scholarly journals Level of Radiofrequency (RF) Radiations from GSM Base Stations and its Biological Effects on Albino Mice, Mus musculus

2010 ◽  
Vol 14 (3) ◽  
Author(s):  
AA Otitoloju ◽  
VO Osunkalu ◽  
IA Obe ◽  
OA Adewale ◽  
OR Akinde
Keyword(s):  
Mus Musculus ◽  
Biological Effects ◽  
Base Stations ◽  
Albino Mice

scholarly journals Effect of Poly (N-IsoPropylAcrylamide) “PNIPAM” on Hepatic Cells of Swiss Albino Mice, Mus musculus

2013 ◽  
Vol 1 (4) ◽  
pp. 208-213 ◽  
Author(s):  
Md. Imtiyaz Aslam ◽  
Rohit Kumar Verma ◽  
Richa Roy ◽  
S. P. Roy
Keyword(s):  
Aqueous Solution ◽  
Mus Musculus ◽  
Quantitative Estimation ◽  
Polymeric Compound ◽  
Swiss Albino Mice ◽  
Hepatic Cells ◽  
Isopropyl Acrylamide ◽  
Albino Mice ◽  
Xenobiotic Compound ◽  
Histological Result

The present paper deals with the effect of polymeric compound poly (N-isopropyl acrylamide) “PNIPAM” for their toxicity on hepatic cells. The nanoparticle is a xenobiotic compound that accumulates in the liver for their metabolism. Non-metabolizing xenobiotic compounds such as PNIPAM produces anomalies in the hepatic cells. Certain enzymes such as ALT and ALP can be assayed for the hepatocytic toxicity.An attempt has been made to know the toxic effect of “PNIPAM” in a concentration of 0.8mg/ml on the hepatic cells of Swiss Albino mice, Mus musculus. The ALT and ALP analysis were performed through test kits for their quantitative estimation. The histological result shows that several lesions were produced after the introduction of aqueous solution of PNIPAM for an incubation period of 48 hours. The toxicity was confirmed Spectrophotometrically by the assessment of enzyme ALT and ALP. The increased concentration of ALT (55.0 IU/L) and slight decrease in ALP (40.0 IU/L) concentration was responsible for the metabolic alteration and production of hepatocytic anomalies in the mice.DOI: http://dx.doi.org/10.3126/ijasbt.v1i4.9104    Int J Appl Sci Biotechnol, Vol. 1(4): 208-213

scholarly journals Preventive measures to reduce harmful effects produced by electromagnetic radiation on health

2021 ◽  
pp. 42-53
Author(s):  
Y. Stein ◽  
◽  
Keyword(s):  
Electromagnetic Radiation ◽  
Electromagnetic Waves ◽  
Preventive Measures ◽  
Biological Effects ◽  
Scientific Evidence ◽  
Base Stations ◽  
Power Lines ◽  
Thermal Safety ◽  
Low Frequencies ◽  
Safety Standards

Man-made electromagnetic waves are the most widely and rapidly expanding exposure in today's world, including exposure in several frequency groups: extremely low frequencies (ELF) from electricity lines, hybrid car batteries and high power lines (>3 Hz–3 kHz), radiofrequency (RF) and microwave frequencies including millimeter waves (3 kHz–300 GHz) from mobile phones, towers, base stations and wireless devices, and intermediate frequencies "Dirty Electricity" emitted from power lines. While such organizations as ICNIRP (the International Commission on Non-Ionizing Radiation Protection) still continue to claim that electromagnetic radiation can cause "only thermal effects", clinging to theory that does not match facts and upholding obsolete thermal safety standards, extensive scientific evidence has clearly demonstrated that non-thermal health effects produced by electromagnetic radiation do exist, are important to health, and should be taken into consideration when safety standards are set. This review aims to highlight some evidence of biologic effects in various body systems, and to suggest preventive measures to reduce such effects on health. Exposure to electromagnetic radiation at intensities lower than thermal safety standards has been associated with non-thermal biological effects including damage and changes to cells and DNA. This review presents evidence of such effects demonstrated in: the hematologic system, the nervous system, the immune system, the reproductive system, the skin and muscles, the cardiovascular system, glucose metabolism, and Electrohypersensitivity ("Microwave sickness"). Protective measures are then suggested to reduce these effects.

scholarly journals Histological Study of the Liver and Kidney of Albino Mice Mus musculus Exposed to Lead

