scholarly journals KETERKAITAN EKSTRAK TELUR KEONG MAS DENGAN TINGKAT KETAHANAN SALINITAS BENIH KEDELAI (Glycine max (L.) Merrill)

2019 ◽  
Vol 6 (2) ◽  
pp. 26-37
Author(s):  
Syukri ◽  
Risky Ridha
Keyword(s):  
Reactive Oxygen Species ◽  
Glycine Max ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Glycine Max L

Wilayah pesisir mempunyai potensi cukup besar untuk dikembangkan menjadi lahan pertanian, namun peningkatan muka air laut akan menyebabkan terjadinya salinitas. Salah satu penyebab kerusakan tanaman pada kondisi salinitas adalah terjadinya cekaman oksidatif yang disebabkan oleh terakumulasinya senyawa Reactive Oxygen Species (ROS). Radikal ini dapat menyebabkan degradasi membran sel yang dapat menyebabkan hilangnya energi untuk biosintesis, cadangan makanan di embrio menjadi habis dan menurunnya viabilitas dan vigoritas benih. Penelitian ini bertujuan untuk mengkaji seberapa besar pengaruh dari ekstrak telur keong mas sebagai antioksidan alami dalam meningkatkan vigoritas benih kedelai (Glycine max (L.) Merril) pada kondisi cekaman salinitas. Penelitian disusun menggunakan Rancangan Acak Lengkap (RAL) faktorial dengan tiga ulangan. Sebagai faktor pertama yaitu kosentrasi ekstrak telur keong mas yang terdiri dari : E1 (45 %) dan E2 (0 %). faktor kedua yaitu kosentrasi larutan NaCl yang terdiri dari : N0 (NaCl 0,0 %), N1 (NaCl 0,5 %), N2 (NaCl 1,0 %) dan N3 (NaCl 1,5 %). Hasil penelitian menunjukkan bahwa pemberian ekstrak telur keong mas kosentrasi 45 % hanya dapat meningkatkan daya berkecambah sebesar 51,59 %, kecepatan tumbuh sebesar 24,18 %/etmal dan vigor kecambah sebesar 33,55 % pada kondisi cekaman salinitas hingga 0,5 %, namun pada cekaman salinitas 1,0 % dan 1,5 % benih sudah tidak mampu untuk berkecambah normal. Peningkatan cekaman salinitas     0 % - 1,0 % menyebabkan pengurangan panjang hipokotil dan panjang akar primer benih kedelai. Pemberian ekstrak telur keong mas kosentrasi 45 % menghasilkan hipokotil dan akar primer benih kedelai yang lebih panjang dibandingkan dengan tanpa pemberian ekstrak. Hasil penelitian membuktikan adanya perbaikan viabilitas dan vigoritas benih yang ditunjukkan oleh indikasi fisiologi yaitu perbaikan performansi panjang hipokotil dan akar primer, meningkatkan nilai kecepatan tumbuh dan vigor kecambah benih kedelai dengan perendaman ekstrak telur keong mas pada kondisi salinitas walaupun pada level yang rendah.

Comparative Analysis of Defence Response of Soybean by Seed Soaking in Gibberellic Acid to Salinity

2020 ◽  
Author(s):  
Burcu Seckin Dinler ◽  
Volkan Gul
Keyword(s):  
Reactive Oxygen Species ◽  
Gibberellic Acid ◽  
Enzyme Activities ◽  
Biomass Yield ◽  
Reactive Oxygen ◽  
Antioxidant Enzyme Activities ◽  
Oxygen Species ◽  
Comparative Response ◽  
Glycine Max L ◽  
Soybean Plants

The present study was conducted to determine the effects of gibberellic acid (GA3) presoaking of soybean (Glycine max L.) seeds on the biomass yield, regulation of reactive oxygen species and some antioxidant enzyme activities in radicle, hypocotyl and cotyledons under salinity. Within this scope, the seeds were subjected to priming with 150 ppm GA3 for 6 h at room temperature. Then, 150 mM NaCl was added in a group and the control was not primed with GA3. The results clearly revealed that salt stress caused a slight induction of biomass yield of radicle and hypocotyl, photosynthetic pigments in cotyledon, reactive oxygen species content in all organs. Moreover, GA3 pretreatment alleviated salt-induced oxidative damage by maintaining superoxide dismutase, ascorbate peroxidase and glutathione reductase enzyme activities in soybean plants. This study firstly elucidates the effects of GA3 priming on the comparative response mechanisms to salinity with different organs of soybean plants.

Mitochondria as a source of reactive oxygen species

2009 ◽  
pp. c3 ◽  
Author(s):  
Helena M. Cochemé ◽  
Michael P. Murphy
Keyword(s):  
Reactive Oxygen Species ◽  
Reactive Oxygen ◽  
Oxygen Species

Clinical aspects of reactive oxygen and nitrogen species

2004 ◽  
Vol 71 ◽  
pp. 121-133 ◽  
Author(s):  
Ascan Warnholtz ◽  
Maria Wendt ◽  
Michael August ◽  
Thomas Münzel
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Risk Factor ◽  
Endothelial Dysfunction ◽  
Vascular Tissue ◽  
Rapid Development ◽  
Reactive Oxygen ◽  
Clinical Aspects ◽  
No Production ◽  
Oxygen Species

Endothelial dysfunction in the setting of cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes mellitus and chronic smoking, as well as in the setting of heart failure, has been shown to be at least partly dependent on the production of reactive oxygen species in endothelial and/or smooth muscle cells and the adventitia, and the subsequent decrease in vascular bioavailability of NO. Superoxide-producing enzymes involved in increased oxidative stress within vascular tissue include NAD(P)H-oxidase, xanthine oxidase and endothelial nitric oxide synthase in an uncoupled state. Recent studies indicate that endothelial dysfunction of peripheral and coronary resistance and conductance vessels represents a strong and independent risk factor for future cardiovascular events. Ways to reduce endothelial dysfunction include risk-factor modification and treatment with substances that have been shown to reduce oxidative stress and, simultaneously, to stimulate endothelial NO production, such as inhibitors of angiotensin-converting enzyme or the statins. In contrast, in conditions where increased production of reactive oxygen species, such as superoxide, in vascular tissue is established, treatment with NO, e.g. via administration of nitroglycerin, results in a rapid development of endothelial dysfunction, which may worsen the prognosis in patients with established coronary artery disease.

Fas ligand expression in rat kupffer cells in response to lipopolysaccharide is mediated by reactive oxygen species

2001 ◽  
Vol 120 (5) ◽  
pp. A361-A361
Author(s):  
K UCHIKURA ◽  
T WADA ◽  
Z SUN ◽  
S HOSHINO ◽  
G BULKLEY ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Kupffer Cells ◽  
Fas Ligand ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Ligand Expression
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