Role of Zinc in Testicular Function of Chronic Renal Failure

PEDIATRICS ◽  
1984 ◽  
Vol 73 (5) ◽  
pp. 741-741
Author(s):  
HAROLD K. MARDER ◽  
LAXMI S. SRIVASTAVA ◽  
STEPHEN BURSTEIN
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Zinc Deficiency ◽  
Testicular Function ◽  
Hypergonadotropic Hypogonadism ◽  
Zinc Levels ◽  
Insight Into

In Reply.— We thank Nishi et al for outlining the role that has been suggested for zinc in normal testicular function and the speculation that zinc deficiency might contribute to the hypergonadotropic hypogonadism found in males with chronic renal failure. We have no data concerning zinc levels in our patients, and so we felt we could offer no further insight into the possibility that zinc deficiency might play a role in the etiology of the hypergonadotropism we described.

Role of Zinc in Testicular Function of Chronic Renal Failure

PEDIATRICS ◽  
1984 ◽  
Vol 73 (5) ◽  
pp. 740-740
Author(s):  
YOSHIKAZU NISHI ◽  
SHUICHI HATANO ◽  
KATSUAKI AIHARA ◽  
TOMOFUSA USUI
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Leydig Cells ◽  
Elevated Serum ◽  
Testicular Function ◽  
Plasma Zinc ◽  
Experimental Animals ◽  
Serum Gonadotropin ◽  
Zinc Levels

To the Editor.— We read with great interest the paper of Marder et al1 on hypergonadotropism in peripubertal boys with chronic renal failure. However, their discussion does not mention the role of zinc in testicular function. Zinc deficiency not only reduces zinc levels in the reproductive tissues but also impairs the responsiveness of the Leydig cells to gonadotropins, and thus may cause primary hypogonadism in humans as well as in experimental animals.2-4 Moreover, testicular hypofunction, elevated serum gonadotropin levels, and subnormal plasma zinc levels have been reported in renal failure.5,6

scholarly journals Role of parathyroid hormone in the glucose intolerance of chronic renal failure.

1985 ◽  
Vol 75 (3) ◽  
pp. 1037-1044 ◽  
Author(s):  
M Akmal ◽  
S G Massry ◽  
D A Goldstein ◽  
P Fanti ◽  
A Weisz ◽  
...  
Keyword(s):  
Renal Failure ◽  
Parathyroid Hormone ◽  
Chronic Renal Failure ◽  
Glucose Intolerance

Prevention of Cardiac Hypertrophy in Experimental Chronic Renal Failure by Long-Term ACE Inhibitor Administration: Potential Role of Lysosomal Proteinases

1995 ◽  
Vol 15 (2) ◽  
pp. 129-136 ◽  
Author(s):  
Hiroaki Suzuki ◽  
Liliana Schaefer ◽  
Hong Ling ◽  
Roland M. Schaefer ◽  
Jobst Dämmrich ◽  
...  
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Cardiac Hypertrophy ◽  
Potential Role ◽  
Ace Inhibitor ◽  
Lysosomal Proteinases

scholarly journals ROLE OF CARDIAC MARKERS IN CHRONIC RENAL FAILURE PATIENTS.

2020 ◽  
pp. 81-82
Author(s):  
Ramesh Chandra Thanna ◽  
B K Agarwal ◽  
Rakesh Romday ◽  
Neha Sharma
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Positive Association ◽  
Morbidity And Mortality ◽  
Control Group ◽  
High Morbidity ◽  
C Reactive Protein ◽  
Reactive Protein ◽  
Hs Crp

Introduction: Cardiovascular diseases (CVD) are known as important reasons of the increased morbidity and mortality observed in patients with chronic renal failure (CRF). The association of serum Interlukin-6 , homocysteine as well as other cardiovascular risk factors in relation to existence and cause of CVD were investigated. Method: In this study 200 CRF patients were recruited and further stratified into group with Male and Female as case groups. Those without renal failure were assigned as control group (n=200). Results: The patients with CRF showed a significant increase in plasma levels of Cpk-MB homocysteine and C-reactive protein (CRP) compared to control. The positive association were observed between homocysteine, Urea and Hs-CRP, IL_6 . It shows a significant Association of parameters in CRF . Conclusion: The results demonstrated elevation in plasma values IL-6 , homocysteine and HS-CRP in patients with CRF . However, these modifications may be lead to atherosclerosis and consequence CVD event. These parameters may be important with respect to the high morbidity and mortality of CVD found in patients with CRF.

ANEMIA IN CHRONIC RENAL FAILURE (CRF): ROLE OF OXIDATIVE STRESS AND INFLAMMATION

ASAIO Journal ◽  
2006 ◽  
Vol 52 (2) ◽  
pp. 75A ◽  
Author(s):  
J Simoni ◽  
G Simoni ◽  
J F Moeller ◽  
A Khanna ◽  
W T Bohannon ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Renal Failure ◽  
Chronic Renal Failure

Impaired prolactin response to arginine infusion and insulin hypoglycaemia in chronic renal failure

1983 ◽  
Vol 102 (4) ◽  
pp. 486-491 ◽  
Author(s):  
O. Schmitz ◽  
J. Møller
Keyword(s):  
Growth Hormone ◽  
Renal Failure ◽  
Chronic Renal Failure ◽  
Elevated Level ◽  
Matched Control ◽  
Chronic Haemodialysis ◽  
Arginine Infusion ◽  
Pituitary Dysfunction ◽  
Prolactin Response ◽  
Insight Into

Abstract. The elevated level of circulating prolactin present in the majority of uraemic patients on chronic haemodialysis is primarily due to hypothalamic pituitary dysfunction. So far this defect has been illustrated by demonstration of a blunted prolactin response to TRH and failure of l-dopa to suppress prolactin levels. In the present study two powerful prolactin and growth hormone stimuli, namely iv arginine infusion and insulin hypoglycaemia were applied in a group of uraemic patients on chronic haemodialysis and in age matched control subjects. The prolactin increments to arginine infusion (4.4 ± 1.2 ng/ml vs 17.6 ± 4.6 ng/ml, mean ± se) and to insulin hypoglycaemia (7.9 ± 1.7 ng/ml vs 31.5 ± 5.4 ng/ml) were significantly suppressed in the uraemic patients compared to the controls (P < 0.05). In contradistinction the growth hormone rise provoked by the tests were similar in the two groups. Our results provide further insight into the hypothalamic pituitary derangement in uraemic patients and confirm the presumption of an insensitivity of the lactotrophs to stimulation in uraemic patients.

scholarly journals Omega 3 Polyunsaturated Fatty Acids Improve Endothelial Dysfunction in Chronic Renal Failure: Role of eNOS Activation and of Oxidative Stress

Nutrients ◽  
2017 ◽  
Vol 9 (8) ◽  
pp. 895 ◽  
Author(s):  
Michela Zanetti ◽  
Gianluca Gortan Cappellari ◽  
Davide Barbetta ◽  
Annamaria Semolic ◽  
Rocco Barazzoni
Keyword(s):  
Oxidative Stress ◽  
Fatty Acids ◽  
Renal Failure ◽  
Chronic Renal Failure ◽  
Endothelial Dysfunction ◽  
Polyunsaturated Fatty Acids ◽  
Omega 3
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