The Encephalopathy Associated with Septic Illness: Clinical Neurological Features

1997 ◽  
pp. 94-106
Author(s):  
G. B. Young
Keyword(s):  
Neurological Features

NEUROLOGICAL FEATURES IN CHILDREN AND DIPHENYLARSINIC ACID PROBABLY DERIVED FROM CHEMICAL WARFARE AGENTS

2006 ◽  
Vol 37 (S 1) ◽  
Author(s):  
N Iwasaki ◽  
S Miyamoto ◽  
K Ishii ◽  
T Takeda ◽  
T Ohto ◽  
...  
Keyword(s):  
Chemical Warfare Agents ◽  
Chemical Warfare ◽  
Warfare Agents ◽  
Neurological Features

CLINICAL AND ELECTROMYOGRAPHIC PREDICTION OF FACIAL NERVE NEUROPATHY IN CHILDREN

2020 ◽  
Vol 3 (1) ◽  
pp. 38-40
Author(s):  
Nozima Asadova ◽  
◽  
Aziza Djurabekova ◽  
Saodat Igamova
Keyword(s):  
Facial Nerve ◽  
Children And Adolescents ◽  
Medical Literature ◽  
Latency Period ◽  
Pain Syndrome ◽  
Literature Discussions ◽  
Clinical Forms ◽  
Diagnosis And Prognosis ◽  
Neurological Features ◽  
Blinking Reflex

In modern medical literature, discussions continue about neuropathies of the facial nerve in children,disease factors, clinical and neurological features, the severity of the course, the difficulties of diagnosis and prognosis of the disease. The work is devoted to the analysis of the neuropathy of the facial nerve in children and adolescents, depending on the clinical forms of the disease, using electroneurosympathetic indicators, the latency period, the blinking reflex and thenatureof the lesion of the facial nerve accompanied by pain syndrome

CLINICAL AND NEUROLOGICAL FEATURES OF TRAUMATIC BRAIN DISEASE DURING THE STAGES OF REHABILITATION

2020 ◽  
Vol 3 (1) ◽  
pp. 51-53
Author(s):  
Rano Azizova ◽  
◽  
Umida Shamsiyeva ◽  
Mirzohid Turabbayev ◽  
Begzod Jorayev ◽  
...  
Keyword(s):  
Mechanical Energy ◽  
Pathological Process ◽  
Brain Disease ◽  
Clinical Forms ◽  
Neurological Features ◽  
Mechanisms Of Development ◽  
The Brain

Traumatic brain disease (TBHD) is a pathological process triggered by the damaging effect of mechanical energy on the brain and is characterized — with a variety of clinical forms — by the unity of etiology, pathogenetic and sanogenetic mechanisms of development and outcomes.

Venglustat combined with imiglucerase positively affects neurological features and brain connectivity in adults with Gaucher disease type 3

2021 ◽  
Vol 132 (2) ◽  
pp. S95
Author(s):  
Raphael Schiffmann ◽  
Timothy Cox ◽  
Jean-François Dedieu ◽  
Sebastiaan Gaemers ◽  
Julia B. Hennermann ◽  
...  
Keyword(s):  
Gaucher Disease ◽  
Brain Connectivity ◽  
Disease Type ◽  
Neurological Features ◽  
Type 3

scholarly journals Gasperini Syndrome: A Case Report and Systematic Review and Proposing a New Definition

2020 ◽  
Vol 12 (3) ◽  
pp. 433-439
Author(s):  
Riwaj Bhagat ◽  
Siddharth Narayanan ◽  
Marwa Elnazeir ◽  
Thong Diep Pham ◽  
Robert Paul Friedland ◽  
...  
Keyword(s):  
Systematic Review ◽  
Brain Mri ◽  
Cranial Nerves ◽  
Clinical Manifestations ◽  
The Other ◽  
Neurological Deficits ◽  
Brainstem Stroke ◽  
Neurological Features ◽  
The Right ◽  
Core Features

Gasperini syndrome (GS), a rare brainstem syndrome, is featured by ipsilateral cranial nerves (CN) V–VIII dysfunction with contralateral hemibody hypoesthesia. While there have been 18 reported cases, the GS definition remains ambiguous. We report a new case and reviewed the clinical features of this syndrome from all published reports to propose a new definition. A 57-year-old man with acute brainstem stroke had right CN V–VIII and XII palsies, left body hypoesthesia and ataxia. Brain MRI showed an acute stroke in the right caudal pons and bilateral cerebellum. After a systematic review, we classified the clinical manifestations into core and associate features based on the frequencies of occurring neurological deficits. We propose that a definitive GS requires the presence of ipsilateral CN VI and VII palsies, plus one or more of the other three core features (ipsilateral CN V, VIII palsies and contralateral hemibody hemihypalgesia). Additionally, GS, similar to Wallenberg’s syndrome, represents a spectrum that can have other associated neurological features. The revised definition presented in this study may enlighten physicians with the immediate recognition of the syndrome and help improve clinical localization of the lesions and its management.

