scholarly journals Corticotropin releasing factor-overexpressing mouse is a model of chronic stress-induced muscle atrophy

PLoS ONE ◽  
2020 ◽  
Vol 15 (2) ◽  
pp. e0229048 ◽  
Author(s):  
Wesuk Kang ◽  
Tao Tong ◽  
Taesun Park
1998 ◽  
Vol 60 (1) ◽  
pp. 209-215 ◽  
Author(s):  
Herbert E. Ward ◽  
Elizabeth A. Johnson ◽  
Irving J. Goodman ◽  
Dale L. Birkle ◽  
Dominic J. Cottrell ◽  
...  

2014 ◽  
Vol 46 ◽  
pp. 350
Author(s):  
Travis Parkulo ◽  
Dale Riggs ◽  
Barbara Jackson ◽  
Stanley Zaslau ◽  
Evan DeVallance ◽  
...  

2008 ◽  
Vol 28 (9) ◽  
pp. 1836-1848 ◽  
Author(s):  
Carmen Sandi ◽  
M. Isabel Cordero ◽  
Annarosa Ugolini ◽  
Emilio Varea ◽  
Laura Caberlotto ◽  
...  

2011 ◽  
Vol 2011 ◽  
pp. 1-11 ◽  
Author(s):  
Yolanda Diz-Chaves

Obesity has become one of the leading causes of illness and mortality in the developed world. Preclinical and clinical data provide compelling evidence for ghrelin as a relevant regulator of appetite, food intake, and energy homeostasis. In addition, ghrelin has recently emerged as one of the major contributing factors to reward-driven feeding that can override the state of satiation. The corticotropin-releasing-factor system is also directly implicated in the regulation of energy balance and may participate in the pathophysiology of obesity and eating disorders. This paper focuses on the role of ghrelin in the regulation of appetite, on its possible role as a hedonic signal involved in food reward, and on its interaction with the corticotropin-releasing-factor system and chronic stress.


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