scholarly journals Enterocolitis Caused by Ehrlichia sp. in the Horse (Potomac Horse Fever)

1986 ◽  
Vol 23 (4) ◽  
pp. 471-477 ◽  
Author(s):  
D. O. Cordes ◽  
B. D. Perry ◽  
Y. Rikihisa ◽  
W. R. Chickering
Keyword(s):  
Mast Cells ◽  
Epithelial Cells ◽  
Acute Phase ◽  
Whole Blood ◽  
Clinical Signs ◽  
Post Mortem ◽  
Potomac Horse Fever ◽  
Incubation Periods ◽  
Mucous Membranes ◽  
Post Mortem Findings

Potomac horse fever was reproduced in 15 ponies by transfusion of whole blood originally from two natural cases and subsequently from ponies infected by the transfusions. Incubation periods varied from 9 to 15 days. Affected ponies developed varying degrees of fever, diarrhea, anorexia, depression, and leukopenia. Eleven affected ponies were killed, three died in the acute phase of the disease, and one did not show clinical signs. The most consistent post-mortem findings were fluid contents in the cecum and large colon, and areas of hyperemia (of inconstant degree and distribution) in mucosae of both small and large intestines. Multifocal areas of necrosis occurred in mucous membranes. Ehrlichial organisms were most common in the cytoplasm of epithelial cells, macrophages, and mast cells of the large colon.

scholarly journals A REVIEW ON ARSENIC POISONING AND ITS POST-MORTEM FINDINGS

2020 ◽  
pp. 91-97
Author(s):  
Kirti Sharma ◽  
S. R. Inchulkar ◽  
Yuvraj Kaushik
Keyword(s):  
Alimentary Canal ◽  
Cell Metabolism ◽  
Skin Pigmentation ◽  
The Body ◽  
Post Mortem ◽  
Rigor Mortis ◽  
Arsenic Poisoning ◽  
Mucous Membranes ◽  
Rain Drop ◽  
Post Mortem Findings

Arsenic is a grey substance, which is insoluble in water and therefore cannot be absorbed from the alimentary canal. Arsenic is absorbed through all routes mainly by skin, inhalation and GIT mucosa. Arsenic causes toxicity by combining with sulphydryl enzymes and thus interfering with cell metabolism. Locally it causes irritation of the mucous membranes and remotely depression of the nervous system. Arsenic poisoning can be homicidal, suicidal, accidental, occupational, environmental, iatrogenic or un-iatrogenic. The character of post-mortem appearances depends very largely upon the quantity taken and period which has elapsed before death. Externally the body presents dehydrated, cyanosed, sunken eyeballs jaundiced in post-mortem findings. Rigor mortis lasts longer than usual. Internally red velvet stomach, petechial hemorrhages under the endocardium of the left ventricle, patchy fatty degenerative changes with jaundice in liver, rain drop skin pigmentation and mee’s line in nails findings seen in post-mortem.

Studies on Tyzzer's disease in rats

1978 ◽  
Vol 12 (1) ◽  
pp. 1-4 ◽  
Author(s):  
A. Schaich Fries ◽  
O. Svendsen
Keyword(s):  
Antibody Titre ◽  
Causative Agent ◽  
Clinical Signs ◽  
Post Mortem ◽  
Immunofluorescence Technique ◽  
Epidemic Disease ◽  
Breeding Colony ◽  
Indirect Immunofluorescence Technique ◽  
Post Mortem Findings ◽  
The Individual

An outbreak of an epidemic disease occurred in a specified-pathogen-free (SPF) breeding colony of rats. The clinical signs and the post-mortem findings were characteristic for Tyzzer's disease. The causative agent, Bacillus piliformis, was demonstrated microscopically in ileum, liver and myocardium, and transmitted to mice where its pathogenicity appeared to be similar to that of another strain isolated from mice. B. piliformis from spontaneously-infected rats was demonstrated by indirect immunofluorescence technique. By means of the same technique it was found that the fluorescence antibody titre obtained of the individual sera from spontaneously-infected mice, rats and rabbits was the same, whether the antigen employed was organisms isolated from rats or mice. By testing sera from healthy rats in 3 different colonies by use of immunofluorescence technique, antibodies were found in several sera.

Diseases

1933 ◽  
Vol 6 (1-2) ◽  
pp. 99-125
Keyword(s):  
Present State ◽  
Whole Blood ◽  
Rapid Test ◽  
Post Mortem ◽  
Laboratory Workers ◽  
Post Mortem Findings ◽  
Scientific Value

Bacillary White Diarrhœa. De Bacillaire Witte Diarrhe of Witte Afgang der Kuikens. Dr. G. Vandevelde-Vlaamsch-Diergeneeskundig Tjdschrift, 1933, p. 57.Summary:The pullorin- or wattle-test, in its present state of development, cannot be commended for eradication of pullorin disease from poultry flocks. This, according to our own experiments and those of several investigators and laboratory-workers in B. W. D. control. The pullorin reactions don't check with the bacteriological and post-;mortem findings. The only accurate, reliable and suitable methods for the research of the infected birds are the seroagglutination and the whole blood rapid test. The two methods are of similar scientific value.

scholarly journals Enterotoxaemia involving Clostridium perfringens iota toxin in a hysterectomy-derived rabbit colony

1980 ◽  
Vol 14 (4) ◽  
pp. 347-351 ◽  
Author(s):  
P. Eaton ◽  
D. S. Fernie
Keyword(s):  
Clostridium Perfringens ◽  
Clinical Signs ◽  
Post Mortem ◽  
Toxin A ◽  
Clostridium Perfringens Iota Toxin ◽  
Post Mortem Findings

During an explosive outbreak of fatal enteropathic disease involving Clostridium perfringens iota (i) toxin. a total of 183 deaths occurred in 18 weeks. The clinical signs and post-mortem findings are reported. Examinations for virus, Bacillus piliformis and coccidia were negative. Clostridium perfringens i toxin was detected in 22 of 27 animals examined (81·5%), but clostridia were not isolated. Various treatments wcre attempted. It is concluded that i toxin and the syndrome described are closely related.

