scholarly journals Mealtime Anxiety and Coping Behaviour in Parents and Children during Weaning in PKU: A Case-Control Study

Nutrients ◽  
2019 ◽  
Vol 11 (12) ◽  
pp. 2857
Author(s):  
Sharon Evans ◽  
Anne Daly ◽  
Jo Wildgoose ◽  
Barbara Cochrane ◽  
Catherine Ashmore ◽  
...  

Solid food introduction may create anxiety for parents of children with phenylketonuria (PKU) due to the burden associated with protein substitute (PS) administration and natural protein restriction. In a longitudinal, prospective study, 20 mothers of children with PKU and 20 non-PKU control mothers completed 4 questionnaires (mealtime emotions, feed-time, Beck’s anxiety inventory and the coping health inventory for parents), examining parent/child mealtime emotions, anxiety, stress and coping strategies at child ages: weaning start, 8 months (m), 12 m, 15 m, 18 m and 24 m. Overall, mothers of children with PKU cope well with solid food introduction when applying a low-phenylalanine diet, with comparable low levels of stress and anxiety reported in both PKU and non-PKU groups. However, mothers of children with PKU reported peak scores in anxiety for emotive/cognitive symptoms at a child age of 15 m, and higher use of coping strategies at 15 m and 24 m (p < 0.05) of age. Generally, there was a trend that maternal anxiety regarding child rejection of PS increased with time, peaking between 12–24 m. In PKU, a child age of 12–18 m is identified as a key period when mothers feel most anxious/stressed with feeding, coinciding with raised blood phenylalanine levels probably associated with teething, illness and developing independence. Health professionals should be conscious of this vulnerable period and be prepared to offer more directional support as required.

2021 ◽  
Vol 17 (4) ◽  
pp. 956
Author(s):  
Fatih Karatas ◽  
Habibe Inci ◽  
Fatih Inci ◽  
Süleyman Ersoy ◽  
Didem Adahan

2012 ◽  
Vol 59 (2) ◽  
pp. 154-159 ◽  
Author(s):  
Hamidreza Roohafza ◽  
Mohammad Talaei ◽  
Zahra Pourmoghaddas ◽  
Fereshteh Rajabi ◽  
Masoumeh Sadeghi

2019 ◽  
Author(s):  
Suleyman Ersoy ◽  
Habibe İnci ◽  
Fatih İnci ◽  
Velittin Selçuk Engin ◽  
Didem Adahan

2014 ◽  
Vol 98 (3) ◽  
pp. 371-376 ◽  
Author(s):  
Caterina Gagliano ◽  
Salvatore Caruso ◽  
Giuseppe Napolitano ◽  
Giulia Malaguarnera ◽  
Maria Vittoria Cicinelli ◽  
...  

2016 ◽  
Vol 8 (3) ◽  
pp. 143 ◽  
Author(s):  
Ab Hameed Raina ◽  
MohammadSultan Allai ◽  
ZafarAmin Shah ◽  
KhalidHamid Changal ◽  
ManzoorAhmad Raina ◽  
...  

1997 ◽  
Vol 27 (10) ◽  
pp. 1151-1159 ◽  
Author(s):  
J. E. MILES ◽  
G. M. E. GARDEN ◽  
W. S. TUNNICLIFFE ◽  
R. M. CAYTON ◽  
J. G. AYRES

2001 ◽  
Vol 100 (6) ◽  
pp. 661-665 ◽  
Author(s):  
Katherine M. ENGLISH ◽  
Peter J. PUGH ◽  
Helen PARRY ◽  
Nanette E. SCUTT ◽  
Kevin S. CHANNER ◽  
...  

The effect of smoking on androgen levels is important given the recent interest in the link between low levels of androgens and the development of cardiovascular disease. Numerous studies examining the effects of cigarette smoking on the levels of total and free testosterone have reported conflicting findings, but there has been no accurate assessment of the effects of cigarette smoking on the levels of bioavailable testosterone [not bound to sex hormone-binding globulin (SHBG)]. We attempted to determine whether smoking affects the level of bioavailable testosterone. We undertook a case-control study of 25 healthy male smokers and 25 healthy never-smokers, matched by age and body mass index. Early morning levels of total, free and bioavailable testosterone, 17β-oestradiol, SHBG and cotinine were determined and compared between the two groups. Levels of total (18.5 ± 4.6 ± nM versus 15.1 ± 4.9 ± nM, P = 0.01) and free testosterone (462 ± 91 ± pM versus 402 ± 93 ± pM, P = 0.03) were found to be higher in smokers compared with non-smokers respectively, as was SHBG (34.1 ± 12.8 versus 28.1 ± 9.0 ± nM, P = 0.06). There were no significant differences in the levels of bioavailable testosterone (3.78 ± 1.59 versus 3.51 ± 1.26 ± nM, P = 0.49) or 17β-oestradiol (44.5 ± 11.4 versus 42.3 ± 11.5 ± pM, P = 0.50) between smokers and non-smokers respectively. These data suggest that cigarette smoking has no significant effect on the biologically active fraction of testosterone, but may influence the levels of total and free testosterone through changes in the levels of SHBG.


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