2009 ◽  
Vol 20 (3) ◽  
pp. 42-51
Author(s):  
Aticka A. El-Nouri ◽  
Muna H. Jankeer
Keyword(s):  
Mus Musculus ◽  
Histological Study ◽  
Liver And Kidney ◽  
Albino Mice

scholarly journals Arsenic induced genotoxic and histopathological changes in male Swiss albino mice, Mus musculus

2011 ◽  
Vol 3 (2) ◽  
pp. 329-339
Author(s):  
Animesh K. Mohapatra ◽  
Deepika Rai ◽  
Anika Tyagi
Keyword(s):  
Arsenic Trioxide ◽  
Mus Musculus ◽  
Seminiferous Tubules ◽  
Histopathological Changes ◽  
Histological Changes ◽  
Hydropic Degeneration ◽  
Swiss Albino Mice ◽  
Cytoplasmic Vacuolization ◽  
Liver And Kidney ◽  
Albino Mice

The present study was carried out to investigate the effect of arsenic trioxide on the DNA and histomorphology of testis, liver and kidney of Swiss albino mice, Mus musculus. Oral administration of arsenic trioxide induced DNA damage in the testis, liver and kidney marked by light pink staining of nuclei after Feulgen’s reaction with reduced fine chromatin. Simultaneously severe histological changes were noted like distortion of seminiferous tubules, disorganization of spermatogonia, spermatocytes and spermatids with cytoplasmic vacuolization and nuclear pycnosis in testis. There was almost disappearance of sinusoids due to disruption of hepatic plates, inflammatory cellular infiltration around central veins and cytoplasmic vacuolization in hepatocytes with large irregular nuclei in liver of treated mice. Disorganized glomeruli with distorted Bowman’s capsules and mild to severe multifocal cloudy and hydropic degeneration with necrosis of tubules were observed in the kidney of treated mice. Inference drawn from the study indicated that arsenic induced both genotoxic histotoxic lesions.

scholarly journals Acute toxicity study of Phyllanthus niruri and its effect on the cyto-architectural structure of nephrocytes in Swiss albino mice Mus-musculus

2015 ◽  
Vol 8 (1) ◽  
pp. 77-80
Author(s):  
Tanuja Singh ◽  
Ruchi x ◽  
Anjali Singh ◽  
Ravish Kumar ◽  
Jitendra Kumar Singh
Keyword(s):  
Acute Toxicity ◽  
Mus Musculus ◽  
Toxicity Study ◽  
Phyllanthus Niruri ◽  
Swiss Albino Mice ◽  
Acute Toxicity Study ◽  
Albino Mice

scholarly journals PENGARUH TRITERPEN TOTAL PEGAGAN (Centella asiatica (L) Urban) TERHADAP FUNGSI KOGNITIF BELAJAR DAN MENGINGAT PADA MENCIT JANTAN ALBINO (Mus musculus) YANG DIHAMBAT DENGAN SKOPOLAMIN

Molekul ◽  
2010 ◽  
Vol 5 (2) ◽  
pp. 89
Author(s):  
Herlina Herlina
Keyword(s):  
Cognitive Function ◽  
Learning And Memory ◽  
Mus Musculus ◽  
Memory Performance ◽  
Centella Asiatica ◽  
Positive Control ◽  
Negative Control ◽  
Learning Ability ◽  
Control Group ◽  
Albino Mice

Pegagan (Centella asiatica (L) Urban) has been described to posses CNS effects such as improving cognitive function, learning and memory. The aim of the research was to evaluate the effects of total triterpen’s pegagan extract on cognitive functions as the learning and memory performance in male albino mice (Mus musculus) inhibited by scopolamine. The research design was Complete Randomized Design (RAL) – factorial on thirty six mice divided into 4 groups. One control group received only aquabidest (negative control). Three treatment groups received total triterpen 16 mg/kg BW, 32 mg/kg BW orally and piracetam 500 mg/kg BW by intra peritoneally (positive control) for 21 days. Data indicating learning and memory process of all subjects were obtained from one-trial passive avoidance test. Data were analyzed by two way ANOVA and BNT (p<0,05). Result showed that total triterpen 32 mg/kg BW had significantly prolonged the retention time compared to control group indicating improvement in cognitive function (505,03 second vs -18,53 second) (p<0,05) and it was not significantly different to piracetam 500 mg/kg BW group (505,03 second vs 522,48 second) (p>0,05). In conclusion, total triterpen from pegagan (Centella asiatica (L) Urban) improved learning ability and memory of male albino mice (Mus musculus) even though, it was inhibited by scopolamine.

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