Treatment of Three Cats with Hyperviscosity Syndrome and Congestive Heart Failure Using Plasmapheresis

2011 ◽  
Vol 47 (1) ◽  
pp. 50-55 ◽  
Author(s):  
Tonya E. Boyle ◽  
Marie K. Holowaychuk ◽  
Allison K. Adams ◽  
Steven L. Marks
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Plasma Cell ◽  
Whole Blood ◽  
Clinical Signs ◽  
Post Mortem ◽  
Respiratory Effort ◽  
Hyperviscosity Syndrome ◽  
Heart Murmurs ◽  
Appropriate Therapy

Three cats were evaluated at a veterinary teaching hospital for congestive heart failure (CHF) secondary to hyperviscosity syndrome from plasma cell neoplasia. All cats had severe hyperproteinemia due to hyperglobulinemia. Multiple myeloma or plasma cell neoplasia was diagnosed based on cytopathology and post mortem examination. The cats presented with signs of CHF including acute collapse, tachypnea, increased respiratory effort, and pulmonary crackles. All cats had heart murmurs and echocardiographic signs consistent with hypertrophic cardiomyopathy. An enlarged left atrium was found in all cats and two of three cats also had spontaneous echocardiographic contrast. Plasmapheresis (centrifugal plasma exchange) was performed on all three cats by the removal of whole blood and the infusion of a balanced electrolyte solution while the whole blood was centrifuged and separated. The RBCs were then washed before being readministered to the patient. Plasmapheresis alleviated the clinical signs of CHF (tachypnea) in all three cats. Plasmapheresis should be considered in cases of CHF secondary to hyperviscosity syndrome to rapidly alleviate clinical signs associated with heart failure while diagnosis of the underlying cause is made and appropriate therapy implemented.

scholarly journals Aldicarb poisoning of dogs and cats in Gauteng during 2003

2004 ◽  
Vol 75 (4) ◽  
Author(s):  
R.S. Verster ◽  
C.J. Botha ◽  
V. Naidoo ◽  
O.L. Van Schalkwyk
Keyword(s):  
South Africa ◽  
Survival Rate ◽  
Treatment Regimen ◽  
Companion Animals ◽  
Clinical Signs ◽  
Post Mortem ◽  
Duration Of Treatment ◽  
Gauteng Province ◽  
Private Practitioners ◽  
Post Mortem Findings

A survey of aldicarb poisoning in companion animals was conducted by posting questionnaires to all private practitioners in Gauteng Province, South Africa. The survey was designed to determine the percentage of aldicarb cases seen, treatment regimen, clinical signs observed, proposals for preventative actions and more effective treatments. Other questions included duration of treatment, survival rate, cost to clients, post mortem findings and reasons for poisonings

scholarly journals Chocolate ingestion-induced non-cardiogenic pulmonary oedema in a puppy: a case report

2013 ◽  
Vol 58 (No. 2) ◽  
pp. 109-112 ◽  
Author(s):  
CF Agudelo ◽  
Z. Filipejova ◽  
P. Schanilec
Keyword(s):  
Pulmonary Oedema ◽  
Clinical Signs ◽  
Neurological Complications ◽  
Small Animals ◽  
Post Mortem ◽  
Cardiogenic Pulmonary Oedema ◽  
Clinical Presentations ◽  
Life Threatening ◽  
Post Mortem Findings ◽  
First Time

Chocolate intoxication in small animals may be life-threatening and associated with serious morbidity and mortality. The main clinical presentations are cardio-respiratory and neurological complications. One of the most common post-mortem findings is pulmonary oedema which can be a cause of immediate death. We report a case of non-cardiogenic pulmonary oedema thought to be triggered by chocolate intoxication in a three-month-old puppy Dachshund dog. To the authors’ knowledge this is the first time such a complication has been reported after chocolate ingestion. The history, clinical signs, diagnostic approach (radiographs, methylxanthine concentrations, electrocardiography and echocardiography), and treatment are described. The effects of a combination of methylxanthines and other triggers of non-cardiogenic pulmonary oedema are discussed.    

scholarly journals A case report of an outbreak of coccidiosis in a pig farm at Abeokuta

2021 ◽  
Vol 41 (1) ◽  
pp. 220-223
Author(s):  
S. A. V. Abakpa ◽  
M. I. Takeet ◽  
A. O. Talabi ◽  
I. K. Oyewusi
Keyword(s):  
Drinking Water ◽  
Case Report ◽  
Clinical Signs ◽  
Post Mortem ◽  
Neonatal Piglets ◽  
Counting Technique ◽  
Pig Farm ◽  
Isospora Suis ◽  
Post Mortem Findings ◽  
Haemorrhagic Diarrhoea

An outbreak of coccidiosis was reported in a pig farm housing four boars, six sows, twenty one growers and eleven piglets. Eleven neonatal piglets in two litters of three and five days old were suspected to be involved. Three (27%) out of eleven piglets presented signs of profuse foul smelling diarrhoea and pyrexia, while there was haemorrhagic diarrhoea in one and mortality was 9%. A diagnosis of coccidiosis was made based on the clinical signs, post mortem findings of ulceration and fibrinonecrotic enteritis and laboratory identification of 4,600 coccidial oocysts per gramme of faeces by Mc Master counting technique which were identified as Isospora suis. The two herds were treated with Tripple sulfa-Trim® in drinking water for three days. This medication was effective as the diarrhoea resolved within four days of its onset

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