scholarly journals Essential role of Ca2+ release channels in angiotensin II-induced Ca2+ oscillations and mesangial cell contraction

2006 ◽  
Vol 70 (1) ◽  
pp. 130-138 ◽  
Author(s):  
Z. Feng ◽  
C. Wei ◽  
X. Chen ◽  
J. Wang ◽  
H. Cheng ◽  
...  
1993 ◽  
Vol 43 (2) ◽  
pp. 324-333 ◽  
Author(s):  
Carmen García-Escribano ◽  
Maria Luisa Díez-Marqués ◽  
Mercedes González-Rubio ◽  
Manuel Rodríguez-Puyol ◽  
Diego Rodríguez-Puyol

2005 ◽  
Vol 280 (44) ◽  
pp. 36737-36746 ◽  
Author(s):  
Shahrzad Abbasi ◽  
Bing Su ◽  
Rodney E. Kellems ◽  
JianHua Yang ◽  
Yang Xia

2002 ◽  
Vol 8 (12) ◽  
pp. 830-840 ◽  
Author(s):  
Saurabh Lodha ◽  
Dhimant Dani ◽  
Rajeev Mehta ◽  
Madhu Bhaskaran ◽  
Krishna Reddy ◽  
...  

1981 ◽  
Vol 240 (1) ◽  
pp. R75-R80 ◽  
Author(s):  
M. C. Lee ◽  
T. N. Thrasher ◽  
D. J. Ramsay

The role of the renin-angiotensin system in drinking induced by water deprivation and caval ligation was assessed by infusion of saralasin into the lateral ventricles of rats. This technique was first validated by demonstrating its capability to specifically antagonize drinking to both systemic and central angiotensin II. However, neither the latency to drink nor the amount of water consumed following 24- or 30-h water deprivation was affected by saralasin. Furthermore, saralasin had no significant effect on the recovery of blood pressure or on the water intake following ligation of the abdominal vena cava. These observations suggest that the renin-angiotensin system alone does not play an essential role in the control of drinking following water deprivation or caval ligation in rats.


2006 ◽  
Vol 98 (8) ◽  
pp. 1032-1039 ◽  
Author(s):  
Wansheng Wang ◽  
Xiao R. Huang ◽  
Ellery Canlas ◽  
Kazuhiro Oka ◽  
Luan D. Truong ◽  
...  

2006 ◽  
Vol 45 (3) ◽  
pp. e6
Author(s):  
Sean P. Didion ◽  
Dale A. Kinzenbaw ◽  
Frank M. Faraci

Hypertension ◽  
2004 ◽  
Vol 44 (3) ◽  
pp. 264-270 ◽  
Author(s):  
Qingwei Zhao ◽  
Minako Ishibashi ◽  
Ken-ichi Hiasa ◽  
Chunyan Tan ◽  
Akira Takeshita ◽  
...  

2011 ◽  
Vol 2011 ◽  
pp. 1-10 ◽  
Author(s):  
Jin-Shuen Chen ◽  
Li-Chien Chang ◽  
Chia-Chao Wu ◽  
Lai-King Yeung ◽  
Yuh-Feng Lin

The aim of this study is to investigate the role of chaperonin-containing t-complex polypeptide 1 beta (CCT2) in the regulation of mouse mesangial cell (mMC) contraction, proliferation, and migration with filamentous/globular-(F/G-) actin ratio under high glucose induction. A low CCT2 mMC model induced by treatment of small interference RNA was established. Groups with and without low CCT2 induction examined in normal and high (H) glucose conditions revealed the following major results: (1) low CCT2 or H glucose showed the ability to attenuate F/G-actin ratio; (2) groups with low F/G-actin ratio all showed less cell contraction; (3) suppression of CCT2 may reduce the proliferation and migration which were originally induced by H glucose. In conclusion, CCT2 can be used as a specific regulator for mMC contraction, proliferation, and migration affected by glucose, which mechanism may involve the alteration of F-actin, particularly for cell contraction.


Sign in / Sign up

Export Citation Format

Share Document