scholarly journals Adverse Cardiovascular Effects of Traffic Noise with a Focus on Nighttime Noise and the New WHO Noise Guidelines

2020 ◽  
Vol 41 (1) ◽  
pp. 309-328 ◽  
Author(s):  
Thomas Münzel ◽  
Swenja Kröller-Schön ◽  
Matthias Oelze ◽  
Tommaso Gori ◽  
Frank P. Schmidt ◽  
...  

Exposure to traffic noise is associated with stress and sleep disturbances. The World Health Organization (WHO) recently concluded that road traffic noise increases the risk for ischemic heart disease and potentially other cardiometabolic diseases, including stroke, obesity, and diabetes. The WHO report focused on whole-day noise exposure, but new epidemiological and translational field noise studies indicate that nighttime noise, in particular,is an important risk factor for cardiovascular disease (CVD) through increased levels of stress hormones and vascular oxidative stress, leading to endothelial dysfunction and subsequent development of various CVDs. Novel experimental studies found noise to be associated with oxidative stress–induced vascular and brain damage, mediated by activation of the NADPH oxidase, uncoupling of endothelial and neuronal nitric oxide synthase, and vascular/brain infiltration with inflammatory cells. Noise-induced pathophysiology was more pronounced in response to nighttime as compared with daytime noise. This review focuses on the consequences of nighttime noise.

Author(s):  
Angel M. Dzhambov ◽  
Peter Lercher

Unlike other World Health Organization evidence reviews, the systematic review on mental disorders could not provide a quantitative estimate of the effect of environmental noise. With that in mind, we aimed to update it with additional studies published through to 18 August 2019 in order to allow for a formal meta-analysis of the association of residential road traffic noise with anxiety and depression. The quality effects and random effects estimators were used for meta-analysis and the robustness of findings was tested in several sensitivity analyses. Ten studies were included in the qualitative synthesis, from which we extracted 15 estimates for depression (n = 1,201,168) and five for anxiety (n = 372,079). Almost all studies were cross-sectional and the risk of bias in them was generally high. We found 4% (95% CI: −3%, 11%) higher odds of depression and 12% (95% CI: −4%, 30%) of anxiety associated with a 10 dB(A) increase in day–evening–night noise level (Lden). Both models suffered from moderate heterogeneity (55% and 54%), but there was evidence of publication bias only in the depression model. These findings were robust with no evidence of study-level moderators. A sensitivity analysis on an alternative set of categorically-reported estimates supported a linear relationship between Lden and depression. Taking into account an overall quality assessment for the included studies, we conclude that there is evidence of “very low” quality that increasing exposure to road traffic noise may be associated with depression and anxiety.


Acoustics ◽  
2020 ◽  
Vol 2 (1) ◽  
pp. 73-86
Author(s):  
Philippe Apparicio ◽  
Jérémy Gelb

According to the World Health Organization, high levels of exposure to road traffic noise are associated with adverse health effects. Earlier studies suggest that cyclists are exposed to higher noise levels than motorists. Other studies have demonstrated that cyclists’ exposure to noise could vary significantly according to their routes. The aim of this study is to compare cyclists’ exposure to noise and their determinants in three cities. Three participants cycled equipped with noise dosimeters and GPS watches: 1823, 967, and 1362 km in Copenhagen, Paris, and Montreal, respectively. We fitted three generalized additive mixed model with an autoregressive term models to predict the cyclists’ exposure to noise according to the type of route and bicycle infrastructure after controlling for the day of the week, as well as spatial and temporal trends. The overall noise means were 73.4, 70.7, and 68.4 dB(A) in Paris, Montreal, and Copenhagen, respectively. The exposure to road traffic noise is strongly associated with the type of bicycle infrastructure taken by the cyclist; riding on a cycleway significantly decreases it, while riding in a shared lane has no impact. Our findings demonstrate that it is possible to achieve a substantial reduction in cyclists′ exposure by adopting new practices that include noise exposure in the planning of future cycling infrastructure.


2021 ◽  
Vol 263 (4) ◽  
pp. 2550-2554
Author(s):  
Timothy Van Renterghem ◽  
Pieter Thomas ◽  
Dick Botteldooren

Excessive road traffic noise exposure in (sub)urban parks hinders its restorative function and will negatively impact the number of visitors. Especially in such green environments, noise abatements by natural means, well integrated in the landscape, are the most desired solutions. Although dense vegetation bordering the park or raised berms could come first in mind, local landscape depressions are typically underused. In this work, a case-study of a small suburban park, squeezed in between two major arterial roads, is analyzed. The spatially dependent road traffic noise exposure in the park is assessed in detail by mobile sound pressure level measurements. Local reductions of up to 6-7 dBA are found at landscape depressions of only a few meters deep. It can therefore be concluded that this is an efficient measure and should be added to the environmental noise control toolbox for noise polluted parks.


