scholarly journals Tai-chi-chuan and yoga onpostexercise hypotension: comparison to aerobic and resistance exercise

2016 ◽  
Vol 29 (3) ◽  
pp. 543-552
Author(s):  
João Douglas Alves ◽  
Jorge Luiz de Brito Gomes ◽  
Caio Victor Coutinho de Oliveira ◽  
José Victor de Miranda Henriques Alves ◽  
Fabiana Ranielle de Siqueira Nogueira ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Young Women ◽  
Tai Chi ◽  
Cardiovascular Effects ◽  
Cardiovascular Responses ◽  
Tai Chi Chuan ◽  
Post Exercise ◽  
Significant Difference ◽  
Apparently Healthy

Abstract Introduction: Tai-Chi-Chuan and Yoga have becoming popular practices. However is unclear the cardiovascular effects, and if they present similar behavior to aerobic and resistance sessions. Objective: To evaluate the cardiovascular responses during the session and post-exercise hypotension (PEH) of Tai Chi Chuan (TS) and Yoga (YS) in comparison to aerobic (AS) and resistance (SR) exercises. Methods: Fourteen young women (22.3 ± 2 years) apparently healthy performed four sessions (AS, RS, TS and YS). The heart rate (HR), systolic (SBP) and diastolic blood pressure (DBP) were recorded at resting, during (every 10 minutes) and until 50 minutes of recovery. Results: AS, RS, TS e YS showed significant increase in HR compared to resting.AS at 10, 30 e 50 minutes in relation to RS, TS e YS. The RS in relation to TS and YS at 10, 30 and 50 minutes. No significant difference between TS and YS. SBP was significantly increased in AS, RS, TS e YS at 10, 30 e 50 minutes during the session, in relation to rest. AS was significantly higher at 30 e 50 minutes than RS and higher than TS and YS at 10, 30 e 50 minutes. No significant difference in DBP. For PEH, AS, RS and TS significantly reduced at 10, 30 and 50 minutes. YS reduced at 50 minutes. No significant diastolic PEH. Conclusion: TS and YS showed as safe alternatives of exercising in the normotensive young adult woman, despite having lower values, they promote similar hemodynamic behavior to AS and RS.

scholarly journals Cardiovascular Responses to Repetitive McKenzie Lumbar Spine Exercises

2001 ◽  
Vol 81 (9) ◽  
pp. 1524-1533 ◽  
Author(s):  
Saud Al-Obaidi ◽  
Joseph Anthony ◽  
Elizabeth Dean ◽  
Nadia Al-Shuwai
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Low Back Pain ◽  
Back Pain ◽  
Lumbar Spine ◽  
Cardiovascular Effects ◽  
Cardiovascular Responses ◽  
Low Back ◽  
Pulmonary Risk ◽  
Flexion And Extension

Abstract Background and Purpose. Repetitive exercises of the type recommended by McKenzie for the lumbar spine, such as flexion and extension exercises in standing (FIS and EIS) and lying positions (FIL and EIL), have been used in the management of low back pain for over 20 years. The cardiovascular effects of exercises that involve postural stabilization and the arms and of exercises performed in a lying position are well known. Therefore, the purpose of this study was to examine the cardiovascular effects of 4 exercises used in the McKenzie system. Subjects and Methods. One hundred subjects without cardiovascular or cardiopulmonary disease (mean age=31 years, SD=6.1, range=22–44) and who were representative of people susceptible to low back pain were studied. Subjects were randomly assigned to 1 of 4 exercise groups (ie, FIS, EIS, FIL, and EIL). Subjects performed sets of 10, 15, and 20 repetitions of the assigned exercise, with a 15-minute rest between sets. Heart rate, blood pressure, and rate-pressure product (an index of myocardial work) were recorded before and after each set of repetitions. Results. After 10 repetitions, flexion and extension in lying were more hemodynamically demanding than in standing. This trend persisted for 15 and 20 repetitions; however, at 20 repetitions, the hemodynamic demands were different across exercise groups (ie, FIL>EIL>FIS>EIS). Discussion and Conclusion. Repetitive exercises of the type suggested by McKenzie for the lumbar spine can have cardiovascular effects in people with no cardiovascular or cardiopulmonary conditions. These effects may be important with respect to cardiac work, and patients for whom these exercises are indicated should have a cardiac and pulmonary risk factor assessment to determine whether heart rate and blood pressure should be monitored.

