scholarly journals The relationship between HSP70 and level of leptin and luteinizing hormones in female rats exposed to chronic and acute heat stress

2020 ◽  
Vol 34 (1) ◽  
pp. 37-43
Author(s):  
Hiyam N. Matty ◽  
Ashwaq A. Hassan
1997 ◽  
Vol 17 (2) ◽  
pp. 83-85 ◽  
Author(s):  
Xiong Yili ◽  
Wu Tangchun ◽  
Zhang Yongxing ◽  
R. M. Tanguay ◽  
L. Nicole ◽  
...  

2021 ◽  
pp. 103040
Author(s):  
Máté Mackei ◽  
Gábor Mátis ◽  
Andor Molnár ◽  
Csilla Sebők ◽  
Júlia Vörösházi ◽  
...  

2018 ◽  
Vol 58 (12) ◽  
pp. 2200 ◽  
Author(s):  
Shu-cheng Huang ◽  
Ya-Fen Fu ◽  
Mujeeb Ur Rehman ◽  
Kun Li ◽  
Yan-fang Lan ◽  
...  

This study was undertaken to investigate the relationship between gut-derived endotoxin and the Toll-like receptor 4 (TLR4) expression in the liver of broilers under acute heat stress (AHS). For this purpose, 120 Arbor Acres chicks were randomised into two groups: control temperature group (CT group, 22 ± 1°C) and high temperature group (HT group, 38 ± 1°C). The chicks received AHS at Day 28 and their small intestine, liver and blood samples were collected after 2 h, 5 h and 10 h to examine the histopathology, biochemical parameters, endotoxin concentrations and TLR4 expression. The results showed that damaged intestinal villi and severe congestion of the hepatic sinusoids were observed, especially after 10 h of AHS in the HT group. In addition, the levels of alanine transferase, aspartate transaminase, and direct bilirubin, except alkaline phosphataseafter were significantly elevated (P < 0.05) and total bilirubin (P < 0.01) and albumin (P < 0.05) were decreased after 10 h of AHS as compared with the CT group, which are associated with liver function. Moreover, the mRNA expression of TLR4 in the liver was noticeably upregulated (P < 0.05) during AHS with significantly increased in endotoxin concentration (P < 0.01) of broilers. Altogether, these findings suggest that the upregulated expression of TLR4 mRNA was triggered via gut-derived endotoxin in heat stress-induced liver injury.


2020 ◽  
Vol 94 ◽  
pp. 102759
Author(s):  
Yoshimitsu Ouchi ◽  
Hiroshi Tanizawa ◽  
Jun-ichi Shiraishi ◽  
John F. Cockrem ◽  
Vishwajit S. Chowdhury ◽  
...  

animal ◽  
2020 ◽  
pp. 100106
Author(s):  
P. Adu-Asiamah ◽  
Y. Zhang ◽  
K. Amoah ◽  
Q.Y. Leng ◽  
J.H. Zheng ◽  
...  

2003 ◽  
Vol 26 (5) ◽  
pp. 701-708 ◽  
Author(s):  
Naoki Harikai ◽  
Kanji Tomogane ◽  
Mitsue Miyamoto ◽  
Keiko Shimada ◽  
Satoshi Onodera ◽  
...  

Author(s):  
Andrea Kőrösi Molnár ◽  
László Kőrösi ◽  
Bence Balázs ◽  
András Gáspárdy

AbstractHeat stress affects the performance of poultry species and also induces immunosuppression. Chickens can be treated by thermal conditioning to have better heat stress tolerance. Our purpose was to determine the effect of acute heat stress on the immune response, i.e. antibody production against Newcastle disease virus (NDV) and change in the proportion of leukocyte components, in chicks subjected to prenatal heat conditioning. Eighty as-hatched broiler chicks from the same parent stock were used: control (40 chicks incubated at 36.7 °C from days 18–20 of embryonic life) and thermally manipulated (TM) (40 chicks incubated at 38.4 °C from day 18–20 of embryonic life; 4 h/day). The chickens were exposed to heat stress: at day 19 (31 °C/8 h) and at day 35 (32 °C/10 h). The first heat stress (day 19) decreased the lymphocyte counts and significantly increased the heterophil counts (P < 0.05) in both treatments (from 34.25 to 55% in the controls and from 37 to 60.06% in the TM chicks). The second heat stress (day 35) did not alter the heterophil and lymphocyte profiles of the chickens. Before the heat stress, all of the chickens (control and TM) presented the same positive antibody titres to NDV vaccination. After the first heat stress, 50% of the control samples and 40% of samples from the TM chickens were negative. After the second heat stress, 80% of the TM samples were negative.


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