2021 ◽  
Vol 263 (3) ◽  
pp. 3282-3293
Author(s):  
Jens Forssén ◽  
Andreas Gustafson ◽  
Meta Berghauser Pont ◽  
Marie Haeger-Eugensson ◽  
Christine Achberger ◽  
...  

For a model set of 31 different building morphologies in an urban setting, road traffic noise exposure has been calculated and analysed. For five of the building morphologies also vegetation surfaces on facades and roofs were studied. Facade exposures were analysed for both smaller (single-sided) flats and larger (floor-through) flats, considering the direct exposure from the roads as well as the non-direct exposure at noise-shielded positions like inner yards, applying a noise mapping software in combination with a prediction model for the non-direct exposure. Using noise indicators Lden and Lnight, the disease burden, in terms of DALY (Disability-Adjusted Life Years) per person, was estimated and analysed, via predictions of annoyance and sleep disturbance. The resulting effects of varying the building morphology and adding vegetation are shown and discussed, including effects of a bonus model for flats having additional facade elements with lower noise exposure.


2021 ◽  
pp. 2004594
Author(s):  
Shuo Liu ◽  
Youn-Hee Lim ◽  
Marie Pedersen ◽  
Jeanette T. Jørgensen ◽  
Heresh Amini ◽  
...  

BackgroundWhile air pollution has been linked to the development of chronic obstructive pulmonary disease (COPD), evidence on the role of environmental noise is just emerging. We examined the associations of long-term exposure to air pollution and road traffic noise with COPD incidence.MethodsWe defined COPD incidence for 24 538 female nurses from the Danish Nurse Cohort (age>44 years) as the first hospital contact between baseline (1993 or 1999) and 2015. We estimated residential annual mean concentrations of particulate matter with diameter<2.5 µm (PM2.5) since 1990 and nitrogen dioxide (NO2) since 1970 by the Danish DEHM/UBM/AirGIS modeling system, and road traffic noise (Lden) since 1970 by the Nord2000 model. Time-varying Cox regression models were applied to assess the associations of air pollution and road traffic noise with COPD incidence.Results977 nurses developed COPD during 18.6 years’ mean follow-up. We observed associations with COPD for all three exposures with hazard ratios (HRs) and 95% confidence intervals (CIs) of 1.19 (1.01, 1.41) per 6.26 µg·m−3 for PM2.5, 1.13 (1.05, 1.20) per 8.19 µg·m−3 for NO2, and 1.15 (1.06, 1.25) per 10 dB for Lden. Associations with NO2 and Lden attenuated slightly after mutual adjustment, but were robust to adjustment for PM2.5. Associations with PM2.5 were attenuated to null after adjustment for either NO2 or Lden. No potential interaction effect was observed between air pollutants and noise.ConclusionsLong-term exposure to air pollution, especially traffic-related NO2, and road traffic noise were independently associated with COPD.


2015 ◽  
Vol 66 (1) ◽  
pp. 15-21 ◽  
Author(s):  
Angel M. Dzhambov ◽  
Donka D. Dimitrova

Abstract Road traffic noise is a widely studied environmental risk factor for ischaemic heart disease and myocardial infarction in particular. Given that myocardial infarction is a leading disability and mortality cause in Bulgaria and that a significant proportion of the urban population is exposed to high noise levels, quantification of the burden of disease attributable to traffic noise is essential for environmental health policy making and noise control engineering. This study aimed at estimating the burden of the myocardial infarction cases attributable to road traffic noise in the Bulgarian urban population. We used the methodology for estimating the burden of disease attributable to environmental noise outlined by the World Health Organization. Risk data were extracted from a recently published meta-analysis providing updated exposureresponse relationship between traffic noise and the risk for myocardial infarction. Based on these data we calculated the fraction of myocardial infarction cases attributable to traffic noise, loss of quality-adjusted life-years (QALYs), and the economic burden, assuming € 12,000 per QALY. About 2.9 % or 101 of all myocardial infarction cases could be attributed to road traffic noise. Fifty-five of these were fatal. Nine hundred and sixty-eight QALYs were lost to these cases. The monetary value of these QALYs was about € 11.6 million. Although the measures used in this study are crude and give only an approximation of the real burden of disease from road traffic noise, they are indicative of the important social and economic aspect of noise pollution in Bulgaria. Hopefully, these results will direct the attention of epidemiologists, environmental hygienists, and health economists to this pivotal environmental issue.


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