Cerebrospinal fluid γ-aminobutyric acid levels in dogs with chronic portosystemic encephalopathy

1986 ◽  
Vol 71 (6) ◽  
pp. 749-753 ◽  
Author(s):  
J. E. Maddison ◽  
D. Yau ◽  
P. Stewart ◽  
G. C. Farrell
Keyword(s):  
Cerebrospinal Fluid ◽  
Aminobutyric Acid ◽  
Plasma Ammonia ◽  
Portosystemic Encephalopathy ◽  
Dog Model ◽  
Csf Levels ◽  
Neurological Features ◽  
The Brain ◽  
Biochemical Abnormalities

1. Cerebrospinal fluid (CSF) γ-aminobutyric acid (GABA) levels were measured in a dog model of spontaneous chronic portosystemic encephalopathy. 2. Dogs with congenital portacaval shunts (intra- or extra-hepatic) develop neurological features of abnormal psychomotor behaviour and depressed consciousness that are consistent with the symptoms of chronic portosystemic encephalopathy in humans. In the five dogs studied, plasma ammonia was elevated, as was CSF tryptophan, both usual biochemical abnormalities in portosystemic encephalopathy. 3. CSF levels of GABA in five dogs with portosystemic encephalopathy (100 ± 13 pmol/ml) were not significantly different from those in five control dogs (96 ± 14 pmol/ml). CSF levels of GABA were not altered after ammonia infusion. 4. If enhanced GABA-ergic neurotransmission, due to influx of gut-derived GABA into the brain, is responsible for the pathophysiology of chronic portosystemic encephalopathy in this model, it is not reflected by increased levels of GABA in CSF.

scholarly journals Idiopathic cerebellar ataxia associated with celiac disease: lack of distinctive neurological features

1999 ◽  
Vol 66 (1) ◽  
pp. 32-35 ◽  
Author(s):  
M. T. Pellecchia ◽  
R. Scala ◽  
A. Filla ◽  
G. De Michele ◽  
C. Ciacci ◽  
...  
Keyword(s):  
Celiac Disease ◽  
Cerebellar Ataxia ◽  
Neurological Features ◽  
Idiopathic Cerebellar Ataxia

scholarly journals ATP1A3-Related Disorders: An Ever-Expanding Clinical Spectrum

2021 ◽  
Vol 12 ◽  
Author(s):  
Philippe A. Salles ◽  
Ignacio F. Mata ◽  
Tobias Brünger ◽  
Dennis Lal ◽  
Hubert H. Fernandez
Keyword(s):  
Original Description ◽  
Rapid Onset ◽  
Acute Onset ◽  
Clinical Spectrum ◽  
Pes Cavus ◽  
Sodium Potassium ◽  
Pathogenic Variants ◽  
Alternating Hemiplegia Of Childhood ◽  
The Central Nervous System ◽  
Neurological Features

The Na+/K+ ATPases are Sodium-Potassium exchanging pumps, with a heteromeric α-β-γ protein complex. The α3 isoform is required as a rescue pump, after repeated action potentials, with a distribution predominantly in neurons of the central nervous system. This isoform is encoded by the ATP1A3 gene. Pathogenic variants in this gene have been implicated in several phenotypes in the last decades. Carriers of pathogenic variants in this gene manifest neurological and non-neurological features in many combinations, usually with an acute onset and paroxysmal episodes triggered by fever or other factors. The first three syndromes described were: (1) rapid-onset dystonia parkinsonism; (2) alternating hemiplegia of childhood; and, (3) cerebellar ataxia, pes cavus, optic atrophy, and sensorineural hearing loss (CAPOS syndrome). Since their original description, an expanding number of cases presenting with atypical and overlapping features have been reported. Because of this, ATP1A3-disorders are now beginning to be viewed as a phenotypic continuum representing discrete expressions along a broadly heterogeneous clinical spectrum.

scholarly journals Pasmodium Vivax Malaria Complicated by Neurological, Jaundice and Haemophagocytic Syndrome

1970 ◽  
Vol 30 (3) ◽  
pp. 168-170 ◽  
Author(s):  
TV Ram Kumar ◽  
S Baghmar
Keyword(s):  
Plasmodium Vivax ◽  
Vivax Malaria ◽  
Haemophagocytic Syndrome ◽  
Plasmodium Vivax Malaria ◽  
Neurological Features

We are describing a case of plasmodium vivax malaria in a child with complications including pancytopenia secondary to marrow involvement, jaundice and neurological features. The child improved with antimalarial treatment.DOI: 10.3126/jnps.v30i3.3922J Nep Paedtr Soc 2010;30(3):168-170

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