One of Barry Cunliffe’s abiding research interests has been in the character of cross-Channel interaction during the Iron Age, a topic that he has pursued and illuminated through a sustained programme of excavations and artefact studies in southern England, northern France, and the Channel Islands. Although the exchanges were undoubtedly two-way—and must also be seen in the context of a longer-term pattern of maritime contacts between Britain and its neighbours across the ocean (cf. Cunliffe 2001)— it remains true that for the late Iron Age, much of the material evidence for relations between Britain and France is in the form of continental imports found in Britain (e.g. Cunliffe 1987), rather than the other way around. We are therefore very pleased here, following a new find of British Iron Age coins in France, to be able to offer Barry a study of a relatively rare example of a group of objects moving in the opposite direction, not least because another of Barry’s contributions over the years has been to ensure that the Celtic Coin Index in Oxford has continued to develop into the unparalleled research tool for Iron Age studies that it represents today. The British exports in question are four Flat-Linear potin coins found in a mid-first-century BC context in ongoing excavations at the hilltop oppidum of Corent, in the Auvergne region of central France, over 600km from their home territory in southeast England (figure 14.1). Coins belonging to this series have been previously recorded from northern France, where there have also been a number of new finds in recent years, but never south of the Loire. We will begin by describing these new discoveries in more detail, starting with Corent, before going on to assess their implications for our understanding of the late Iron Age in southeastern England, which are considerable. In conclusion, we will offer some possible explanations as to why these coins may have been exported to France in the first century BC.
Receptor-operated Ca2+ channels in gastric parietal cells: gastrin and carbachol induce Ca2+ influx in depleting intracellular Ca2+ stores