scholarly journals A case report of an outbreak of coccidiosis in a pig farm at Abeokuta

2021 ◽  
Vol 41 (1) ◽  
pp. 220-223
Author(s):  
S. A. V. Abakpa ◽  
M. I. Takeet ◽  
A. O. Talabi ◽  
I. K. Oyewusi
Keyword(s):  
Drinking Water ◽  
Case Report ◽  
Clinical Signs ◽  
Post Mortem ◽  
Neonatal Piglets ◽  
Counting Technique ◽  
Pig Farm ◽  
Isospora Suis ◽  
Post Mortem Findings ◽  
Haemorrhagic Diarrhoea

An outbreak of coccidiosis was reported in a pig farm housing four boars, six sows, twenty one growers and eleven piglets. Eleven neonatal piglets in two litters of three and five days old were suspected to be involved. Three (27%) out of eleven piglets presented signs of profuse foul smelling diarrhoea and pyrexia, while there was haemorrhagic diarrhoea in one and mortality was 9%. A diagnosis of coccidiosis was made based on the clinical signs, post mortem findings of ulceration and fibrinonecrotic enteritis and laboratory identification of 4,600 coccidial oocysts per gramme of faeces by Mc Master counting technique which were identified as Isospora suis. The two herds were treated with Tripple sulfa-Trim® in drinking water for three days. This medication was effective as the diarrhoea resolved within four days of its onset

scholarly journals Enterocolitis Caused by Ehrlichia sp. in the Horse (Potomac Horse Fever)

1986 ◽  
Vol 23 (4) ◽  
pp. 471-477 ◽  
Author(s):  
D. O. Cordes ◽  
B. D. Perry ◽  
Y. Rikihisa ◽  
W. R. Chickering
Keyword(s):  
Mast Cells ◽  
Epithelial Cells ◽  
Acute Phase ◽  
Whole Blood ◽  
Clinical Signs ◽  
Post Mortem ◽  
Potomac Horse Fever ◽  
Incubation Periods ◽  
Mucous Membranes ◽  
Post Mortem Findings

Potomac horse fever was reproduced in 15 ponies by transfusion of whole blood originally from two natural cases and subsequently from ponies infected by the transfusions. Incubation periods varied from 9 to 15 days. Affected ponies developed varying degrees of fever, diarrhea, anorexia, depression, and leukopenia. Eleven affected ponies were killed, three died in the acute phase of the disease, and one did not show clinical signs. The most consistent post-mortem findings were fluid contents in the cecum and large colon, and areas of hyperemia (of inconstant degree and distribution) in mucosae of both small and large intestines. Multifocal areas of necrosis occurred in mucous membranes. Ehrlichial organisms were most common in the cytoplasm of epithelial cells, macrophages, and mast cells of the large colon.

Studies on Tyzzer's disease in rats

1978 ◽  
Vol 12 (1) ◽  
pp. 1-4 ◽  
Author(s):  
A. Schaich Fries ◽  
O. Svendsen
Keyword(s):  
Antibody Titre ◽  
Causative Agent ◽  
Clinical Signs ◽  
Post Mortem ◽  
Immunofluorescence Technique ◽  
Epidemic Disease ◽  
Breeding Colony ◽  
Indirect Immunofluorescence Technique ◽  
Post Mortem Findings ◽  
The Individual

An outbreak of an epidemic disease occurred in a specified-pathogen-free (SPF) breeding colony of rats. The clinical signs and the post-mortem findings were characteristic for Tyzzer's disease. The causative agent, Bacillus piliformis, was demonstrated microscopically in ileum, liver and myocardium, and transmitted to mice where its pathogenicity appeared to be similar to that of another strain isolated from mice. B. piliformis from spontaneously-infected rats was demonstrated by indirect immunofluorescence technique. By means of the same technique it was found that the fluorescence antibody titre obtained of the individual sera from spontaneously-infected mice, rats and rabbits was the same, whether the antigen employed was organisms isolated from rats or mice. By testing sera from healthy rats in 3 different colonies by use of immunofluorescence technique, antibodies were found in several sera.

scholarly journals Enterotoxaemia involving Clostridium perfringens iota toxin in a hysterectomy-derived rabbit colony

1980 ◽  
Vol 14 (4) ◽  
pp. 347-351 ◽  
Author(s):  
P. Eaton ◽  
D. S. Fernie
Keyword(s):  
Clostridium Perfringens ◽  
Clinical Signs ◽  
Post Mortem ◽  
Toxin A ◽  
Clostridium Perfringens Iota Toxin ◽  
Post Mortem Findings

During an explosive outbreak of fatal enteropathic disease involving Clostridium perfringens iota (i) toxin. a total of 183 deaths occurred in 18 weeks. The clinical signs and post-mortem findings are reported. Examinations for virus, Bacillus piliformis and coccidia were negative. Clostridium perfringens i toxin was detected in 22 of 27 animals examined (81·5%), but clostridia were not isolated. Various treatments wcre attempted. It is concluded that i toxin and the syndrome described are closely related.

