Vital Force and homœopathy

1948 ◽  
Vol 38 (01) ◽  
pp. 32-45
Author(s):  
C.F. Hacker

Summary1. A connection is found between Hahnemann's conception of Vital Force and that of Cannon's “homœostasis”, which is maintained by the autonomic nervous system.2. Vital Force is traced to source in the embryo as it builds its body. It is seen laying down the “dynamic determination” on the multiplying tissue cells, and operating from the central mesodermal column, the notochord, to induct the Individuation Field of the elongating gastrula and differentiating neurula.3. A vital link between the notochord and the diencephalic centres isemphasized.4. The period of functional development sees the embryonic strugglebetween the autonomic system and the gene award. Such conflict is causative of defective autonomic balance, and the results have been noted by Hahnemann as the miasma, the psora, the sycoses.5. The thalamus, as the seat of sensory consciousness receiving both visceral and somatic sensory impulses transmits the sensory tone of the underlying autonomic fluster to the cerebral cortex for cognition. Such cognition is reported as symptoms, “I. …, My …”, thus giving the homœopathic picture of the “abnormally disturbed Vital Force”.6. The factors required in the blood's biochemical changes to maintain constant its pH level are given. The varying balance of the NaHCO3/H2CO3 content and Ca/K ratio may be related to Schuessler's “tissue remedies”.7. Such acid-base equilibrium is dependent on the autonomic functioning, which if impaired in the embryonic conflict is causative of allergy.8. Investigation of allergic factors produces an interesting formula ofhomœopathic significance, that shows the selected homœopathic remedy to be the catalyst key which declutches the autonomic imbalance.9. The varying bioelectric potential across the autonomic nerve gangliabetween diencephalic centres and fluid matrix brings about their functional activity and depression.10. A plea is made for a biochemical test of autonomic functioning.The autonomic nervous system is the meeting-ground of the homœopathicand allopathic divisions of medicine.If the disease process is acute or prolonged without permitting adequate intervals of functional rest to enable regrowth of its essential chromatin store, neurone degeneration follows. Such changes are not related to any specific bacterial action, but result from continuous functioning to exhaustion.The initial build-up of the autonomic path in reaction-response to externalstimuli is the essential basis of the individual's “modalities”.

1993 ◽  
Vol 14 (12) ◽  
pp. 489-492
Author(s):  
Jeffrey S. Rubenstein

The last 20 years have seen an explosion in our knowledge of the autonomic nervous system and our ability to manipulate its parasympathetic and sympathetic portions pharmacologically to achieve therapeutic goals. This article will briefly review the structure and function of the autonomic nervous system, with particular focus on the sympathetic branch. Included in the review is a discussion of the major receptors of the sympathetic system, concentrating on their intracellular mechanism of action, their effects on major target organ systems, and some commonly used pharmacologic agents that influence these organ systems through their actions on sympathetic receptors. Structure and Function of the System The autonomic (or involuntary) nervous system innervates the heart, visceral organs, blood vessels, smooth muscles, and glands. It can be divided functionally into the parasympathetic and sympathetic systems, which have opposing functions. All autonomic nerve pathways consist of two nerves in sequence. Presynaptic nerves begin in the central nervous system and transmit impulses to the postsynaptic nerves. Postsynaptic nerves then carry impulses to the effector organ. Actions of the parasympathetic nervous system include bradycardia, vasodilation in skeletal muscle and skin, contraction of bronchial smooth muscle, increased gastrointestinal motility, pupillary miosis, and contraction of the bladder detrusor coupled with relaxation of the bladder trigone (necessary for spontaneous voiding).


1997 ◽  
Vol 7 (2) ◽  
pp. 119-126 ◽  
Author(s):  
Kenneth J Collins

Autonomic disorders in old age can be attributed to several main features associated with aging: the intrinsic neurobiological changes that occur with age, degenerative changes in effector organs innervated by autonomic nerves, and secondary involvement of the autonomic nervous system (ANS) in disease processes. As in most areas of clinical geriatrics, the distinction between disorders ascribed to ‘normal’ aging and those attributable to diseases of old age is difficult to make with any degree of certainty. Neurobiological changes with age have become the subject of intense investigation in recent years, with improvements in techniques for assessing autonomic nerve structure and function. This has included a better understanding of neurotransmitter and receptor transformations during development and aging. The versatility of the ANS, or ‘plasticity’, involves interactions with target organs, e.g. via nerve growth factor (NGF) and with other neurons, and it is as vital to the mature and aging autonomic neuron as it is during development. Some neurotrophic features of aging in the ANS and in disease processes in old age are considered in this paper.


