T-wave inversion: cardiac memory or myocardial ischemia?

2009 ◽  
Vol 27 (7) ◽  
pp. 898.e1-898.e4 ◽  
Author(s):  
Carol Chen-Scarabelli ◽  
Tiziano M. Scarabelli
Kardiologiia ◽  
2020 ◽  
Vol 60 (5) ◽  
pp. 136-145
Author(s):  
I. R. Tsoy ◽  
I. P. Kolos

The T-wave inversion (TWI) is a common electrocardiographic finding. Causes for TWI are numerous and sometimes TWI may appear in life-threatening conditions. The aim of this review was to provide an up-to-date analysis of TWI, including i) definition and prevalence; ii) causes, and iii) differential diagnosis in benign TWI, reversible myocardial ischemia of the left ventricular anterior wall; takotsubo cardiomyopathy; subarachnoid hemorrhage; pulmonary embolism; right ventricular arrhythmogenic cardiomyopathy; and “cardiac memory”. The review presents practical electrophysiological criteria, which allow suspecting in time a life-threatening condition to choose an up-to-date treatment.


2020 ◽  
Author(s):  
Sitaram Mittal

T- wave inversion in electrocardiogram immediately attracts attention because myocardial ischemia is a common and serious cause. There are, however, several other, relatively benign, cause that can also cause inversion of T wave. We observed that medial placement of lead V3 electrode can result in false shallow T wave inversion in lead V3. Upright T wave in lead V2 and V4 should give a clue to this technical error. To the best of our knowledge, this cause of T wave inversion localized to lead V3 only with positive T wave in leads V2and V4has not been reported in the past.


2008 ◽  
Vol 3 (4) ◽  
pp. 375-378 ◽  
Author(s):  
Johanna Rottensteiner ◽  
Angelika Kaneppele ◽  
Ingrid Stockner ◽  
Carmen Ladurner ◽  
Georgio Panizza ◽  
...  

2011 ◽  
Vol 2011 ◽  
pp. 1-3
Author(s):  
David Rott ◽  
David Leibowitz ◽  
A. Teddy Weiss

Giant precordial T wave inversion (GPTI) on ECG may be the result of several pathologies, including myocardial ischemia, pulmonary edema, pulmonary embolism, subarachnoid hemorrhage, apical hypertrophy, and postpacing. We describe a case of a 75-year-old woman who developed GPTI after an episode of gastroenteritis. To our knowledge, this is the first report of this ECG pattern associated with gastroenteritis.


Circulation ◽  
2008 ◽  
Vol 118 (suppl_18) ◽  
Author(s):  
Shafeeq Ahmed ◽  
Saneka Chakravarty ◽  
Craig I Coleman ◽  
Jeffrey Kluger

Post pacing induced T-wave inversion after resumption of normal ventricular activation is known as cardiac memory. Animal studies have shown that the transient outward current Ikto and L-type calcium channels are altered in cardiac memory. We hypothesize that pacing induced cardiac memory is modified by amiodarone due to its I kto and L-type calcium channel blocking effects. We conducted a prospective cohort evaluation of paced patients at a single institution. Patients were excluded if they were pacemaker dependant or had underlying bundle branch block. All eligible patients had a pacemaker-inhibited standard 12-lead electrocardiogram after informed consent and collection of basic demographic information. Patients who were receiving amiodarone (n=11) at the time of pacemaker interrogation were compared to a control group of patients that were not taking amiodarone (n=28). Cardiac memory was deemed to be present if T-wave inversions were observed in any pre-cordial or inferior lead (V1-V6, II, III, and AVF). Univariate statistics were used to compare both the incidence of cardiac memory and the mean number of cardiac memory leads. Seven patients receiving and 27 patients not receiving amiodarone exhibited cardiac memory (63.6% vs. 96.4%; p=0.02†). (See Table ) In addition, patients receiving amiodarone had cardiac memory observed in significantly fewer pre-cordial or inferior leads (2.91 vs. 5.18; p=0.02†) Amiodarone appears to be associated with attenuation of pacing induced cardiac memory. Table


Sign in / Sign up

Export Citation Format

Share Document