scholarly journals THE EFFECT OF FREQUENT CONSUMPTION OF ENERGY DRINKS ON BLOOD PRESSURE, HEART RATE AND BLOOD GLUCOSE IN HEALTHY ADULTS WORKING OUT IN THE GYMNASIUM

2021 ◽  
Vol 39 (Supplement 1) ◽  
pp. e139
Author(s):  
Abhinav Verma ◽  
Narsingh Vemra ◽  
Mohd Ehsan Siddiqui
2019 ◽  
Vol 2019 ◽  
pp. 1-9 ◽  
Author(s):  
Dariusz Nowak ◽  
Michał Gośliński ◽  
Anna Wesołowska ◽  
Karolina Berenda ◽  
Cezary Popławski

The purpose of this study has been to determine the effect of acute consumption of noni and chokeberry juices vs. energy drinks on blood pressure, heart rate, and blood glucose. The subjects divided into 4 groups, which consumed three portions of noni or chokeberry juices (30 mL or 200 mL, respectively) and energy drink (ED) or water (200 mL) at one-hour intervals. All participants had their blood pressure (BP), both systolic and diastolic BP (SBP and DBP), as well as heart rate (HR) and blood glucose (BG), measured. Consumption of noni juice caused a significant decrease in SBP and DBP of 5.0% and 7.5%, respectively, while, the consumption of chokeberry juice slightly decreased only DBP by 3.6%. On the contrary, consumption of three portions of EDs caused a significant increase in DBP by 14.7%. The BG of participants consuming noni juice decreased by 7.3%, while the consumption of EDs increased BG by as much as 15.8%. Acute consumption of noni juice contributed to a significantly decreased SBP, DBP, and HR as well as a mild reduction of BG. Consumption of chokeberry juice caused only a slight reduction of DBP. Contrary to juices, EDs consumption resulted in an increase of blood pressure (especially DBP) and blood glucose. The results of the study showed that noni juice may be effective in lowering blood pressure and blood sugar levels, but there is a need to continue research on the long-term effect of this juice.


Circulation ◽  
2014 ◽  
Vol 130 (suppl_2) ◽  
Author(s):  
Anna Svatikova ◽  
Naima Covassin ◽  
Krishen Somers ◽  
Filip Soucek ◽  
Tomas Kara ◽  
...  

Introduction: An increasing number of healthy adults consume energy drinks to enhance their physical and mental performance. Energy drinks contain caffeine and multiple other “natural” stimulants and their combined effects on cardiac hemodynamics in healthy individuals are unclear. Hypothesis: We hypothesized that drinking a commercially available energy drink, Rockstar , compared to a placebo drink, increases resting blood pressure and heart rate in healthy adults, and these increases would be further accentuated by stress conditions. Methods: We enrolled 25 healthy, normotensive subjects (14 males), aged 29±1 years, with body mass index 24.5±1 kg/m 2 , in a randomized, double-blind, placebo-controlled, crossover study. Each subject consumed a placebo drink and a commercially available Rockstar energy drink (473 ml), in random order on two separate study days. Blood pressure and heart rate responses were recorded and compared before and 30 minutes after drink consumption, both at rest and then in response to physical, mental and cold stressors. Results: The Rockstar energy drink induced a 6±1% increase in resting systolic blood pressure, compared to 3±1% with the placebo drink (P=0.0078). Diastolic blood pressure increased by 7±1% vs 0±1% with the placebo drink (P=0.0007; Figure 1). Heart rate increased similarly in both groups, by 5±2% in the energy drink group vs. 7±2% in the placebo group. Blood pressure and heart rate increases during the stress stimuli (sustained handgrip, mental stress and cold stress) did not differ between energy drink and placebo. Conclusions: Drinking a commercially available Rockstar energy drink significantly increases resting blood pressure in young healthy adults. Blood pressure increases are not further accentuated by exercise, mental or cold pressor stress stimuli. These hemodynamic changes induced by energy drink consumption could predispose to cardiovascular events.


2019 ◽  
Vol 3 (Supplement_1) ◽  
Author(s):  
Bailey Sapa ◽  
Ted Wilson ◽  
James Gronseth ◽  
Francis Ragsdale

Abstract Objectives Energy drinks have been anecdotally associated with deleterious cardiovascular health outcomes. These drinks contain ingredients which could affect glycemic response to the carbohydrate content. The aim of this study was to determine if acute exercise would alter the glycemic and physiological response to the consumption of Monster Energy. Methods Following an overnight fast, human subjects (19.1 ± 1.4 years; 30 female; 5 male) were randomized to exercise on a stationary bike at 33% of their predicted VO2 max for 10 minutes with a five minute sitting rest after, or sitting (control) for 15 minutes prior to ingestion of Monster Energy. Blood glucose, heart rate, and blood pressure were measured 0-, 30-, 60-, and 90-minutes postprandially. Data is represented as LSM ± SE with significance analyzed using a t-test, or multiple comparison test (P < 0.05). Results Blood glucose (mg/dL) for control at 0-, 30-, 60-, and 90-minutes 91.5 ± 1.2, 133.7 ± 4.3, 96.3 ± 4.2, and 82.4 ± 1.8 mg/dL, respectively. For those receiving bike exercise prior to ingestion blood glucose was 92.2 ± 1.2, 126.6 ± 3.8, 92.5 ± 2.3, 84.0 ± 1.8 mg/dL. Within group significance in both groups was observed between 0 and 30, 30 and 60, and 60 and 90, for control only, there were no significant blood glucose differences observed between groups across time. Heart rate for control at 0-, 30-, 60-, and 90-minutes was 78.0 ± 3.1, 78.2 ± 2.5, 78.6 ± 2.6, and 74.4 ± 1.9 respectively. For those receiving bike exercise prior to ingestion heart rate was 88.7 ± 3.71, 85.5 ± 2.7, 83.1 ± 2.3, and 80.3 ± 2.9, with significance between 0 and 90 minutes. Mean arterial blood pressure for control at 0-, 30-, 60-, and 90-minutes was 86.8 ± 2.6, 89.2 ± 2.1, 89.2 ± 2.5, and 87.6 ± 2.6. For those receiving bike exercise prior to ingestion blood pressure was 90.6 ± 3.1, 88.98 ± 2.3, 88.2 ± 2.0, and 87.0 ± 2.9 with no significant differences. Conclusions Exercise probably does not alter the glycemic and physiological response to energy drinks. Funding Sources WSU Biology Department.