scholarly journals Acute cardiovascular responses to a session of Manual Lymphatic Drainage

2015 ◽  
Vol 28 (1) ◽  
pp. 41-48 ◽  
Author(s):  
Plínio dos Santos Ramos ◽  
Isabelle Regina Moreira Marinho Cunha ◽  
Mariana Cristina Rachel ◽  
Priscilla Stephan Souza Pacca ◽  
Ana Paula Ferreira ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Young Women ◽  
Interstitial Fluid ◽  
Cardiovascular Responses ◽  
Lymphatic Drainage ◽  
Point Of View ◽  
Hemodynamic Parameters ◽  
Manual Lymphatic Drainage ◽  
Hemodynamic Changes

Introduction Manual Lymphatic Drainage (MLD) creates pressure differentials to promote the movement of lymph and interstitial fluid, for their relocation into the bloodstream and can thus act on the variables responsible for determining blood pressure (BP). Objective To investigate the behavior of hemodynamic parameters, such as BP and Heart Rate (HR) during and immediately after a session of MLD. Materials and methods 23 healthy young volunteers with a mean age of 22 ± 2.97 years (18-29), underwent a session of MLD and evaluation of systolic BP, diastolic BP and HR after 10 minutes of initial rest, at the end of the implementation of the protocol in SP, PP and after 15 minutes of final rest. Results No changes were found in systolic BP or diastolic BP before, during and immediately after the MLD session (p = 0.57, p = 0.20, respectively). There was increased heart rate after the final 15 minute rest when compared to the HR after MLD in SP (72 ± 1.9 vs. 76 ± 1.9, p = 0.002), however, without clinical relevance. Conclusion During a session of MLD with healthy young women, no significant hemodynamic changes occurred, demonstrating that this technique is safe from the cardiovascular point of view.

Post-Exercise Reduction of Blood Pressure in Hypertensive Men is Not Due to Acute Impairment of Baroreflex Function

1984 ◽  
Vol 67 (1) ◽  
pp. 97-103 ◽  
Author(s):  
T. Bennett ◽  
R. G. Wilcox ◽  
I. A. MacDonald
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Atmospheric Pressure ◽  
Intermittent Exercise ◽  
Cardiovascular Responses ◽  
Lower Body ◽  
Post Exercise ◽  
Blood Pressures ◽  
Normotensive Subjects ◽  
Exercise Induced

1. Two experiments were carried out in male subjects. In the first experiment heart rate and blood pressures were measured before, during and for 90 min after a 50 min period of intermittent exercise in seven hypertensive subjects. After exercise there was a marked reduction in systolic and diastolic blood pressures; this effect lasted throughout the 90 min observation period and was unaccompanied by tachycardia. 2. In the second experiment heart rate and blood pressures were measured before and during the rest periods in a 50 min session of intermittent exercise in nine hypertensive and nine normotensive subjects. Following the first 10 min bout of exercise, resting blood pressures were significantly reduced in the hypertensive subjects; the reduction in blood pressure progressively increased following successive exercise periods. The normotensive subjects did not show a significant reduction in resting blood pressures until the fifth bout of exercise had been completed. 3. In the second experiment also, the competence of baroreflexes was assessed by measuring cardiovascular responses to lower body sub-atmospheric pressure (LBSP) 30 min before and 30 and 60 min after exercise. The post-exercise reduction in blood pressure was not due to exercise-induced impairment of baroreflex mechanisms since the reduced blood pressure after exercise was well maintained during lower body sub-atmospheric pressure. Furthermore, after exercise, exposure to lower body subatmospheric pressure elicited greater increases in heart rate and forearm vascular resistance than were seen before exercise.

