scholarly journals Building damage in Westport

Author(s):  
A. L. Andrews

The earthquake of 24 May 1968, was the second of damaging intensity to be experienced in Westport recently. The earlier (1962) quake was of smaller magnitude; but centred, as it was, at Westport it was probably felt with comparable intensity there. Restoration of widespread damage it caused undoubtedly reduced the extent of damage from the 1968 earthquake. Nevertheless the survey after the Inangahua earthquake showed clearly that some of the "remedial work” that followed the 1962 earthquake was simply patching up of surfaces, with no structural significance. Not surprisingly the 1962 wounds so "treated" appeared again in 1968.

Author(s):  
D. I. D. Bird ◽  
S. C. Gentry ◽  
J. P. Hollings

This paper covers building damage occurring in the area between Inangahua Landing (approximately 14 miles north of Reefton,) Reefton itself and down to Totara Flat (approximately 16 miles south-west of Reefton). Reefton is an old mining town on the upper reaches of the Inangahua River some 20 miles above and to the south of Inangahua itself. It is administered as a County Town. The township of Reefton and in fact practically all structures in the area were situated on alluvial river flats, a fact of some significance in the damage observed. Almost without exception every property owner in the area suffered some damage.
The degree of damage was greatest in the Rotokehu area, north of Reefton, but the extent of damage was of course greatest in the Reefton Township. There were in all some 665 claims against the Earthquake and War Damage Commission totalling some $205,000. There were few structures of significance and most buildings in the area were old or very old, but a number of new structures have been built in the last few years. On account of age and arrears of maintenance many buildings in the area were in poor condition before the earthquake, although their age indicates they must have survived previous earthquakes.


Author(s):  
R.F. Dodson ◽  
L.W-F Chu ◽  
N. Ishihara

The extent of damage surrounding an implanted electrode in the cerebral cortex is a question of significant importance with regard to attaining consistency and validity of physiological recordings. In order to determine the extent of such tissue changes, 150 micron diameter platinum electrodes were implanted in the cortex of four adult baboons, and after eight days the animals were sacrificed by whole body perfusion with a 3% glutaraldehyde in 0.1M phosphate fixative.The calvarium was carefully removed and the electrode tracts were readily discernible in the firm, glutaraldehyde fixed tissue.Careful dissection of the zone of the electrode tract resulted in a small block which was further sectioned into tip, mid-tract and surface areas. Ultrastructurally, damage extended from the electrode sheath to the greatest extent of from 0.2 to 3.5 mm.


2013 ◽  
Vol 13 (2) ◽  
Author(s):  
Wisyanto Wisyanto

Tsunami which was generated by the 2004 Aceh eartquake has beenhaunting our life. The building damage due to the tsunami could be seenthroughout Meulaboh Coastal Area. Appearing of the physical loss wasclose to our fault. It was caused by the use dan plan of the land withoutconsidering a tsunami disaster threat. Learning from that event, we haveconducted a research on the pattern of damage that caused by the 2004tsunami. Based on the analysis of tsunami hazard intensity and thepattern of building damage, it has been made a landuse planning whichbased on tsunami mitigation for Meulaboh. Tsunami mitigation-based ofMeulaboh landuse planning was made by intergrating some aspects, suchas tsunami protection using pandanus greenbelt, embankment along withhigh plants and also arranging the direction of roads and setting of building forming a rhombus-shaped. The rhombus-shaped of setting of the road and building would reduce the impact of tsunamic wave. It is expected that these all comprehensive landuse planning will minimize potential losses in the future .


2019 ◽  
Vol 57 (9) ◽  
pp. 733-742
Author(s):  
Y. Maida ◽  
T. Mukai ◽  
H. Miyauchi

2020 ◽  
pp. 304-312

Background: Insult to the brain, whether from trauma or other etiologies, can have a devastating effect on an individual. Symptoms can be many and varied, depending on the location and extent of damage. This presentation can be a challenge to the optometrist charged with treating the sequelae of this event as multiple functional components of the visual system can be affected. Case Report: This paper describes the diagnosis and subsequent ophthalmic management of an acquired brain injury in a 22 year old male on active duty in the US Army. After developing acute neurological symptoms, the patient was diagnosed with a pilocytic astrocytoma of the cerebellum. Emergent neurosurgery to treat the neoplasm resulted in iatrogenic cranial nerve palsies and a hemispheric syndrome. Over the next 18 months, he was managed by a series of providers, including a strabismus surgeon, until presenting to our clinic. Lenses, prism, and in-office and out-of-office neurooptometric rehabilitation therapy were utilized to improve his functioning and make progress towards his goals. Conclusions: Pilocytic astrocytomas are the most common primary brain tumors, and the vast majority are benign with excellent surgical prognosis. Although the most common site is the cerebellum, the visual pathway is also frequently affected. If the eye or visual system is affected, optometrists have the ability to drastically improve quality of life with neuro-optometric rehabilitation.


Author(s):  
Sergey Staroverov ◽  
Sergey Kozlov ◽  
Alexander Fomin ◽  
Konstantib Gabalov ◽  
Alexey Volkov ◽  
...  

Background: The liver disease problem prompts investigators to search for new methods of liver treatment. Introduction: Silymarin (Sil) protects the liver by reducing the concentration of free radicals and the extent of damage to the cell membranes. A particularly interesting method to increase the bioavailability of Sil is to use synthesized gold nanoparticles (AuNPs) as reagents. The study considered whether it was possible to use the silymarin-AuNP conjugate as a potential liver-protecting drug. Method: AuNPs were conjugated to Sil and examine the liver-protecting activity of the conjugate. Experimental hepatitis and hepatocyte cytolysis after carbon tetrachloride actionwere used as a model system, and the experiments were conducted on laboratory animals. Result: For the first time, silymarin was conjugated to colloidal gold nanoparticles (AuNPs). Electron microscopy showed that the resultant preparations were monodisperse and that the mean conjugate diameter was 18–30 nm ± 0.5 nm (mean diameter of the native nanoparticles, 15 ± 0.5 nm). In experimental hepatitis in mice, conjugate administration interfered with glutathione depletion in hepatocytes in response to carbon tetrachloride was conducive to an increase in energy metabolism, and stimulated the monocyte–macrophage function of the liver. The results were confirmed by the high respiratory activity of the hepatocytes in cell culture. Conclusion: We conclude that the silymarin-AuNP conjugate holds promise as a liver-protecting agent in acute liver disease caused by carbon tetrachloride poisoning.


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