Eczema

2018 ◽  
Author(s):  
Ben Esdaile
Keyword(s):  
Allergic Rhinitis ◽  
Atopic Dermatitis ◽  
Atopic Eczema ◽  
Inflammatory Disease ◽  
Chronic Inflammatory Disease ◽  
Descriptive Term ◽  
Symptoms And Signs

Eczema is a descriptive term for a set of symptoms and signs which usually involve itchy inflammation of the skin, and is subcategorized into many different forms. Atopic eczema (atopic dermatitis) is the most common chronic inflammatory disease amongst children. Other types of eczema include seborrhoeic eczema, discoid eczema, asteatotic eczema, contact allergic eczema, contact irritant eczema, pompholyx eczema, venous eczema, juvenile plantar dermatosis, and lichen simplex. This chapter covers atopic eczema, which is referred to as ‘atopic’ due to its association with other atopic disorders such as asthma and allergic rhinitis.

scholarly journals The Results of the Examination of Patients with eczema in Greece

2019 ◽  
Vol 4 (4) ◽  
Keyword(s):  
Innate Immunity ◽  
Allergic Rhinitis ◽  
Atopic Dermatitis ◽  
Environmental Factors ◽  
Natural History ◽  
Atopic Eczema ◽  
Skin Disease ◽  
Genetic Defects ◽  
Clinical Criteria ◽  
Recurrent Inflammation

Atopic dermatitis or atopic eczema is the most frequent skin disease of childhood. It is characterized by a chronic recurrent inflammation of the skin accompanied by disturbing itching. The etiology is complex including the genetic defects of the epidermic barrier and of the innate immunity as well as environmental factors. Diagnosis is based on clinical criteria and laboratory investigation is supportive of the atopic predisposition being not always relative to the disease severity and natural history. Eczema is usually the first manifestation of the atopic symptoms starting the so called ‘atopic march’, being followed by allergic rhinitis and asthma.

FEATURES OF RESPIRATORY PATHOLOGY IN CHILDREN LIVING IN THE CONDITIONS OF AEROGENIC EXPOSURE TO ACROLEIN AND FORMALDEHYDE

2018 ◽  
pp. 27-30
Author(s):  
О.А. Maklakova ◽  
I.P. Korotaeva ◽  
O.Yu. Ustinova
Keyword(s):  
Allergic Rhinitis ◽  
Bronchial Asthma ◽  
Inflammatory Disease ◽  
Respiratory Diseases ◽  
Chronic Inflammatory Disease ◽  
Blood Levels ◽  
Nasal Breathing ◽  
Second Child ◽  
Low Level ◽  
Respiratory Pathology

A survey of 158 children aged 4-7 years living in a chronic aerogenic low-level exposition to acrolein and formaldehyde was carried out. 82.3 % of children with high blood levels of acrolein and formaldehyde are diagnosed with respiratory diseases. Every second child has a chronic inflammatory disease of the nasal mucosa, including 29.1 % of allergic rhinitis, which are manifested by complaints of nasal breathing, not related to seasonality, nasal eosinophilia and the presence of nasal obstruction. In 6.3 % of children living in a chronic low-level exposition to acrolein and formaldehyde, the presence of bronchial asthma, manifested by signs of peripheral obstruction, was revealed.

Atopic dermatitis: new horizons of therapy

2019 ◽  
Vol 1 (7) ◽  
pp. 29-32 ◽  
Author(s):  
L. S. Kruglova ◽  
E. M. Gensler
Keyword(s):  
Blood Pressure ◽  
Immune Response ◽  
Allergic Rhinitis ◽  
Atopic Dermatitis ◽  
Targeted Therapy ◽  
Inflammatory Response ◽  
Bronchial Asthma ◽  
New Horizons ◽  
The Past

Over the past decades, the first breakthrough milestone in the treatment of severe forms of atopic dermatitis (AD) has been targeted therapy aimed at inhibiting IL-4 and IL-13. This was made possible thanks to advances in the understanding of the pathogenesis of AD, the driver of which is the Th2-type immune response, which also underlies such manifestations of atopy as bronchial asthma, allergic rhinitis, and polynosis. In the case of the Th2-type immune response, cytokines IL-4 and IL-13 are secreted, which are the main promoters of the inflammatory response in AD. Inhibition of IL-4 and IL-13 leads to the prevention of inflammation and is an effective approach to therapy. The use of therapy aimed at inhibition of cytokines allows you to effectively cope with the manifestations of severe and moderately severe blood pressure.

