◽  
Generalized Peritonitis ◽  
White Rats ◽  
Fecal Suspension

The main cause of mortality in acute generalized peritonitis (AGP) is the development of multiple organ insufficiency. The intestine is the organ where the first changes develop in this pathology. The aim of the study – to research and evaluate the morphological changes in the small intestine wall of animals with experimental AGP. Materials and Methods. 32 white rats were used in this study. Acute peritonitis was modeled by introduction of 10 % fecal suspension in the dosage of 0.5 ml per 100 g of the animal's weight into the abdominal cavity of rats by puncture. The terms of observation: the 1st, 3rd and the 7th days from the beginning of the peritonitis modeling. For histological study the intestinal tissue was taken. The resulting pieces of the organ were fixed in a 10 % neutral formalin solution, which were then stained with hematoxylin and eosin. Results and Discussion. On the 3rd day of the experiment in animals with a modeled AGP, vascular changes were manifested first of all by the rounding (retraction) of endothelial cells or their desquamation and the appearance of defects, that allow plasma proteins and the formed elements of blood to leave circulation boundaries of the vascular bed. On the 7 th day in animals with a modeled AGP increased vascular permeability of the mucous membrane of the small intestine was accompanied by a significant edema of the stroma of the villi and by focal hemorrhages. Conclusion. Consequently, the distinct inflammatory changes in all terms of the injury were seen in the wall of the small intestine at the simulated AGP. The significant expansion of the capillaries and venules against the background of inflammatory infiltration in the stroma of the glandular component of the small intestine was noted on the 1st day from the beginning of the experiment. An increase in the height of intestinal villus and a crypt with retraction and desquamation of endothelial cells in the wall of vessels, which caused platelet adhesion in the areas of destruction was observed on the 3rd day. The areas of focal necrosis of the superficial epithelium, which were accompanied by multiple hemorrhages per diapedesis in the perivascular space of the mucous membrane, were seen in the wall of the small intestine of animals with a simulated AGP on the 7th day.

scholarly journals Adaptation of the Relationship in the "Parasite–Host" System with Parasitism Paramphistomum сervi in the Small Intestine of Sheeps

2020 ◽  
Vol 14 (1) ◽  
pp. 46-52
Author(s):  
Oksana I. Bibik ◽  
Ivan A. Arkhipov ◽  
Lyubov V. Nacheva ◽  
Miсhail S. Boborykin
Keyword(s):  
Small Intestine ◽  
Mucous Membrane ◽  
Microstructural Analysis ◽  
Epithelial Tissue ◽  
Host System ◽  
Mutual Relations ◽  
The Relationship ◽  
Histological Procedure ◽  
Histological Staining

The purpose of the research is studying microstructural changes in the process of adaptation of components during the formation of the “parasite–host” system by the example of Paramphistomum cervi (Zeder, 1790) parasitizing in the small intestine of spontaneously infected sheep. Materials and methods. Peculiarities of changes in the microstructure of the small intestine in the “parasite–host” system were studied using parasitization of P. cervi in the intestines of sheep using well-known histological methods. Pieces of tissue of the small intestine of sheep with P. cervi, after being preserved in 70% alcohol, were processed according to the generally accepted histological procedure and embedded in paraffin. Sections 5–7 μm in thickness were stained with histological stains and examined under a light microscope. Results and discussion. Microstructural analysis of the characteristics of the relationship in the "parasite–host" system showed that the mucous membrane of the small intestine of sheep in the presence of P. cervi looks sharply thickened. Swelling of the epithelium of villi and crypts, its vacuolization and albuminoid degeneration were found. In some places, proliferation of the epithelial layer of the mucous membrane was detected, due to which epithelial hyperplasia is observed, in some cases turning into metaplasia. Proliferation and hyperplasia (metaplasia) in the endostation of the host in the presence of the parasite contribute to: 1. stability of the parasite-host system and the participation of trematodes in trophism; 2. determination of the clinical and morphological picture of trematodose; 3. predicting the effects of this pathology on the host. Adhesion in the microstructural complex “trematode tegument – epithelial tissue of the villi of the small intestine” in ovine paramphistomosis at the contact level of two glycocalyx layers – the tegument of P. cervi and the surface of the epithelial cells of the villi of the host’s intestine, as well as single histological staining of the sites of contact between the parasite and the host as a result of mixing the components of the trematode tegument and the tissue of the host’s small intestine with deep adhesion indicate the established mutual relations between the components of a single system "parasite–host".

A Histochemical Study of Fat Absorption in the Small Intestine of the Rat

1954 ◽  
Vol s3-95 (30) ◽  
pp. 153-158
Author(s):  
W. HEWITT
Keyword(s):  
Small Intestine ◽  
Epithelial Cells ◽  
Mucous Membrane ◽  
Histochemical Study ◽  
Fat Absorption ◽  
Red Coloration

Experiments are described in which sections from the small intestine of rats, fed on triolein 2¾ hours previously, were examined with Sudan black and the acid haematein test, with and without Sudan red coloration. The histochemical appearances in the epithelial cells of the mucous membrane are described. Lipoid can be observed not only within the epithelial cells and their free border but also as particles between the cells.

Further Observations on the Histochemistry of Fat Absorption in the Small Intestine of the Rat

1956 ◽  
Vol s3-97 (38) ◽  
pp. 199-203
Author(s):  
W. HEWITT
Keyword(s):  
Small Intestine ◽  
Fatty Acid ◽  
Epithelial Cells ◽  
Mucous Membrane ◽  
Direct Relationship ◽  
Fat Absorption ◽  
Previous Description ◽  
Histochemical Tests

Studies have been made of the small intestine of rats previously fed with triolein. A previous description of the location of the lipid and phospholipid has been confirmed by using Sudan black and the acid haematein test. In addition histochemical tests have been applied to detect neutral fat and fatty acid. The lipid in the free border of the epithelial cells of the mucous membrane consisted of fatty acid and probably neutral fat, which would support the belief that when neutral fat is absorbed some of it is unhydrolysed. Additional evidence for this is provided by the fact that neutral fat could also be detected between the lateral parts of the cells. Occasionally fatty acid was also detectable in this location. Variable proportions of neutral fat, fatty acid, and phospholipid were to be found inside the epithelial cells and it appeared possible that there was a direct relationship between the amounts of neutral fat and phospholipid present.

STUDIES OF SMALL INTESTINE DURING DEVELOPMENT

PEDIATRICS ◽  
1964 ◽  
Vol 34 (1) ◽  
pp. 38-50 ◽  
Author(s):  
Philip Sunshine ◽  
Norman Kretchmer
Keyword(s):  
Small Intestine ◽  
Clinical Symptoms ◽  
Congenital Absence ◽  
Acute Period ◽  
Symptoms And Signs ◽  
Intestinal Disaccharidases

Five infants with intolerance to lactose or sucrose and one infant with intolerance to both lactose and sucrose have been studied. These infants were symptomatic with fermentative diarrhea, vomiting, and failure to gain weight. The tolerance of these patients to disaccharides was measured, and the sugar or sugars to which they were intolerant were removed from their diets. The infants began to gain weight as their diarrhea abated. After they were asymptomatic and thriving for a period of time, they were re-examined and were found to respond normally to the ingestion of disaccharides. Disacchariduria was detected in 5 of the 6 infants during the acute period of their disorders, but not at the time of their reexamination. This finding is probably correlated to the amount of disaccharide ingested and the activity of the specific intestinal disaccharidase at the time of the test. The clinical symptoms and signs of these patients with acquired intolerance to disaccharides were identical to those of patients with congenital absence of intestinal disaccharidases.

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