RECENT INVESTIGATIONS BEARING ON INFECTIOUS DISEASES OF UNKNOWN ETIOLOGY.

JAMA ◽  
1903 ◽  
Vol XLI (7) ◽  
pp. 405
Author(s):  
LUDVIG HEKTOEN
Keyword(s):  
Infectious Diseases ◽  
Unknown Etiology

scholarly journals Bilateral Optic Atrophy with Thickened Meninges (Idiopathic Hypertrophic Pachymeningitis) - A Rare Entity

JMS SKIMS ◽  
2011 ◽  
Vol 14 (1) ◽  
pp. 25-27
Author(s):  
Ravouf Asmi ◽  
Feroze Ahmad ◽  
Rangreze Imran ◽  
Zarka Amin Masoodi ◽  
Mushtaq A Wani
Keyword(s):  
Infectious Diseases ◽  
Optic Atrophy ◽  
Visual Loss ◽  
Unknown Etiology ◽  
Inflammatory Disorder ◽  
Rare Entity ◽  
Hypertrophic Pachymeningitis ◽  
Open Biopsy

Idiopathic hypertrophic pachymeningitis (IHP) is a rare, chronic nonspecific and granulomatous inflammatory disorder of the dura with unknown etiology. The diagnosis can be established by an open biopsy and exclusion of all other specific granulomatous and infectious diseases. We report a case of Idiopathic hypertrophic pachymeningitis who manifested primarily by headache and visual loss with optic atrophy. The patient was thoroughly investigated and showed no evidence of underlying cause. The diagnosis was established by an open biopsy. JMS 2011;14(1):25-27

scholarly journals Rickettsioses as paradigms of new or emerging infectious diseases.

1997 ◽  
Vol 10 (4) ◽  
pp. 694-719 ◽  
Author(s):  
D Raoult ◽  
V Roux
Keyword(s):  
Molecular Biology ◽  
Infectious Diseases ◽  
Emerging Infectious Diseases ◽  
Unknown Etiology ◽  
Pathogenic Potential ◽  
Laboratory Methods ◽  
Rickettsia Species ◽  
Intracellular Location ◽  
The Impact

Rickettsioses are caused by species of Rickettsia, a genus comprising organisms characterized by their strictly intracellular location and their association with arthropods. Rickettsia species are difficult to cultivate in vitro and exhibit strong serological cross-reactions with each other. These technical difficulties long prohibited a detailed study of the rickettsiae, and it is only following the recent introduction of novel laboratory methods that progress in this field has been possible. In this review, we discuss the impact that these practical innovations have had on the study of rickettsiae. Prior to 1986, only eight rickettsioses were clinically recognized; however, in the last 10 years, an additional six have been discovered. We describe the different steps that resulted in the description of each new rickettsiosis and discuss the influence of factors as diverse as physicians' curiosity and the adoption of molecular biology-based identification in helping to recognize these new infections. We also assess the pathogenic potential of rickettsial strains that to date have been associated only with arthropods, and we discuss diseases of unknown etiology that may be rickettsioses.

Case Studies

2017 ◽  
Author(s):  
George J. Armelagos ◽  
Dennis P. Van Gerven
Keyword(s):  
Differential Diagnosis ◽  
Infectious Diseases ◽  
Case Studies ◽  
Unknown Etiology ◽  
Congenital Defects ◽  
Medical Evidence ◽  
Neoplastic Diseases

Chapter 6 presents case studies of diseases that afflict individuals rather than their communities at large. The conditions are presented in these categories: neoplastic diseases (cancers), congenital defects, infectious diseases, and diseases of unknown etiology. A differential diagnosis is made for each disease using a combination of the skeletal and medical evidence. The diseases are then discussed in terms of their likely impact on the lives of the afflicted individual.

Immunohemolytic Anemia and Hodgkin Disease

PEDIATRICS ◽  
1982 ◽  
Vol 70 (2) ◽  
pp. 320-321
Author(s):  
Ugo Carpentieri ◽  
C. W. Daeschner ◽  
Mary Ellen Haggard
Keyword(s):  
Infectious Diseases ◽  
Hemolytic Anemia ◽  
Gastrointestinal Diseases ◽  
Hodgkin Disease ◽  
Unknown Etiology ◽  
Immune Disorders ◽  
Erythrocyte Transfusion ◽  
Chemical Agents ◽  
Common Causes ◽  
Made In

The proteiform presentation of Hodgkin disease is well known. Also recognized are the immune disorders such as immunohemolytic anemia and immunothrombocytopenia which may be associated with, and sometimes precede, the disease.1(pp1025-1030) A diagnosis of warm agglutinin Coombs positive hemolytic anemia of unknown etiology was made in an 8-year-old black boy after the most common causes, such as chemical agents, viral syndromes, malignancies, infectious diseases, collagen diseases, gastrointestinal diseases, and thyroid problems were apparently excluded. The patient was treated with erythrocyte transfusion and steroids.

