scholarly journals Asociaciones entre alimentos, flora intestinal y sistema nervioso central

2018 ◽  
Vol 2 (1) ◽  
pp. 35
Author(s):  
Angel Eladio Caballero Torres ◽  
Yumy Estela Fernández Vélez
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Scientific Information ◽  
Intestinal Flora ◽  
Intestinal Microbiome ◽  
National Library ◽  
El Sistema ◽  
System Functioning ◽  
The Central Nervous System ◽  
Relationship Of

  Existe un incremento del interés sobre posibles efectos de la alimentación en el funcionamiento del sistema nervioso central, aunque la importancia de esa influencia parece requerir mayores respaldos científicos. Estos señalamientos son motivos para   valorar, sobre la base de los aportes de otros autores, el posible significado de la relación de alimentos, flora intestinal y sistema nervioso. Para cumplir este objetivo fue necesario una búsqueda de informaciones científicas en la US National Library of Medicine sobre este tema, una selección de publicaciones relevantes y análisis de los datos encontrados. Según esas publicaciones, la protección de la inocuidad de los alimentos, probióticos y algunas sustancias químicas de la dieta tienen relación con la composición y funcionamiento de la flora intestinal que puede afectar el eje intestino cerebro y causar alteraciones en el funcionamiento del sistema nervioso central. Se acepta que son insuficientes las explicaciones sobre relaciones específicas entre componentes de la dieta y efectos en el sistema nervioso central de los consumidores de alimentos, incluyendo los posibles mecanismos de esas relaciones.   Palabras clave: alimentos, eje intestino cerebro, sistema nervioso central, microbiota intestinal.   Abstract   There is an increased interest in possible effects of food on the central nervous system functioning, although the importance of this influence seems to require more scientific support. These indications are reasons to value, based on the contributions of other authors, the possible meaning of the relationship of food with the intestinal flora and the nervous system. To accomplish this goal, a search of scientific information was necessary for the US National Library of Medicine on this topic, a selection of relevant publications and analysis of the data found. According to those publications, food safety, probiotics and some dietary chemicals has a relationship with to the composition and functioning of the intestinal microbiome that can affect the brain intestinal axis and cause alterations in the functioning of the central nervous system. It’s accepted that explanations about specific relationships between diet components and effects on the central nervous system of food consumers, including the possible mechanisms of these relationships, are insufficient.   Key words: food, intestine brain axis, central nervous system, nutrition, intestinal microbiota.

scholarly journals Asociaciones entre alimentos, flora intestinal y sistema nervioso central

2018 ◽  
Vol 2 (1) ◽  
pp. 35
Author(s):  
Angel Eladio Caballero Torres ◽  
Yumy Estela Fernández Vélez
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Scientific Information ◽  
Intestinal Flora ◽  
Intestinal Microbiome ◽  
National Library ◽  
El Sistema ◽  
System Functioning ◽  
The Central Nervous System ◽  
Relationship Of

  Existe un incremento del interés sobre posibles efectos de la alimentación en el funcionamiento del sistema nervioso central, aunque la importancia de esa influencia parece requerir mayores respaldos científicos. Estos señalamientos son motivos para   valorar, sobre la base de los aportes de otros autores, el posible significado de la relación de alimentos, flora intestinal y sistema nervioso. Para cumplir este objetivo fue necesario una búsqueda de informaciones científicas en la US National Library of Medicine sobre este tema, una selección de publicaciones relevantes y análisis de los datos encontrados. Según esas publicaciones, la protección de la inocuidad de los alimentos, probióticos y algunas sustancias químicas de la dieta tienen relación con la composición y funcionamiento de la flora intestinal que puede afectar el eje intestino cerebro y causar alteraciones en el funcionamiento del sistema nervioso central. Se acepta que son insuficientes las explicaciones sobre relaciones específicas entre componentes de la dieta y efectos en el sistema nervioso central de los consumidores de alimentos, incluyendo los posibles mecanismos de esas relaciones.   Palabras clave: alimentos, eje intestino cerebro, sistema nervioso central, microbiota intestinal.   Abstract   There is an increased interest in possible effects of food on the central nervous system functioning, although the importance of this influence seems to require more scientific support. These indications are reasons to value, based on the contributions of other authors, the possible meaning of the relationship of food with the intestinal flora and the nervous system. To accomplish this goal, a search of scientific information was necessary for the US National Library of Medicine on this topic, a selection of relevant publications and analysis of the data found. According to those publications, food safety, probiotics and some dietary chemicals has a relationship with to the composition and functioning of the intestinal microbiome that can affect the brain intestinal axis and cause alterations in the functioning of the central nervous system. It’s accepted that explanations about specific relationships between diet components and effects on the central nervous system of food consumers, including the possible mechanisms of these relationships, are insufficient.   Key words: food, intestine brain axis, central nervous system, nutrition, intestinal microbiota.

