Plasma fatty acids and reproductive performance of lactating beef cows fed rumen-protected fat

2012 ◽  
Vol 28 (1) ◽  
pp. 89-96 ◽  
Author(s):  
P. Moriel ◽  
E.P. Gonçalves ◽  
B.I. Cappellozza ◽  
V. Nayigihugu ◽  
E. Block ◽  
...  
2011 ◽  
Vol 89 (10) ◽  
pp. 3116-3124 ◽  
Author(s):  
C. N. Lopes ◽  
R. F. Cooke ◽  
M. M. Reis ◽  
R. F. G. Peres ◽  
J. L. M. Vasconcelos

2016 ◽  
Vol 94 (suppl_5) ◽  
pp. 580-581 ◽  
Author(s):  
P. L. P. Fontes ◽  
N. Oosthuizen ◽  
F. M. Ciriaco ◽  
D. D. Henry ◽  
M. E. Garcia-Ascolani ◽  
...  

2020 ◽  
Vol 98 (Supplement_4) ◽  
pp. 202-202
Author(s):  
Reinaldo F Cooke

Abstract Our research group has focused on how Ca salts of soybean oil (CSSO) supplementation, a source of omega-6 fatty acids (FA), impacts reproductive performance of beef cows. In Nelore (Bos indicus) grazing cows assigned to fixed-time artificial insemination (AI), supplementing 100 g/d (as-fed basis) of CSSO for 21 d after AI increased (P < 0.01) pregnancy rates from 38.1% (623/1635 as pregnant/total non-supplemented cows) to 49.0% (843/1720 as pregnant/total CSSO-supplemented cows). These outcomes were later associated with enhanced early embryonic development and pregnancy establishment from supplemental omega-6 FA. More specifically, supplementing 100 g/d of CSSO to Nelore cows increased (P ≤ 0.05) incorporation of omega-6 FA in circulation and in the conceptus, increased (P ≤ 0.04) conceptus length (2.2-fold increase), and upregulated mRNA expression of interferon-tau (4.1-fold increase) on d 15 of gestation. Similar outcomes were noted in B. taurus beef cows receiving 100 g/d of CSSO for 21 d after AI. Supplementing CSSO to Angus-influenced cows increased (P = 0.01) pregnancy rates from 51.7% (193/388 as pregnant/total non-supplemented cows) to 60.2% (226/383 as pregnant/total CSSO-supplemented cows), increased (P < 0.01) plasma concentrations of omega-6 FA, and upregulated (P = 0.05) mRNA expression of interferon-tau in d 15 conceptuses (1.8-fold increase). Across these research efforts, CSSO was mixed with granular feed ingredients and hand-fed to cows, which demands labor and may discourage the use of CSSO supplementation by commercial cow-calf producers. Accordingly, our research group also demonstrated that CSSO inclusion into a self-fed low-moisture block resulted in similar incorporation (P ≥ 0.35) of omega-6 FA in the circulation of Angus-influenced cows compared with CSSO inclusion into a hand-fed granular concentrate. Collectively, CSSO improves reproductive performance of B. indicus and B. taurus beef cows, and may be supplemented via self-fed low-moisture blocks to minimize labor needs.


2021 ◽  
Vol 99 (Supplement_1) ◽  
pp. 137-138
Author(s):  
Morgan T Thayer ◽  
Jacob A Richert ◽  
Karissa N Rulon ◽  
Matthew D Asmus ◽  
Daniel B Jones ◽  
...  

Abstract Forty-seven sows and their progeny were used to determine if feeding gestating and lactating sows a Bacillus licheniformis direct-fed microbial (DFM), an organic acid blend of medium chain and short chain fatty acids (OA), or in combination improves sow lactation feed and water intake, litter growth, and subsequent reproductive performance. On approximately d80 of gestation, sows were fed one of four diets in a 2 x 2 factorial design: 1) gestation control (CON; 0.55% SID Lysine), 2) CON with DFM (1.6x109 CFU/kg of complete feed), 3) CON with 0.4% OA, 4) CON with both DFM and OA. Dietary treatments were also fed throughout lactation (1.00% SID Lysine) starting on approximately d112 of gestation when sows entered farrowing facility. There was a tendency (P = 0.079) for DFM to decrease the amount of sow body weight loss in lactation by approximately 6% compared to sows not consuming the DFM, likely related to DFM sows numerically (P = 0.124) consuming 8.4% more feed during d7-14 of lactation. Sows fed the OA diets had fewer mummies/litter (P = 0.038) compared to diets not containing OA. Sows fed diets with the DFM gave birth to lighter pigs born alive (P = 0.003) compared to non-DFM fed sows, and a tendency for an interaction (P = 0.092) existed where feeding OA+DFM lessened the decrease in born alive BW. There was an interaction tendency (P = 0.133) where sows fed DFM returned to estrus 22 hours sooner than CON, but only 8 hours sooner when sows were fed the OA+DFM diet. In conclusion, feeding a Bacillus licheniformis DFM to sows may decrease pig born alive weight but reduce sow BW loss through 6.4% more lactation feed intake, quickening the return to estrus. Feeding the OA alone or in combination did not improve sow reproductive and litter growth performance in this study, and may require a larger sample size.


Author(s):  
Sara C. Di Rienzi ◽  
Elizabeth L. Johnson ◽  
Jillian L. Waters ◽  
Elizabeth A. Kennedy ◽  
Juliet Jacobson ◽  
...  

1999 ◽  
Vol 276 (5) ◽  
pp. R1425-R1433 ◽  
Author(s):  
Gertjan van Dijk ◽  
Randy J. Seeley ◽  
Todd E. Thiele ◽  
Mark I. Friedman ◽  
Hong Ji ◽  
...  

To investigate whether brain leptin involves neuropeptidergic pathways influencing ingestion, metabolism, and gastrointestinal functioning, leptin (3.5 μg) was infused daily into the third cerebral ventricular of rats for 3 days. To distinguish between direct leptin effects and those secondary to leptin-induced anorexia, we studied vehicle-infused rats with food available ad libitum and those that were pair-fed to leptin-treated animals. Although body weight was comparably reduced (−8%) and plasma glycerol was comparably increased (142 and 17%, respectively) in leptin-treated and pair-fed animals relative to controls, increases in plasma fatty acids and ketones were only detected (132 and 234%, respectively) in pair-fed rats. Resting energy expenditure (−15%) and gastrointestinal fill (−50%) were reduced by pair-feeding relative to the ad libitum group, but they were not reduced by leptin treatment. Relative to controls, leptin increased hypothalamic mRNA for corticotropin-releasing hormone (CRH; 61%) and for proopiomelanocortin (POMC; 31%) but did not reduce mRNA for neuropeptide Y. These results suggest that CNS leptin prevents metabolic/gastrointestinal responses to caloric restriction by activating hypothalamic CRH- and POMC-containing pathways and raise the possibility that these peripheral responses to CNS leptin administration contribute to leptin’s anorexigenic action.


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