infectious disease model
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2022 ◽  
Vol 2022 ◽  
pp. 1-12
Author(s):  
Zulqurnain Sabir ◽  
Muhammad Asif Zahoor Raja ◽  
Yolanda Guerrero Sánchez

The aim of the current work is to perform the numerical investigation of the infectious disease based on the nonlinear fractional order prey-predator model using the Levenberg–Marquardt backpropagation (LMB) based on the artificial neuron networks (ANNs), i.e., LMBNNs. The fractional prey-predator model is classified into three categories, the densities of the susceptible, infected prey, and predator populations. The statistics proportions for solving three different variations of the infectious disease based on the fractional prey-predator model are designated for training 80% and 10% for both validation and testing. The numerical actions are performed using the LMBNNs to solve the infectious disease based on the fractional prey-predator model, and comparison is performed using the database Adams–Bashforth–Moulton approach. The infectious disease based on the fractional prey-predator model is solved using the LMBNNs to reduce the mean square error (M.S.E). In order to validate the exactness, capability, consistency, and competence of the proposed LMBNNs, the numerical procedures using the correlation, M.S.E, regression, and error histograms are drawn.


Author(s):  
Auwal Abdullahi

In this paper, the dynamics of Hepatitis C infectious disease model with two treatment effects are studied through the Ito Stochastic Differential Equations (SDEs). While the first treatment rate reduces the reproduction of virion, the other mitigates the new infections. Though the deterministic behaviour of the model has been extensively studied, little is known about its stochastic properties. Thus, we examine sufficient conditions for the existence and uniqueness of the ergodic stationary distribution of the model via stochastic Lyapunov approach. The existence of a unique positive solution is also studied. The numerical simulations of the SDE model are performed through the Euler-Maruyama method and compared with their deterministic counterparts. The results obtained by SDEs are found to conform to those reported through their deterministic analogues.


2021 ◽  
Vol 11 (9) ◽  
pp. 534-537
Author(s):  
Daria Żuraw ◽  
Paulina Oleksa ◽  
Mateusz Sobczyk

Introduction: Obesity has been recognized as a global epidemic by the WHO, followed by a wealth of empirical evidence supporting its contagiousness. However, the dynamics of the spread of obesity between individuals are rarely studied.  A distinguishing feature of the obesity epidemic is that it is driven by a process of social contagion that cannot be perfectly described by the infectious disease model. There is also social discrimination in the obesity epidemic. Social discrimination against obese people plays quite different roles in two cases: on the one hand, when obesity cannot be eliminated, social discrimination can reduce the number of obese people; on the other hand, when obesity is eradicable, social discrimination can cause it to explode.(1)   Materiał and methods: A literature analysis on obesity epidemic was carried out within the Pubmed, Google scholar and Research Gate platform. The following keywords were used in serach: obesity, epidemy, children, body max index.    Purpose of the work: The aim of the following analysis is to present an obesity as an infectious disease. The steadily increasing percentage of obese people, including children, shows that there is an obesity epidemic. This is the phenomenon of social contagion, which partially explains the concept of homophily, which involves the grouping of people with similar characteristics. Potential explanations are also provided by sharing a living environment with similar access to certain foods and similar opportunities for physical activity, which defines the occurrence of analogous health habits


2021 ◽  
Vol 2021 ◽  
pp. 1-11
Author(s):  
Hashim M. Alshehri ◽  
Hasib Khan ◽  
Zareen A. Khan

In the present paper, we study a mathematical model of an imperfect testing infectious disease model in the sense of the Mittage-Leffler kernel. The Banach contraction principle has been used for the existence and uniqueness of solutions of the suggested model. Furthermore, a numerical method equipped with Lagrangian polynomial interpolation has been utilized for the numerical outcomes. Diagramming and discussion are used to clarify the effects of related parameters in the fractional-order imperfect testing infectious disease model.


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