Content of conjugated linoleic acid in neutral and polar lipid fractions of milk of different ruminant species

2009 ◽  
Vol 19 (5) ◽  
pp. 342-344 ◽  
Author(s):  
Giovanna Contarini ◽  
Valeria Pelizzola ◽  
Milena Povolo
2001 ◽  
Vol 42 (7) ◽  
pp. 1056-1061 ◽  
Author(s):  
Sebastiano Banni ◽  
Gianfranca Carta ◽  
Elisabetta Angioni ◽  
Elisabettta Murru ◽  
Paolo Scanu ◽  
...  

2007 ◽  
Vol 2007 ◽  
pp. 15-15
Author(s):  
G. A. Romero-Perez ◽  
R. W. Mayes ◽  
J. R. Scaife ◽  
J .A. Milne

Proportions of polyunsaturated fatty acid (PUFA) in ruminant tissues are generally low as dietary PUFA including linoleic acid (LA) and α-linolenic acid (ALA) undergo biohydrogenation to stearic acid in the rumen. Conjugated linoleic acid (CLA) is formed during biohydrogenation as an intermediate metabolite. Feeding PUFA-rich sources can enhance deposition in the tissues of ruminants (Bolte et al, 2002), but the degree of lipid hydrogenation may depend on the source form and the ruminant species (Rowell-Schäffer et al, 2001). Bolte et al, (2002) showed that oilseed-fed sheep had higher PUFA proportions in their tissues than sheep fed a beet pulp-based diet but Rowell-Schäffer et al (2001) found in deer tissues higher PUFA proportions than in those of sheep. Possible effects of the form of the lipid source and the ruminant species on the fatty acids deposited were tested.


2012 ◽  
Vol 142 (9) ◽  
pp. 1645-1651 ◽  
Author(s):  
Kristin L. Ritzenthaler ◽  
Alam M. Shahin ◽  
Terry D. Shultz ◽  
Nairanjana Dasgupta ◽  
Mark A. McGuire ◽  
...  

2010 ◽  
Vol 80 (1) ◽  
pp. 65-73 ◽  
Author(s):  
Pei-Min Chao ◽  
Wan-Hsuan Chen ◽  
Chun-Huei Liao ◽  
Huey-Mei Shaw

Conjugated linoleic acid (CLA) is a collective term for the positional and geometric isomers of a conjugated diene of linoleic acid (C18:2, n-6). The aims of the present study were to evaluate whether levels of hepatic α-tocopherol, α-tocopherol transfer protein (α-TTP), and antioxidant enzymes in mice were affected by a CLA-supplemented diet. C57BL/6 J mice were divided into the CLA and control groups, which were fed, respectively, a 5 % fat diet with or without 1 g/100 g of CLA (1:1 mixture of cis-9, trans-11 and trans-10, cis-12) for four weeks. α-Tocopherol levels in plasma and liver were significantly higher in the CLA group than in the control group. Liver α-TTP levels were also significantly increased in the CLA group, the α-TTP/β-actin ratio being 2.5-fold higher than that in control mice (p<0.01). Thiobarbituric acid-reactive substances were significantly decreased in the CLA group (p<0.01). There were no significant differences between the two groups in levels of three antioxidant enzymes (superoxide dismutase, glutathione peroxidase, and catalase). The accumulation of liver α-tocopherol seen with the CLA diet can be attributed to the antioxidant potential of CLA and the ability of α-TTP induction. The lack of changes in antioxidant enzyme protein levels and the reduced lipid peroxidation in the liver of CLA mice are due to α-tocopherol accumulation.


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