scholarly journals Myxoid liposarcoma - soleus muscle

2021 ◽  
Author(s):  
Ammar Haouimi
Author(s):  
Aggeliki Bistaraki ◽  
Maria Zarokosta ◽  
Theodoros Mariolis Sapsakos ◽  
George Skarpas ◽  
Georgios Nousios ◽  
...  

Diabetes ◽  
1987 ◽  
Vol 36 (9) ◽  
pp. 1041-1046 ◽  
Author(s):  
S. Sasson ◽  
D. Edelson ◽  
E. Cerasi

Diabetes ◽  
1992 ◽  
Vol 41 (2) ◽  
pp. 215-221 ◽  
Author(s):  
A. Bornemann ◽  
T. Ploug ◽  
H. Schmalbruch

Author(s):  
Evgeniya S. Shitova ◽  
Inga S. Malakhova ◽  
Vladislav I. Lemeshko

Introduction. The use of classical methods for diagnosing muscle fatigue of physical workers, including dynamometry and electromyography, is often limited due to the complexity of the process, the inability to use them in production, and the subjectivity of the methodology. At the same time, such a method as myotonometry does not have these disadvantages, but the main area of its use at the moment is clinical practice. The aim of study was to determine the possibility of using myotonometry to assess muscle fatigue. Materials and methods. In the course of the study, the biomechanical characteristics of muscles that differ in their depth were evaluated using the "MyotonPro" device (Myoton AS, Estonia). We determined the tone and stiffness of the biceps of the shoulder, the soleus muscle, and the muscle that straightens the back at different periods of time under the influence of maximum load on them. Results. It was found that the studied parameters of these muscles immediately after performing a series of exercises "to failure" statistically significantly increased, and after 30 minutes - again decreased, but did not reach the initial level. Conclusions. The myotonometric study showed objectivity, reliability, repeatability and proved to be one of the most promising modern methods for assessing muscle fatigue, including for solving problems in the field of occupational health.


Nutrients ◽  
2021 ◽  
Vol 13 (2) ◽  
pp. 379
Author(s):  
Luchuanyang Sun ◽  
Nobuyuki Miyaji ◽  
Min Yang ◽  
Edward M. Mills ◽  
Shigeto Taniyama ◽  
...  

Astaxanthin (AX) is a carotenoid that exerts potent antioxidant activity and acts in the lipid bilayer. This study aimed to investigate the effects of AX on muscle-atrophy-mediated disturbance of mitochondria, which have a lipid bilayer. Tail suspension was used to establish a muscle-atrophied mouse model. AX diet fed to tail-suspension mice prevented loss of muscle weight, inhibited the decrease of myofiber size, and restrained the increase of hydrogen peroxide (H2O2) production in the soleus muscle. Additionally, AX improved downregulation of mitochondrial respiratory chain complexes I and III in the soleus muscle after tail suspension. Meanwhile, AX promoted mitochondrial biogenesis by upregulating the expressions of adenosine 5′-monophosphate–activated protein kinase (AMPK) α-1, peroxisome proliferator–activated receptor (PPAR)-γ, and creatine kinase in mitochondrial (Ckmt) 2 in the soleus muscle of tail-suspension mice. To confirm the AX phenotype in the soleus muscle, we examined its effects on mitochondria using Sol8 myotubes derived from the soleus muscle. We found that AX was preferentially detected in the mitochondrial fraction; it significantly suppressed mitochondrial reactive oxygen species (ROS) production in Sol8 myotubes. Moreover, AX inhibited the activation of caspase 3 via inhibiting the release of cytochrome c into the cytosol in antimycin A–treated Sol8 myotubes. These results suggested that AX protected the functional stability of mitochondria, alleviated mitochondrial oxidative stress and mitochondria-mediated apoptosis, and thus, prevented muscle atrophy.


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