Bisphenol A is not detectable in media or selected contact materials used in IVF

2012 ◽  
Vol 25 (6) ◽  
pp. 608-611 ◽  
Author(s):  
Shruthi Mahalingaiah ◽  
Russ Hauser ◽  
Donald G. Patterson ◽  
Million Woudneh ◽  
Catherine Racowsky
2016 ◽  
Vol 850 ◽  
pp. 137-141
Author(s):  
Zhuo Jun Sun ◽  
Chun Ping Li ◽  
Jian Gao ◽  
Shu Zhen Pan ◽  
Shu Li Zhang ◽  
...  

The degradation of polycarbonate (PC) food contact materials was studied based on analysis with GC-MS, FTIR, UV-Vis and TG techniques. It was evident that the processing temperature of the materials significantly enhanced the release of bisphenol A, an endocrine disruptor. Investigation of the redshift of UV band also revealed that high temperatures accelerated the aging degradation of PC.


2021 ◽  
Vol 139 ◽  
pp. 109959
Author(s):  
Antonella Cavazza ◽  
Chiara Bignardi ◽  
Maria Grimaldi ◽  
Paola Salvadeo ◽  
Claudio Corradini

Author(s):  
Chung Stephen WC ◽  
Lau Jason SY

This study aimed (1) to determine the levels of seven phthalates including Di-Ethyl Phthalate (DEP), Di-n-Butyl Phthalate (DnBP), Butyl Benzyl Phthalate (BBzP), Di-(2-Ethylhexyl) Phthalate (DEHP), Di-n-Octyl Phthalate (DnOP), Di-isononyl Phthalate (DiNP) and Di-isodecyl Phthalate (DiDP) in selected foods that are commonly consumed in Hong Kong as well as foods those are reported to be adulterated with phthalates; (2) to estimate the dietary exposure to phthalates of the Hong Kong adult populations at territory-wide scale; and (3) to assess the health risk associated with the exposure. Total Diet Study (TDS) approach was used to assess the associated health risk to the local people. Among the seven phthalates examined, DEHP was the most commonly detected phthalate, followed by DiNP, DnBP, BBzP, DiDP, DEP and DnOP. The maximum detected levels were found to be 23, 43, 93, 560, 3,500, 3,800 and 7,900 µg kg-1 for DnOP, DEP, BBzP, DnBP, DEHP, DiDP and DiNP respectively. It is believed that elevated levels of phthalates detected in isolated samples were more related to chemical nature of the food substrates. Food contact materials used in food manufacturing and packaging may also explain the situation. This study estimated that the dietary exposures to seven phthalates analyzed in the average adult consumer population ranged from a low of 0.098 μgkg-bw-1 day-1 for DnOP (upper bound) to a high of 4.8 μgkg-bw-1 day-1 in the case of DiNP. The exposure to both average and high consumers (95th percentile, or P95) of the adult populations were well within the corresponding Health-Based Guidance Values (HBGVs) for individual phthalate (maximum 13% of HBGVs). Furthermore, no age-sex population sub-group exceeded their respective HBGVs. The findings indicate that dietary exposures to seven phthalates analyzed in this study were unlikely to pose an unacceptable health risk to the Hong Kong population. The food group cereal and its products was the major contributor for DnBP, BBzP, DEHP, DnOP and DiNP dietary exposure, while non-alcoholic drinks and poultry were the major contributors for DEPs and DiDPs, respectively.


Author(s):  
Galen J Gorence ◽  
Hannah C Pulcastro ◽  
Crystal A Lawson ◽  
Roy R Gerona ◽  
Matthew Friesen ◽  
...  

Accidental exposure of our mice to bisphenol A (BPA) from damaged polycarbonate cages 20 y ago provided some of the first evidence of the harmful effects of exposure to this common chemical. Recently we found that housing mice in damaged polysulfone cages resulted in similar harmful effects due to exposure to bisphenol S (BPS). This problem was unexpected for 2 reasons. First, polysulfone is a far more chemically resistant polymer than polycarbonate. Second, BPS is not a component in the manufacture of polysulfone. We report here our efforts to verify the source of the BPS and eliminate the exposure. Our analysis of new polysulfone caging materials confirmed that BPS is a breakdown product of damaged polysulfone plastic. Furthermore, we found that BPS can cross-contaminate new or undamaged cages in facilities that process damaged caging materials. Neither the use of disposable cages nor replacement of caging materials used solely for our colony was sufficient to eliminate exposure effects. Only the replacement of all cages and water bottles in the facility corrected the problem and allowed us to resume our studies. Taken together, our previous and current findings underscore the concern that chemicals from plastics are harmful environmental contaminants for both humans and animals. Furthermore, our results provide strong evidence that the presence of damaged plastic in a facility may be sufficient to affect research results and, by extension, animal health.


Foods ◽  
2021 ◽  
Vol 10 (4) ◽  
pp. 714
Author(s):  
Md Abu bakar Siddique ◽  
Sabine M. Harrison ◽  
Frank J. Monahan ◽  
Enda Cummins ◽  
Nigel P. Brunton

Bisphenol A (BPA) is a commonly used compound in many industries and has versatile applications in polycarbonate plastics and epoxy resins production. BPA is classified as endocrine-disrupting chemical which can hamper fetal development during pregnancy and may have long term negative health outcomes in humans. Dietary sources, main route of BPA exposure, can be contaminated by the migration of BPA into food during processing. The global regulatory framework for using this compound in food contact materials is currently not harmonized. This review aims to outline, survey, and critically evaluate BPA contamination in meat products, including level of BPA and/or metabolites present, exposure route, and recent advancements in the analytical procedures of these compounds from meat and meat products. The contribution of meat and meat products to the total dietary exposure of BPA ranges between 10 and 50% depending on the country and exposure scenario considered. From can lining materials of meat products, BPA migrates towards the solid phase resulting higher BPA concentration in solid phase than the liquid phase of the same can. The analytical procedure is comprised of meat sample pre-treatment, followed by cleaning with solid phase extraction (SPE), and chromatographic analysis. Considering several potential sources of BPA in industrial and home culinary practices, BPA can also accumulate in non-canned or raw meat products. Very few scientific studies have been conducted to identify the amount in raw meat products. Similarly, analysis of metabolites and identification of the origin of BPA contamination in meat products is still a challenge to overcome.


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