On humoral factors in nervous activity

1935 ◽  
Vol 31 (6) ◽  
pp. 777-787
Author(s):  
D. S. Vorontsov
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Nervous Activity ◽  
Active Part ◽  
Humoral Factors ◽  
The Central Nervous System ◽  
Relationship Of ◽  
The Relationship

Not only in the peripheral working organs, irritating substances are formed, which, as we can see, take an active part in their regulation, but also in the central nervous system, in the relationship of its individual elements, such substances apparently play an important role.

Diabetes mellitus and the central nervous system

2016 ◽  
Vol 88 (10) ◽  
pp. 82-86 ◽  
Author(s):  
E V Surkova
Keyword(s):  
Diabetes Mellitus ◽  
Central Nervous System ◽  
Nervous System ◽  
Blood Glucose Control ◽  
Current Data ◽  
Treatment And Prevention ◽  
Imaging Results ◽  
The Central Nervous System ◽  
Considerable Impact ◽  
Relationship Of

The review considers the current views on the central nervous system (CNS) in diabetes mellitus (DM). It discusses an attitude towards the term «diabetic encephalopathy». The data of investigations of cognitive functions in types 1 and 2 DM and the brain structural imaging results obtained using up-to-date technologies are considered. The results of studies of the factors that induce cerebral changes in DM and their associated cognitive impairments are given. There is evidence that hyperglycemia has a more considerable impact on the above processes than hypoglycemia; other possible factors, apart from blood glucose control, are set out. The current views on the function of insulin in the CNS and the relationship of central insulin resistance to Alzheimer’s disease are outlined. There are current data on intranasal insulin application that is still exploratory, but, as might be judged from the findings, may by a promising method for the treatment and prevention of cognitive decline in both patients with DM and those without this condition.

scholarly journals Maintaining the Balance of Intestinal Flora through the Diet: Effective Prevention of Illness

Foods ◽  
2021 ◽  
Vol 10 (10) ◽  
pp. 2312
Author(s):  
Li Zhang ◽  
Zhenying Zhang ◽  
Lei Xu ◽  
Xin Zhang
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Human Health ◽  
Gut Microbiome ◽  
Intestinal Flora ◽  
Dietary Factors ◽  
Signal Molecules ◽  
Effective Prevention ◽  
The Central Nervous System ◽  
The Brain

The human body is home to a complex community of dynamic equilibrium microbiota, including bacteria, fungi, parasites, and viruses. It is known that the gut microbiome plays a crucial role in regulating innate and adaptive immune responses, intestinal peristalsis, intestinal barrier homeostasis, nutrient uptake, and fat distribution. The complex relationship between the host and microbiome suggests that when this relationship is out of balance, the microbiome may contribute to disease development. The brain–gut–microbial axis is composed of many signal molecules, gastrointestinal mucosal cells, the vagus nerve, and blood–brain barrier, which plays an essential role in developing many diseases. The microbiome can influence the central nervous system function through the brain–gut axis; the central nervous system can also affect the composition and partial functions of the gut microbiome in the same way. Different dietary patterns, specific dietary components, and functional dietary factors can significantly affect intestinal flora’s structure, composition, and function, thereby affecting human health. Based on the above, this paper reviewed the relationship between diet, intestinal flora, and human health, and the strategies to prevent mental illness through the dietary modification of intestinal microorganisms.

scholarly journals XXII. Contributions to the anatomy of the central nervous system vertebrate animals

1878 ◽  
Vol 169 ◽  
pp. 735-776 ◽  
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Art Of Medicine ◽  
General Terms ◽  
The Central Nervous System ◽  
Relationship Of ◽  
Morphological Relationship ◽  
The Brain

Investigations into the histology of the nervous system in Vertebrata, have hitherto been chiefly confined to that of the Mammalia, or if applied to the lower members of that sub-kingdom, have not been undertaken so much with reference to the morphological relationship of animals, as to their bearings on physiology and the art of medicine. It may be stated in general terms that those who have worked at the morphology of the nervous system have not paid much attention to its histology; and, per contra, those who have investigated the histology have neglected its morphological bearing. Stieda, however, has investigated the brain in both aspects of the question, and Lockhart Clarke has made some reference to the nervous system of the lower Vertebrata in his classical investigations, but this bears but a subordinate proportion to the whole. Fritsch has recently published a treatise on the histology of the central nervous system in Fishes, in which the homology of the various parts is especially considered.