A Critical Review on Tamaka Shwasa (Bronchial Asthma) - An Ayurvedic View

2017 ◽  
Vol 2 (3) ◽  
Author(s):  
Sandip R. Baheti ◽  
Deepa Sharma ◽  
Saroj Devi ◽  
Amit Rai
Keyword(s):  
Bronchial Asthma ◽  
Inflammatory Disease ◽  
Critical Review ◽  
Modern Science ◽  
Modern Medicine ◽  
Chronic Inflammatory Disease ◽  
The Body ◽  
Natural Immunity ◽  
Shortness Of Breath ◽  
Asthma Symptoms

Difficulty in breathing or shortness of breath may be simply termed as Shwasa (Asthma), As per Ayurveda, Shwasa is mainly caused by the Vata and Kapha Doshas. Shwasa is broadly classified into five types in Maha Shwasa (Dyspnoea major), Urdhawa Shwasa (Expiratory Dyspnoea), Chinna Shwasa (Chyne-stroke respiration), Kshudra Shwasa (Dyspnoea minor), Tamaka Shwasa (Bronchial Asthma). In modern science Tamaka Shwasa can be correlated with Asthma, Asthma which is a chronic inflammatory disease of airway. In modern medicine there is no cure for Asthma, symptoms can typically be improved. In Ayurveda, Asthma can be effectively and safely manage the condition without inducing any drug dependency where Pachakarma procedures and use of internal medication detoxifies the body, provides nutrition and increases the elasticity of lung tissue it also develops natural immunity of the body thus decreasing episodic recurrence of the disease.

Intermammary Hidradenitis Suppurativa: Considerations on a Unique Presentation

2021 ◽  
pp. 1-4
Author(s):  
Ana Luísa João ◽  
Nélia Cunha ◽  
Joana Cabete
Keyword(s):  
Surgical Approach ◽  
Inflammatory Disease ◽  
Pilonidal Sinus ◽  
Hidradenitis Suppurativa ◽  
Chronic Inflammatory Disease ◽  
Pilonidal Sinus Disease ◽  
Sinus Disease ◽  
Case Presentation ◽  
Overweight Women

Introduction: Hidradenitis suppurativa (HS) is a potentially disabling chronic inflammatory disease. The intermammary location, not clearly specified in the usual phenotypic classifications, entails significant functional and cosmetic compromise. Case Presentation: Eleven cases of predominantly intermammary disease with multiple fistulization and disfiguring scarring were observed at our Department between January 2016 and March 2020. They were young (mean age = 22 years) obese or overweight women, in which the medical-surgical approach has led to variable results. Discussion/Conclusion: Intermammary HS possibly represents a unique phenotypic presentation, likely to be fistulizing and severe. We believe that anatomo-functional considerations, which can be paralleled to pilonidal sinus disease, contribute to its pathophysiology and striking appearance, and further emphasize the importance of multidisciplinary endeavor to reduce the morbidity associated with this seemingly overlooked location.

Efficacy and Safety of Crisaborole in Patients With Mild-to-Moderate Atopic Dermatitis With Comorbid Allergic Rhinitis or Asthma

2021 ◽  
Vol 147 (2) ◽  
pp. AB28
Author(s):  
Jonathan Spergel ◽  
Michael Blaiss ◽  
Peter Lio ◽  
Aharon Kessel ◽  
Liza Takiya ◽  
...  
Keyword(s):  
Allergic Rhinitis ◽  
Atopic Dermatitis ◽  
Efficacy And Safety

scholarly journals The Implications of Pruritogens in the Pathogenesis of Atopic Dermatitis

2021 ◽  
Vol 22 (13) ◽  
pp. 7227
Author(s):  
Lai-San Wong ◽  
Yu-Ta Yen ◽  
Chih-Hung Lee
Keyword(s):  
Atopic Dermatitis ◽  
Environmental Factors ◽  
Immune Responses ◽  
Immune Cells ◽  
Inflammatory Disease ◽  
Targeted Therapies ◽  
Skin Barrier ◽  
Skin Barrier Function ◽  
Inflammatory Molecules

Atopic dermatitis (AD) is a prototypic inflammatory disease that presents with intense itching. The pathophysiology of AD is multifactorial, involving environmental factors, genetic susceptibility, skin barrier function, and immune responses. A recent understanding of pruritus transmission provides more information about the role of pruritogens in the pathogenesis of AD. There is evidence that pruritogens are not only responsible for eliciting pruritus, but also interact with immune cells and act as inflammatory mediators, which exacerbate the severity of AD. In this review, we discuss the interaction between pruritogens and inflammatory molecules and summarize the targeted therapies for AD.

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