Widespread microdysgenesis in therapy-resistant epilepsy – a case report on post-mortem findings

2001 ◽  
Vol 103 (1) ◽  
pp. 74-77 ◽  
Author(s):  
S. Eriksson ◽  
B. Rydenhag ◽  
P. Uvebrant ◽  
K. Malmgren ◽  
C. Nordborg
Keyword(s):  
Case Report ◽  
Post Mortem ◽  
Post Mortem Findings

scholarly journals Aldicarb poisoning of dogs and cats in Gauteng during 2003

2004 ◽  
Vol 75 (4) ◽  
Author(s):  
R.S. Verster ◽  
C.J. Botha ◽  
V. Naidoo ◽  
O.L. Van Schalkwyk
Keyword(s):  
South Africa ◽  
Survival Rate ◽  
Treatment Regimen ◽  
Companion Animals ◽  
Clinical Signs ◽  
Post Mortem ◽  
Duration Of Treatment ◽  
Gauteng Province ◽  
Private Practitioners ◽  
Post Mortem Findings

A survey of aldicarb poisoning in companion animals was conducted by posting questionnaires to all private practitioners in Gauteng Province, South Africa. The survey was designed to determine the percentage of aldicarb cases seen, treatment regimen, clinical signs observed, proposals for preventative actions and more effective treatments. Other questions included duration of treatment, survival rate, cost to clients, post mortem findings and reasons for poisonings

scholarly journals Suspected cyanide toxicity in cattle associated with ingestion of laurel - a case report

2021 ◽  
Vol 74 (1) ◽  
Author(s):  
Aideen Kennedy ◽  
Audrey Brennan ◽  
Celine Mannion ◽  
Maresa Sheehan
Keyword(s):  
Case Report ◽  
Farm Animals ◽  
Response To Treatment ◽  
Post Mortem ◽  
Diet Change ◽  
Cyanide Toxicity ◽  
Large Numbers ◽  
Cyanogenic Plants ◽  
Plant Poisoning ◽  
Post Mortem Findings

Abstract Background Cyanide is one of the most rapidly acting toxins affecting cattle, with poisoning typically occurring following ingestion of cyanogenic plants. Laurel (Prunus laurocerasus), is one such potentially toxic cyanogenic plant. This case report details fatalities in an Irish herd following the ingestion of laurel and aims to raise awareness of the potential risk that access to laurel hedges poses to farm animals. Case presentation Over a twelve-day period, the death occurred of 36 dairy-cross weanlings; the majority (22 weanlings) died over a two-day period. Two days following entry to a field bounded by a laurel hedge, the weanlings displayed signs of lethargy and profuse green diarrhoea. In the majority of animals there was a limited response to treatment with antimicrobials, vitamin B complex and fluid therapy. Recumbency and death ensued. Cyanosis was noted terminally. Two weanlings were submitted for post mortem examination. Laurel leaves were identified in the rumen contents of one weanling. Post mortem findings and additional test results on cohort animals suggested a number of pathological processes may have been involved in the animals, possibly complicating/exacerbating the effects of laurel ingestion. However, cyanide was considered a factor in a least some of the casualties and arrangements were made to test for cyanide on blood samples from a random selection of seven cohort animals. Although collected one week after exposure to the laurel hedge, toxic cyanide levels were recorded in a sample from one animal, which subsequently died. Conclusions The large fatality rate serves as a timely reminder to include plant poisoning as a differential diagnosis when dealing with large numbers of rapid fatalities. Failure to thoroughly examine rumen contents and collect a detailed history in this case, could easily have allowed death to be attributed to other causes and the involvement of cyanide toxicity to be missed. In cases of individual or group fatalities, history is invaluable and recent entry to new grazing areas or any potential diet change or access to garden plants should be thoroughly investigated.

scholarly journals Chocolate ingestion-induced non-cardiogenic pulmonary oedema in a puppy: a case report

2013 ◽  
Vol 58 (No. 2) ◽  
pp. 109-112 ◽  
Author(s):  
CF Agudelo ◽  
Z. Filipejova ◽  
P. Schanilec
Keyword(s):  
Pulmonary Oedema ◽  
Clinical Signs ◽  
Neurological Complications ◽  
Small Animals ◽  
Post Mortem ◽  
Cardiogenic Pulmonary Oedema ◽  
Clinical Presentations ◽  
Life Threatening ◽  
Post Mortem Findings ◽  
First Time

Chocolate intoxication in small animals may be life-threatening and associated with serious morbidity and mortality. The main clinical presentations are cardio-respiratory and neurological complications. One of the most common post-mortem findings is pulmonary oedema which can be a cause of immediate death. We report a case of non-cardiogenic pulmonary oedema thought to be triggered by chocolate intoxication in a three-month-old puppy Dachshund dog. To the authors’ knowledge this is the first time such a complication has been reported after chocolate ingestion. The history, clinical signs, diagnostic approach (radiographs, methylxanthine concentrations, electrocardiography and echocardiography), and treatment are described. The effects of a combination of methylxanthines and other triggers of non-cardiogenic pulmonary oedema are discussed.    

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