Suppression of Exercise-Induced Ventricular Arrhythmias by Propranolol and Diltiazem in a Woman Without Organic Heart Disease—A Case Report

Angiology ◽  
1990 ◽  
Vol 41 (1) ◽  
pp. 70-75
Author(s):  
Katsusuke Yano ◽  
Katsumi Miyauchi ◽  
Takao Mitsuoka ◽  
Kunitake Hashiba
2020 ◽  
Vol 21 ◽  
Author(s):  
Michelle Audrey Darmadi ◽  
Axel Duval ◽  
Hanaa Khadraoui ◽  
Alberto N. Romero ◽  
Blanca Simon ◽  
...  

Author(s):  
Eduard Guasch ◽  
Lluís Mont

The identification of ventricular arrhythmias in athletes should always be carefully considered because of the increased risk of SCD. The main factor determining this risk is the presence of an underlying structural or primary arrhythmic heart disease. Consequently, the identification of ventricular arrhythmias in an athlete should prompt exploratory testing, most commonly involving 24-hour Holter ECG, echocardiography, and an exercise test. Other tests should be considered on an individual basis. In general, the more frequent and complex the ventricular arrhythmias, the higher are the risks of a heart disease, and a more thorough examination is required. While most arrhythmias in athletes without heart disease are idiopathic ventricular arrhythmias, considerable attention has been paid recently to an exercise-induced substrate for ventricular arrhythmias, similar to classic forms of arrhythmogenic ventricular cardiomyopathy. Overall, the identification of a cardiac condition will determine the prognosis, therapeutic approach, and eligibility for competitive sport.


2020 ◽  
Author(s):  
Ying Liang ◽  
Yulong Guan

Abstract Background:Malignant ventricular arrhythmias (MVA) occurring subsequent to a repair of uncomplicated congenital heart disease is scarcely described in literature.Case presentation:One adult patient following congenital atrial septal defect (ASD) repair underwent immediate postoperative refractory MAV and ventricular fibrillation. The recurrent episodes of shocks cannot be suppressed by drugs. Emergent re-exploration was performed and repeated closure of ASD and DeVega's annuloplasty were completed. The patient had uneventful recovery and no occurrence of arrhythmia.Conclusion: Malignant ventricular arrhythmias are rare and should never be overemphasized even during the repair of uncomplicated congenital heart defect. Re-exploration should be taken into consideration when MVA occurred in the early stage postoperatively.


2003 ◽  
Vol 67 (2) ◽  
pp. 125-128 ◽  
Author(s):  
Erdulfo J. Galeano ◽  
Akihiro Yoshida ◽  
Yoshio Ohnishi ◽  
Katsunori Okajima ◽  
Akihiko Ishida ◽  
...  

2020 ◽  
Vol 21 (1) ◽  
Author(s):  
Begona Sanchez-Lechuga ◽  
Muhammad Saqlain ◽  
Nicholas Ng ◽  
Kevin Colclough ◽  
Conor Woods ◽  
...  

2019 ◽  
Vol 8 (2) ◽  
pp. 83-89 ◽  
Author(s):  
Jeffrey J Hsu ◽  
Ali Nsair ◽  
Jamil A Aboulhosn ◽  
Tamara B Horwich ◽  
Ravi H Dave ◽  
...  

Ventricular arrhythmias are challenging to manage in athletes with concern for an elevated risk of sudden cardiac death (SCD) during sports competition. Monomorphic ventricular arrhythmias (MMVA), while often benign in athletes with a structurally normal heart, are also associated with a unique subset of idiopathic and malignant substrates that must be clearly defined. A comprehensive evaluation for structural and/or electrical heart disease is required in order to exclude cardiac conditions that increase risk of SCD with exercise, such as hypertrophic cardiomyopathy and arrhythmogenic right ventricular cardiomyopathy. Unique issues for physicians who manage this population include navigating athletes through the decision of whether they can safely continue their chosen sport. In the absence of structural heart disease, therapies such as radiofrequency catheter ablation are very effective for certain arrhythmias and may allow for return to competitive sports participation. In this comprehensive review, we summarise the recommendations for evaluating and managing athletes with MMVA.


2020 ◽  
Vol 4 (2) ◽  
pp. 244-246
Author(s):  
Orhay Mirzapolos ◽  
Perry Marshall ◽  
April Brill

Introduction: Brugada syndrome is an arrhythmogenic disorder that is a known cause of sudden cardiac death. It is characterized by a pattern of ST segment elevation in the precordial leads on an electrocardiogram (EKG) due to a sodium channelopathy. Case Report: This case report highlights the case of a five-year-old female who presented to the emergency department with a febrile viral illness and had an EKG consistent with Brugada syndrome. Discussion: Fever is known to accentuate or unmask EKG changes associated with Brugada due to temperature sensitivity of the sodium channels. Conclusion: Febrile patients with Brugada are at particular risk for fatal ventricular arrhythmias and fevers should be treated aggressively by the emergency medicine provider. Emergency medicine providers should also consider admitting febrile patients with Brugada syndrome who do not have an automatic implantable cardioverter-defibrillator for cardiac monitoring.


Lung ◽  
2014 ◽  
Vol 192 (2) ◽  
pp. 329-331 ◽  
Author(s):  
Amit Diwakar ◽  
Gregory A. Schmidt

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