scholarly journals Private Weakly-Random Sequences from Human Heart Rate for Quantum Amplification

Entropy ◽  
2021 ◽  
Vol 23 (9) ◽  
pp. 1182
Author(s):  
Maciej Stankiewicz ◽  
Karol Horodecki ◽  
Omer Sakarya ◽  
Danuta Makowiec

We investigate whether the heart rate can be treated as a semi-random source with the aim of amplification by quantum devices. We use a semi-random source model called ε-Santha–Vazirani source, which can be amplified via quantum protocols to obtain a fully private random sequence. We analyze time intervals between consecutive heartbeats obtained from Holter electrocardiogram (ECG) recordings of people of different sex and age. We propose several transformations of the original time series into binary sequences. We have performed different statistical randomness tests and estimated quality parameters. We find that the heart can be treated as a good enough, and private by its nature, source of randomness that every human possesses. As such, in principle, it can be used as input to quantum device-independent randomness amplification protocols. The properly interpreted ε parameter can potentially serve as a new characteristic of the human heart from the perspective of medicine.

1971 ◽  
Author(s):  
D. H. VanDercar ◽  
Neal E. Miller ◽  
Jay M. Weiss ◽  
Henry Solomon

2021 ◽  
Vol 4 (1) ◽  
Author(s):  
Elisa Mejía-Mejía ◽  
James M. May ◽  
Mohamed Elgendi ◽  
Panayiotis A. Kyriacou

AbstractHeart rate variability (HRV) utilizes the electrocardiogram (ECG) and has been widely studied as a non-invasive indicator of cardiac autonomic activity. Pulse rate variability (PRV) utilizes photoplethysmography (PPG) and recently has been used as a surrogate for HRV. Several studies have found that PRV is not entirely valid as an estimation of HRV and that several physiological factors, including the pulse transit time (PTT) and blood pressure (BP) changes, may affect PRV differently than HRV. This study aimed to assess the relationship between PRV and HRV under different BP states: hypotension, normotension, and hypertension. Using the MIMIC III database, 5 min segments of PPG and ECG signals were used to extract PRV and HRV, respectively. Several time-domain, frequency-domain, and nonlinear indices were obtained from these signals. Bland–Altman analysis, correlation analysis, and Friedman rank sum tests were used to compare HRV and PRV in each state, and PRV and HRV indices were compared among BP states using Kruskal–Wallis tests. The findings indicated that there were differences between PRV and HRV, especially in short-term and nonlinear indices, and although PRV and HRV were altered in a similar manner when there was a change in BP, PRV seemed to be more sensitive to these changes.


Author(s):  
Tsu-Wang Shen ◽  
Shan-Chun Chang

Abstract Purpose Although electrocardiogram (ECG) has been proven as a biometric for human identification, applying biometric technology remains challenging with diverse heart rate circumstances in which high intensity heart rate caused waveform deformation may not be known in advance when ECG templates are registered. Methods A calibration method that calculates the ratio of the length of an unidentified electrocardiogram signal to the length of an electrocardiogram template is proposed in this paper. Next, the R peak is used as an axis anchor point of a trigonometric projection (TP) to attain the displacement value. Finally, the unidentified ECG signal is calibrated according to the generated trigonometric value, which corresponds to the trigonometric projection degree of the ratio and the attained displacement measurement. Results The results reveal that the proposed method provides superior overall performance compared with that of the conventional downsampling method, based on the percentage root mean square difference (PRD), correlation coefficients, and mean square error (MSE). Conclusion The curve fitting equation directly maps from the heart rate levels to the TP degree without prior registration information. The proposed ECG calibration method offers a more robust system against heart rate interference when conducting ECG identification.


1999 ◽  
Vol 277 (4) ◽  
pp. H1491-H1497
Author(s):  
Daniel Roach ◽  
Robert Haennel ◽  
Mary Lou Koshman ◽  
Robert Sheldon

We are developing a lexicon of specific heart period changes, or lexons, that recur frequently and whose physiological meaning can be read into ambulatory electrocardiogram (ECG). The transient, reversible “burst” of tachycardia induced by exercise initiation can also be seen on ambulatory ECG. We hypothesized that burst morphology depended on the work that preceded it and on baroreceptor activation. Ten subjects with mean age 38 yr (range 17–69 yr) underwent two protocols of semisupine cycling in which load and duration were varied. Burst duration increased with longer cycling times (median values of 18.0, 25.5, and 23.7 s with 1, 3, and 5 s of cycling, respectively; P= 0.033). Burst shape as assessed by heart period exponential decay constant and burst magnitude did not change. To assess the impact of workload, subjects cycled for 5 s at loads of 0, 25, 50, and 75 W. No significant differences were seen in burst duration, burst magnitude, or burst shape. Tachycardia preceded hypotension by 4.6 ± 2.2 s, which is inconsistent with baroreceptor involvement in the onset of burst tachycardia. Because burst morphology is a nearly quantal response to the initiation of exercise, the presence of a burst on an ambulatory ECG implies the onset of exercise.


2004 ◽  
Vol 29 (7-8) ◽  
pp. 809-813 ◽  
Author(s):  
Arkady L. Maximov ◽  
Natalia N. Maximova
Keyword(s):  

2004 ◽  
Vol 93 (17) ◽  
Author(s):  
Ken Kiyono ◽  
Zbigniew R. Struzik ◽  
Naoko Aoyagi ◽  
Seiichiro Sakata ◽  
Junichiro Hayano ◽  
...  

Author(s):  
Jeremy A. Bigalke ◽  
Ian M. Greenlund ◽  
Jennifer R. Nicevski ◽  
Carl A. Smoot ◽  
Benjamin Oosterhoff ◽  
...  

Chronic insufficient sleep is a common occurrence around the world, and results in numerous physiological detriments and consequences, including cardiovascular complications. The purpose of the present study was to assess the relationship between habitual total sleep time (TST) measured objectively via at-home actigraphy and heart rate (HR) reactivity to nocturnal cortical arousals. We hypothesized that short habitual TST would be associated with exaggerated cardiac reactivity to nocturnal cortical arousals. Participants included in 35 healthy individuals (20 male, 15 female, age: 24 ± 1, BMI: 27 ± 1 kg/m2), and were split using a median analysis into short (SS; n = 17) and normal sleeping (NS; n = 18) adults based on a minimum of 7 days of at-home actigraphy testing. All participants underwent a full overnight laboratory polysomnography (PSG) testing session, including continuous HR (electrocardiogram, ECG) sampling. HR reactivities to all spontaneous cortical arousals were assessed for 20 cardiac cycles following the onset of the arousal in all participants. Baseline HR was not significantly different between groups (P > .05). Spontaneous nocturnal arousal elicited an augmented HR response in the SS group, specifically during the recovery period [F (4.192, 134.134) = 3.413, p = .01]. There were no significant differences in HR reactivity between sexes [F (4.006, 128.189) = .429, p > .05]. These findings offer evidence of nocturnal cardiovascular dysregulation in habitual short sleepers, independent from any diagnosed sleep disorders.


2000 ◽  
Vol 10 (3) ◽  
pp. 107-110 ◽  
Author(s):  
Sebastian H. J. Uijtdehaage ◽  
Julian F. Thayer
Keyword(s):  

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