scholarly journals A randomized controlled study of intravenous esmolol to attenuate the cardiovascular responses to laryngoscopy and endotracheal intubation

2020 ◽  
Vol 8 (3) ◽  
pp. 1103
Author(s):  
Ninad Deepak Chodankar ◽  
Bhagyashree Shivde
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
General Anaesthesia ◽  
Endotracheal Intubation ◽  
Elective Surgery ◽  
Cardiovascular Responses ◽  
Controlled Study ◽  
Control Group ◽  
Significant Difference ◽  
Control Group Group

Background: Objective is to compare the efficacy of intravenous Esmolol to attenuate the cardiovascular responses to laryngoscopy and endotracheal intubation with control group.Methods: Study was done on 60 adults, ASA grade I or II normotensive patients, undergoing elective surgery under general anaesthesia and willing to participate. These patients where be randomly allocated in to either group C (Control) or E (Esmolol). Group ‘C’ Control group. Group ‘E’, patients were given intravenous Esmolol 1.5 mg/kg 2 minutes before start of laryngoscopy. All patients were premedicated, induced and intubated using Thiopentone and Succinyl Choline as per the protocol. Heart Rate (HR), SBP, DBP and MAP were recorded at baseline (taken half an hour prior to anaesthesia), Before sedation, After induction but before intubation, Immediately after endotracheal intubation and Thereafter at 1, 2, 3, 4, 5 and 10 minutes.Results: Heart rate was lower in Group E as compared to Group C, and there was statistically significant difference immediately after intubation till 4 minutes after intubation. While Blood pressure was lower in Group E as compared to Group C, and there was statistically significant difference only immediately after intubation.Conclusions: In Normotensive patients requiring general anaesthesia with laryngoscopy and intubation, authors conclude that intravenous Esmolol 1.5 mg/kg attenuated Heart rate response but fails to satisfactorily prevent rise in blood pressure.

scholarly journals Cardiovascular Effects of Listening to Fast Speech and Normal Speech

2020 ◽  
pp. 1-4
Author(s):  
Perez Quartey ◽  
Blemano David TA ◽  
Odoi Patience
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Pulse Rate ◽  
Cardiovascular Response ◽  
Area Under The Curve ◽  
Cardiovascular Effects ◽  
Cardiovascular Responses ◽  
Limited Data ◽  
Audio Recordings ◽  
Normal Speech

Background: Some previous works on the psychological impact of speech on the cardiovascular system have mainly focused on the speaker as the individual in whom clinical outcomes are being measured. There is limited data on the effects of listening to the fast speech on cardiovascular responses. Aim: The aim of the study was to comparatively examine blood pressure and heart rate changes upon listening to normal and fast speeches. Method: A total of 88 (22 females and 66 males) normotensive adults were recruited for the study from a university population. All subjects were made to listen to two different 13-minutes audio recordings of normal speech (news commentary) and fast speech (a radio sports presentation). Blood pressure and pulse rate changes were taken at 4-minutes time intervals during listening to the audio recordings. Based on the enthusiasm and patronage of the sports program, participants were classified as ‘‘Regular’’ listeners and ‘‘Non-regular’’ listeners. Blood pressure and pulse rate changes were calculated as the mean net area under the curve response and differences were analysed with analysis of variance. Results: Systolic, diastolic and pulse rate responses were significantly higher in both the Regular and Non-Regular listener groups during listening to the fast-speech audio presentation as compared to the News Commentary presentation. Conclusion: Although there is limited data, listening to fast speech itself may act as a psychosocial stressor that predisposes to an increased cardiovascular response manifested as higher blood pressure and heart rate.