Isolation of Asbestos from Lung Tissue and Sputum

1982 ◽  
Vol 40 ◽  
pp. 330-331
Author(s):  
M. G. Williams ◽  
C. Corn ◽  
R. F. Dodson ◽  
G. A. Hurst
Keyword(s):  
Sodium Hypochlorite ◽  
Lung Tissue ◽  
Body Fluids ◽  
Unknown Etiology ◽  
The Past

During this century, interest in the particulate content of the organs and body fluids of those individuals affected by pneumoconiosis, cancer, or other diseases of unknown etiology developed and concern was further prompted with the increasing realization that various foreign particles were associated with or caused disease. Concurrently particularly in the past two decades, a number of methods were devised for isolating particulates from tissue. These methods were recently reviewed by Vallyathan et al. who concluded sodium hypochlorite digestion was both simple and superior to other digestion procedures.

Use of TEM in Plant Disease Diagnosis: A Xylem-Limited Bacterium Associated with Diseased ‘Toronto’ Creeping Bentgrass

1981 ◽  
Vol 39 ◽  
pp. 414-415
Author(s):  
Karen K. Baker ◽  
David L. Roberts
Keyword(s):  
Osmium Tetroxide ◽  
Plant Disease ◽  
Leaf Tissue ◽  
Infectious Agent ◽  
Creeping Bentgrass ◽  
Disease Diagnosis ◽  
Unknown Etiology ◽  
Agrostis Palustris ◽  
Putting Greens ◽  
Plant Disease Diagnosis

Plant disease diagnosis is most often accomplished by examination of symptoms and observation or isolation of causal organisms. Occasionally, diseases of unknown etiology occur and are difficult or impossible to accurately diagnose by the usual means. In 1980, such a disease was observed on Agrostis palustris Huds. c.v. Toronto (creeping bentgrass) putting greens at the Butler National Golf Course in Oak Brook, IL.The wilting symptoms of the disease and the irregular nature of its spread through affected areas suggested that an infectious agent was involved. However, normal isolation procedures did not yield any organism known to infect turf grass. TEM was employed in order to aid in the possible diagnosis of the disease.Crown, root and leaf tissue of both infected and symptomless plants were fixed in cold 5% glutaraldehyde in 0.1 M phosphate buffer, post-fixed in buffered 1% osmium tetroxide, dehydrated in ethanol and embedded in a 1:1 mixture of Spurrs and epon-araldite epoxy resins.

The effects of Cyclosporin-A (CYA) on the Ultrastructure of Gingival Mast Cells (GMC) in Behcet's disease (BD)

1990 ◽  
Vol 48 (3) ◽  
pp. 358-359
Author(s):  
Oktay Arda ◽  
Ulkü Noyan ◽  
Selgçk Yilmaz ◽  
Mustafa Taşyürekli ◽  
İsmail Seçkin ◽  
...  
Keyword(s):  
Unknown Etiology ◽  
Control Group ◽  
Target Cells ◽  
Gingival Hyperplasia ◽  
Cellular Activity ◽  
Direct Relation ◽  
Immune Disease ◽  
Genital Ulceration ◽  
Functional Changes ◽  
The Third

Turkish dermatologist, H. Beheet described the disease as recurrent triad of iritis, oral aphthous lesions and genital ulceration. Auto immune disease is the recent focus on the unknown etiology which is still being discussed. Among the other immunosupressive drugs, CyA included in it's treatment newly. One of the important side effects of this drug is gingival hyperplasia which has a direct relation with the presence of teeth and periodontal tissue. We are interested in the ultrastructure of immunocompetent target cells that were affected by CyA in BD.Three groups arranged in each having 5 patients with BD. Control group was the first and didn’t have CyA treatment. Patients who had CyA, but didn’t show gingival hyperplasia assembled the second group. The ones displaying gingival hyperplasia following CyA therapy formed the third group. GMC of control group and their granules are shown in FIG. 1,2,3. GMC of the second group presented initiation of supplementary cellular activity and possible maturing functional changes with the signs of increased number of mitochondria and accumulation of numerous dense cored granules next to few normal ones, FIG. 4,5,6.

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