scholarly journals Adiposity rebound may be a predictive index of body size for adolescents—Based on retrospective cohort data in a Japanese rural area

Author(s):  
Chiyori Haga ◽  
Hiroshi Yokomichi ◽  
Kyoko Tsuji ◽  
Zentaro Yamagata
2016 ◽  
Vol 8 (1) ◽  
pp. 113-122 ◽  
Author(s):  
R. T. Sabo ◽  
A. Wang ◽  
Y. Deng ◽  
C. S. Sabo ◽  
S. S. Sun

Previous research has shown that childhood body size is associated with blood pressure in adulthood, and that early and rapid growth rates are correlated with adverse cardiovascular outcomes. Our objectives are to estimate associations between childhood body size growth parameters and adult blood pressure, and to examine the effect of early attainment of critical growth milestones on adult blood pressure, relative to normal or late attainment. Lifetime height and body mass index (BMI) measurements in childhood, and systolic blood pressure (SBP) and diastolic blood pressure (DBP) measurements in adulthood are taken from participants in the Fels Longitudinal Study. Childhood growth curves are estimated separately for stature and BMI using the Preece–Baines and third-degree polynomial models, respectively. Associations between the resulting parameter estimates and adult blood pressure are then examined using linear mixed models. Our findings show that the ages of achievement of the stature-based growth onset and peak velocity, as well as the age of achievement of the BMI-based adiposity rebound, are negatively associated with adult blood pressure, implying that early height or BMI growth can lead to increased blood pressure in adulthood. There were subtle differences in these relationships based on age and gender, and also between SBP and DBP. These results expand on the existing literature, showing that not only childhood body size, but also the timing of childhood growth can have a deleterious effect on adult cardiovascular health.


Resuscitation ◽  
2018 ◽  
Vol 130 ◽  
pp. 67-72 ◽  
Author(s):  
Chih-Hung Wang ◽  
Chien-Hua Huang ◽  
Wei-Tien Chang ◽  
Chia-Ming Fu ◽  
Hui-Chih Wang ◽  
...  

2015 ◽  
Vol 15 (1) ◽  
Author(s):  
Kate Hoffman ◽  
Ann Aschengrau ◽  
Thomas F. Webster ◽  
Scott M. Bartell ◽  
Verónica M. Vieira

PLoS ONE ◽  
2017 ◽  
Vol 12 (4) ◽  
pp. e0175837 ◽  
Author(s):  
Fiona G. Kouyoumdjian ◽  
Evgeny M. Andreev ◽  
Rohan Borschmann ◽  
Stuart A. Kinner ◽  
Andres McConnon

The Lancet ◽  
2016 ◽  
Vol 388 ◽  
pp. S53 ◽  
Author(s):  
Xuhua Tan ◽  
Haotian Lin ◽  
YuTing Li ◽  
Li Huang ◽  
Yi Zhu ◽  
...  

PLoS ONE ◽  
2018 ◽  
Vol 13 (12) ◽  
pp. e0208323 ◽  
Author(s):  
Kylie-Ann Mallitt ◽  
David P. Wilson ◽  
James Jansson ◽  
Ann McDonald ◽  
Handan Wand ◽  
...  

2000 ◽  
Vol 59 (2) ◽  
pp. 267-272 ◽  
Author(s):  
Chris Power ◽  
Tessa Parsons

It has been proposed that there are critical periods during childhood that influence the development of obesity, including gestation and early infancy, the period of adiposity rebound that occurs between ages 5 and 7 years, and adolescence. Despite an extensive literature, there is to date only modest evidence for most of the factors such as nutrition, physical activity and other behavioural factors that are suspected as playing a role in the development of obesity. A recent review of this evidence (Parsons et al. 1999) showed, however, a consistent relationship between socio-economic status (SES) of origin and adult obesity, whereby those from lower SES backgrounds were fatter subsequently in adulthood. This association appeared to apply to both men and women, a finding that contrasts with the trends observed in cross-sectional studies, of an association with SES for women only. There are several potential explanations for the SES of origin–adult obesity relationship. SES of origin may be confounded by parental body size; studies to date provide insufficient evidence of an independent association with SES after allowing for parental body size. Alternatively, environment in early life (for which SES of origin is a proxy measure) may have a long-term impact on obesity later in adulthood, through one or more of several processes. Three major potential explanations can be identified: (1) nutrition in infancy and childhood, either over- or undernutrition, followed subsequently by overnutrition; (2) psychological factors, possibly involving emotional deprivation in childhood; (3) cultural or social norms regarding dietary restraint and attitudes to fatness that may be acquired during childhood.


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