Cardiovascular responses to parathyroid hormone

1985 ◽  
Vol 249 (2) ◽  
pp. E187-E194 ◽  
Author(s):  
M. F. Crass ◽  
P. L. Moore ◽  
M. L. Strickland ◽  
P. K. Pang ◽  
M. S. Citak
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Parathyroid Hormone ◽  
Cardiovascular Effects ◽  
Cardiovascular Responses ◽  
Contractile Force ◽  
Mean Blood Pressure ◽  
Pharmacological Characterization ◽  
Amino Terminal ◽  
Dose Dependent Fashion

The synthetic amino terminal fragment of parathyroid hormone, PTH-(1-34), is a potent coronary artery vasodilator in the dog. In the present study, using instrumented open-chest dogs, we have performed the initial characterization of this effect and showed other dose-related cardiovascular effects of the hormone. A near-maximal flow response was obtained after intracoronary injection of 0.024 nmol X kg-1 PTH-(1-34) with minor, if any, concomitant changes in mean blood pressure, contractile force, or heart rate. At higher doses, mean blood pressure decreased while contractile force and heart rate increased in dose-dependent fashion. Infusion of PTH-(1-34) for 20 min showed that the vasodilatory effect could be sustained without a concomitant decrease in mean arterial pressure. Pharmacological characterization showed for the first time that the coronary response to PTH-(1-34) was unaltered in the presence of beta- or alpha-adrenergic, muscarinic, or histaminergic blockades. We conclude that PTH-(1-34), an endogenous circulating calcemic peptide, produces a large increase in coronary blood flow at doses sufficiently low to preclude complicating effects on blood pressure, contractile force, and heart rate. Furthermore, the results suggest that the vasodilatory effects may be specific.

scholarly journals Cardiovascular Responses to Tracheal Intubation: A Comparison of Direct Laryngoscopy and Fibreoptic Intubation

1989 ◽  
Vol 17 (1) ◽  
pp. 44-48 ◽  
Author(s):  
S. R. Finfer ◽  
S. I. P. MacKenzie ◽  
J. M. Saddler ◽  
T. G. L. Watkins
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Tracheal Intubation ◽  
Oxygen Saturation ◽  
Arterial Oxygen Saturation ◽  
Macintosh Laryngoscope ◽  
Cardiovascular Responses ◽  
Arterial Oxygen ◽  
Arterial Blood ◽  
Significant Difference

The cardiovascular responses to tracheal intubation using a fibreoptic bronchoscope or Macintosh laryngoscope were compared in twenty in-patients and twenty day-stay patients. Within these groups patients were randomly allocated to direct laryngoscopic or fibreoptic bronchoscopic intubation. Arterial blood pressure, heart rate and arterial oxygen saturation were recorded before induction and at one-minute intervals until four minutes after intubation. In both groups both laryngoscopic and bronchoscopic intubation resulted in a significant rise in blood pressure and heart rate. At no stage was there a significant difference in mean blood pressure in either group, or in heart rate in the day-stay patients, between the different methods of intubation. In the in-patients mean heart rate was significantly higher in those patients intubated with the bronchoscope at three and four minutes after intubation. Time taken for intubation was significantly longer in those patients intubated with the bronchoscope. In no patient did the arterial oxygen saturation fall below 98%.

Obesity does not Impair Ambulatory Cardiovascular and Autonomic Responses Post-exercise

2021 ◽  
Author(s):  
Gabriel Kolesny Tricot ◽  
Fabiula Isoton Isoton Novelli ◽  
Lucieli Teresa Cambri
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Diastolic Blood Pressure ◽  
Maximal Exercise ◽  
Young Men ◽  
Autonomic Modulation ◽  
Incremental Test ◽  
Post Exercise ◽  
Cardiac Autonomic Modulation ◽  
Main Effect