Author(s):  
Tolga Aksu ◽  
Dhiraj Gupta

Demonstration that the myocardial sleeves of the pulmonary veins (PVs) are the main triggering and maintaining foci for paroxysmal atrial fibrillation (AF) have stimulated studies investigating electrophysiological properties of PVs and the adjacent left atrial (LA) myocardium. It has been shown that PV myocytes have a shorter action potential duration and are more prone to effects of local autonomic nerve stimulation in terms of shortening of action potential duration, early after depolarization formation and triggered firing compared to left atrial myocytes (1). The intrinsic cardiac autonomic nervous system (ICANS) forms clusters of neurons called ganglionic plexi (GPs), and studies using histologic examination of heart sections have shown that these GPs are localized preferentially at certain epicardial sites adjacent to the left and right atria (2). The precise role of ICANS in AF continues to be an area of intense research (3), and matters are not helped by the uncertainty regarding the best way to identify and target ICANS peri-procedurally. As there can be significant variability of GP sites in individual patients, endocardial high-frequency stimulation (HFS) has been used to aid their localization in the electrophysiology laboratory (4).


Author(s):  
Yu.D. Udalov ◽  
L.A. Belova ◽  
V.V. Mashin ◽  
L.A. Danilova ◽  
A.A. Kuvayskaya

When studying oncology diseases, it is necessary not only to assess their nature, progress and outcome depending on the local disease process, but also to take into account overall health, since multiple organ damage has a prognostic value and determines the disease outcome. All mechanisms and structures that have an integrative and homeostatic effect on the overall health should be considered. One of these structures is the autonomic nervous system. Traditionally, the autonomic nervous system has been considered in terms of 3 components: sympathetic, parasympathetic and intestinal. However, in recent years, ideas about neuroendocrine and neuroimmune systems have come to the fore, justifying the expansion of the concept "autonomy of the nervous system." In case of autonomous dysfunction, dysregulation of the involuntary body functions occurs, and autonomic neuropathy develops. There are cardiovascular, gastrointestinal, urogenital, and sudomotor forms of autonomic neuropathy, which are characterized by certain changes in various pathological states, especially in malignant processes. We pay attention to breast cancer, which ranks first in the structure of oncology diseases in the Russian Federation. Nowadays, there are only a few studies devoted to the changes in the autonomic nervous system in patients with breast cancer during anticancer drug therapy, taking into account the disease progress and clinical features, as well as methods for dysfunction remodeling. The article analyzes a number of scientific information sources that can help to study various forms of autonomic neuropathy in patients with breast cancer and allow assessing the use of medical rehabilitation for such patients. Key words: autonomic nervous system, autonomic neuropathy, breast cancer, antitumor drug therapy. При изучении онкологического заболевания необходимо не только оценивать его характер, течение и исход в зависимости от локального процесса, но и учитывать общее состояние организма, так как множественное поражение органов имеет прогностическое значение и определяет исход заболевания. Следует рассматривать все механизмы и структуры, которые оказывают интегративное и гомеостатическое действие на организм в целом. Одной из таких структур является вегетативная нервная система. Традиционно вегетативная нервная система рассматривалась в аспекте 3 составляющих: симпатической, парасимпатической и кишечной. Однако в последнее время на первый план вышли представления о нейроэндокринных и нейроиммунных системах, обосновывающие расширение значения понятия «автономность нервной системы». При поражении автономной нервной системы возникает дисрегуляция непроизвольных функций организма, развивается автономная нейропатия. Выделяют кардиоваскулярную, гастроинтестинальную, урогенитальную, судомоторную формы автономной нейропатии, которые характеризуются определенными изменениями при различных патологических состояниях организма, особенно при злокачественных процессах. Фокус нашего научного внимания был направлен на рак молочной железы, который занимает первое место в структуре онкологических заболеваний в Российской Федерации. В литературе представлены немногочисленные исследования, отражающие изменения состояния вегетативной нервной системы у больных раком молочной железы в процессе противоопухолевой лекарственной терапии с учётом характера течения и клинических особенностей болезни, а также методы рациональной коррекции её дисфункции. В статье приведен анализ ряда научных источников, которые могут помочь в исследовании различных форм автономной нейропатии у больных раком молочной железы и позволят оценить возможность применения медицинской реабилитации для данной категории пациентов. Ключевые слова: вегетативная (автономная) нервная система, автономная нейропатия, рак молочной железы, противоопухолевая лекарственная терапия.


2021 ◽  
Vol 12 ◽  
Author(s):  
Hongyi Duan ◽  
Xueqin Cai ◽  
Yingying Luan ◽  
Shuo Yang ◽  
Juan Yang ◽  
...  

Intestine is composed of various types of cells including absorptive epithelial cells, goblet cells, endocrine cells, Paneth cells, immunological cells, and so on, which play digestion, absorption, neuroendocrine, immunological function. Intestine is innervated with extrinsic autonomic nerves and intrinsic enteric nerves. The neurotransmitters and counterpart receptors are widely distributed in the different intestinal cells. Intestinal autonomic nerve system includes sympathetic and parasympathetic nervous systems, which regulate cellular proliferation and function in intestine under physiological and pathophysiological conditions. Presently, distribution and functional characteristics of autonomic nervous system in intestine were reviewed. How autonomic nervous system regulates intestinal cell proliferation was discussed. Function of autonomic nervous system on intestinal diseases was extensively reviewed. It might be helpful to properly manipulate autonomic nervous system during treating different intestinal diseases.


Sign in / Sign up

Export Citation Format

Share Document