IL-17 controls central nervous system autoimmunity through the intestinal microbiome

2021 ◽  
Vol 6 (56) ◽  
pp. eaaz6563 ◽  
Author(s):  
Tommy Regen ◽  
Sandrine Isaac ◽  
Ana Amorim ◽  
Nicolás Gonzalo Núñez ◽  
Judith Hauptmann ◽  
...  
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Inflammatory Diseases ◽  
Intestinal Microbiome ◽  
Autoimmune Encephalomyelitis ◽  
Interleukin 17A ◽  
The Central Nervous System ◽  
Effector Cytokines ◽  
Experimental Autoimmune

Interleukin-17A– (IL-17A) and IL-17F–producing CD4+ T helper cells (TH17 cells) are implicated in the development of chronic inflammatory diseases, such as multiple sclerosis and its animal model, experimental autoimmune encephalomyelitis (EAE). TH17 cells also orchestrate leukocyte invasion of the central nervous system (CNS) and subsequent tissue damage. However, the role of IL-17A and IL-17F as effector cytokines is still confused with the encephalitogenic function of the cells that produce these cytokines, namely, TH17 cells, fueling a long-standing debate in the neuroimmunology field. Here, we demonstrated that mice deficient for IL-17A/F lose their susceptibility to EAE, which correlated with an altered composition of their gut microbiota. However, loss of IL-17A/F in TH cells did not diminish their encephalitogenic capacity. Reconstitution of a wild-type–like intestinal microbiota or reintroduction of IL-17A specifically into the gut epithelium of IL-17A/F–deficient mice reestablished their susceptibility to EAE. Thus, our data demonstrated that IL-17A and IL-17F are not encephalitogenic mediators but rather modulators of intestinal homeostasis that indirectly alter CNS-directed autoimmunity.

DEATHS IN CONGENITAL ADRENAL HYPERPLASIA

PEDIATRICS ◽  
1962 ◽  
Vol 29 (1) ◽  
pp. 3-17
Author(s):  
William W. Cleveland ◽  
Orville C. Green ◽  
Lawson Wilkins
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Congenital Adrenal Hyperplasia ◽  
Cause Of Death ◽  
Terminal Illness ◽  
Adrenal Hyperplasia ◽  
The Central Nervous System ◽  
Relationship Of

Since the advent of cortisone for treatment of congenital adrenal hyperplasia, eight patients have died, representing a mortality of approximately 8%. These cases are presented, with an analysis of the findings in an attempt to explain the cause of death. In three of these cases errors in management were considered as factors in the terminal illness. One child died of an unrelated disorder. The remaining four died under circumstances that allow no ready explanation for their death. The possible mechanisms in these patients are discussed, including a poorly understood relationship of disturbance in the central nervous system to disorders of adrenal metabolism.

PATHOGENESIS OF LESIONS IN THE NERVOUS SYSTEM IN HYPERNATREMIC STATES

PEDIATRICS ◽  
1959 ◽  
Vol 23 (1) ◽  
pp. 46-53 ◽  
Author(s):  
Laurence Finberg ◽  
Charles Luttrell ◽  
Henry Redd
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Chemical Composition ◽  
Clinical Manifestations ◽  
Brain Cells ◽  
Intracranial Hemorrhages ◽  
The Central Nervous System ◽  
Inorganic Chemical ◽  
Relationship Of ◽  
Hypernatremic Dehydration

In an attempt to verify the possible causal relationship of hypernatremic dehydration and damage to the central nervous system and to study the mechanisms involved, kittens were made hypernatremic by injection with hypertonic solutions of sodium. Many of the clinical manifestations referable to the nervous system as seen in patients with hypernatremic dehydration were produced. Gross anatomic changes consisting of intracranial hemorrhages, such as are sometimes seen in patients, were also present in the animals. A study of the inorganic chemical composition of the tissues revealed a difference in the pattern of osmotic adjustment between cells of brain and of muscle. This is interpreted as a relative excess from development of idiogenic osmols within brain cells. It is suggested that this difference may in part account for the marked manifestations related to the nervous system seen in the animals and, by analogy, in patients.

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