AbstractThis study aimed to assess whether obesity and/or maximal exercise can change 24 h cardiac autonomic modulation and blood pressure in young men. Thirty-nine men (n: 20; 21.9±1.8 kg·m−2, and n: 19; 32.9±2.4 kg·m−2) were randomly assigned to perform a control (non-exercise) and an experimental day exercise (after maximal incremental test). Cardiac autonomic modulation was evaluated through frequency domain heart rate variability (HRV). Obesity did not impair the ambulatory HRV (p>0.05), however higher diastolic blood pressure during asleep time (p=0.02; group main effect) was observed. The 24 h and awake heart rate was higher on the experimental day (p<0.05; day main effect), regardless of obesity. Hypotension on the experimental day, compared to control day, was observed (p<0.05). Obesity indicators were significantly correlated with heart rate during asleep time (Rho=0.34 to 0.36) and with ambulatory blood pressure(r/Rho=0.32 to 0.53). Furthermore, the HRV threshold workload was significantly correlated with ambulatory heart rate (r/Rho=− 0.38 to−0.52). Finally, ambulatory HRV in obese young men was preserved; however, diastolic blood pressure was increased during asleep time. Maximal exercise caused heart rate increase and 24h hypotension, with decreased cardiac autonomic modulation in the first hour, regardless of obesity.

Cardiovascular Responses to Muscle Stretching: A Systematic Review and Meta-analysis

2021 ◽  
Author(s):  
Ewan Thomas ◽  
Marianna Bellafiore ◽  
Ambra Gentile ◽  
Antonio Paoli ◽  
Antonio Palma ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Cardiovascular System ◽  
Vascular Function ◽  
Meta Analysis ◽  
Random Effect ◽  
Cardiovascular Responses ◽  
Qualitative Description ◽  
Cross Sectional ◽  
Quantitative Synthesis

AbstractThe aim of this study will be to review the current body of literature to understand the effects of stretching on the responses of the cardiovascular system. A literature search was performed using the following databases: Scopus, NLM Pubmed and ScienceDirect. Studies regarding the effects of stretching on responses of the cardiovascular system were investigated. Outcomes regarded heart rate(HR), blood pressure, pulse wave velocity (PWV of which baPWV for brachial-ankle and cfPWV for carotid-femoral waveforms), heart rate variability and endothelial vascular function. Subsequently, the effects of each outcome were quantitatively synthetized using meta-analytic synthesis with random-effect models. A total of 16 studies were considered eligible and included in the quantitative synthesis. Groups were also stratified according to cross-sectional or longitudinal stretching interventions. Quality assessment through the NHLBI tools observed a “fair-to-good” quality of the studies. The meta-analytic synthesis showed a significant effect of d=0.38 concerning HR, d=2.04 regarding baPWV and d=0.46 for cfPWV. Stretching significantly reduces arterial stiffness and HR. The qualitative description of the studies was also supported by the meta-analytic synthesis. No adverse effects were reported, after stretching, in patients affected by cardiovascular disease on blood pressure. There is a lack of studies regarding vascular adaptations to stretching.

Increased Pressor Responsiveness to Enkephalin in Spontaneously Hypertensive Rats: The Role of Vasopressin

1980 ◽  
Vol 59 (s6) ◽  
pp. 235s-237s ◽  
Author(s):  
R. W. Rockhold ◽  
J. T. Crofton ◽  
L. Share
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Pressor Response ◽  
Cardiovascular Effects ◽  
Hypertensive Rats ◽  
Methionine Enkephalin ◽  
Spontaneously Hypertensive ◽  
Wistar Kyoto Rats ◽  
Wistar Kyoto

1. The cardiovascular effects of an enkephalin analogue were examined in spontaneously hypertensive and normotensive Wistar-Kyoto rats. (D-Ala2)-methionine enkephalin caused a biphasic increase in blood pressure and an increase in heart rate after intracerebroventricular injection. 2. The initial pressor response to (D-Ala2)-methionine enkephalin was greater in hypertensive than in normotensive rats. No difference was noted between groups during the secondary pressor response. Heart rate increases paralleled the secondary increase in blood pressure. 3. Naloxone pretreatment abolished the secondary increase in blood pressure and the tachycardia, but did not blunt the initial pressor response in female Wistar-Kyoto rats. 4. Plasma levels of arginine vasopressin were depressed during the plateau phase of the pressor response in hypertensive rats given intracerebroventricular (d-Ala2)-methionine enkephalin. 5. The results suggest that the cardiovascular effects of central enkephalin are not due to vasopressin, but may involve activation of the sympathetic nervous system.

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