2008 ◽  
Vol 13 (4) ◽  
pp. 211-217 ◽  
Author(s):  
Tan Xue-rui ◽  
Lv Ying ◽  
Yang Da-zhong ◽  
Chen Xiao-jun

Author(s):  
Toshiki Kutsuna ◽  
Hitoshi Sugawara ◽  
Hideaki Kurita ◽  
Satomi Kusaka ◽  
Tetsuya Takahashi

Background: Low-intensity resistance training (RT) combined with neuromuscular electrical stimulation (NMES) is one method of exercise to improve the deterioration of physical function. However, it is unclear whether low-intensity RT combined with NMES (RT + NMES) can be safely implemented. Objective: This study aimed to examine the influence of low-intensity RT + NMES on autonomic activity and cardiovascular responses in healthy adults. Methods: This study was an open-label, randomized controlled cross-over trial. The exercise intensity of isometric knee extension RT was set to 40% of the maximum voluntary contraction (peak torque). NMES was adjusted to a biphasic asymmetrical waveform with the frequency maintained at 50 Hz and a phase duration of 300 [Formula: see text]s. The difference in the change in autonomic activity and cardiovascular responses was compared by assessing heart rate variability, blood pressure, and heart rate during RT and [Formula: see text]. Results: Twenty healthy male college students (mean age [Formula: see text] years) participated in this study. The ratio of low- and high-frequency components of heart rate variability, systolic blood pressure, and heart rate increased during exercise in the RT and [Formula: see text] sessions ([Formula: see text]). There were no significant differences in autonomic activity and cardiovascular responses throughout the sessions during RT and [Formula: see text]. Conclusion: In conclusion, our results demonstrated that low-intensity [Formula: see text] was safe and did not induce excessive autonomic and cardiovascular responses in healthy adults.


2021 ◽  
Vol 17 (2) ◽  
pp. 72-78
Author(s):  
M.V. Boliuk ◽  
O.A. Halushko

Background. Due to the frequent development of neuropathy in diabetic patients, it is believed that this category of patients is characterized by a high incidence of atypical acute coronary syndrome, but data about this are quite contradictory. The purpose of the study was to determine pain syndrome features and its severity in patients with acute coronary syndrome and diabetes mellitus. Materials and methods. The study involved 24 patients with diabetes (19 men and 5 women) aged 45–83 years, hospitalized urgently for the acute coronary syndrome. Assessment of pain syndrome was performed at the time of hospitalization and immediately after coronary artery revascularization according to the following criteria: visual analogue scale (VAS), numerical rating scale (NRS), clinical data (sweating, tremor, blood pressure, pulse), blood glucose level. Results. Most patients (87.5 %) at the time of hospitalization complained of chest pain, the rest were not bothered by any pain. Patients described pain as “burning” (29.17 %), “squeezing” (29.17 %), “tightness” (25.0 %), “tingling” (4.17 %). There were also complaints of difficulty breathing (12.5 %), shortness of breath (12.5 %), palpitations (41.67 %), excessive sweating (16.67 %). There was no statistically significant difference between the results of pain assessment by VAS and NRS (p > 0.1). The results of the subjective assessment of pain syndrome by VAS and NRS indicate that before revascularization, moderate and severe pain occurred with equal frequency. There were no statistically significant fluctuations in blood pressure and heart rate before and after the intervention in patients with different pain severity (p > 0.1). At the time of hospitalization, the mean systolic blood pressure was 135.71 ± 18.70 mmHg, diastolic blood pressure was 83.71 ± 14.67 mmHg, heart rate was 73.08 ± 11.35 bpm. The mean value of glycemia at the time of hospitalization was 8.19 ± 3.45 mmol/l (8.17 ± 3.61 mmol/l in men, 8.28 ± 3.13 mmol/l in women). Blood glucose level ≥ 10.0 mmol/l was detected in 5 patients, i.e. in 20.83 % of all patients. The majority of these individuals had severe pain (60.0 %). Conclusions. In patients with myocardial infarction and diabetes mellitus, the typical clinical picture of ACS (87.5 %) prevailed over the painless form. Before revascularization, moderate and severe pain occurred with equal frequency; there is no statistical difference between blood pressure, heart rate and blood glucose level (p > 0.1) in patients with severe and moderate pain. Hyperglycemia (≥ 10.0 mmol/l) was detected in 20.83 % of patients, most of them had severe pain (60.0 %). The lack of difference between the values of the studied pain criteria may be due to the sample size, the low sensitivity of the criteria, the development of diabetic neuropathy. As a result, there is a need for further study of the phenomenon of pain syndrome in patients with ACS and diabetes mellitus.


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